Page last updated: 2024-09-05

sb 203580 and Fibrosarcoma

sb 203580 has been researched along with Fibrosarcoma in 5 studies

Research

Studies (5)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's5 (100.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Babo, I; Harisi, R; Jeney, A; Kenessey, I; Olah, JN; Paku, S; Pogany, G; Timar, F1
ENDOH, D; HAYASHI, M; KADOWAKI, S; OKUI, T1
Huang, J; Ikejima, T; Onodera, S; Tashiro, S; Wu, L1
Choi, YH; Kim, GY; Lee, KJ; Moon, DO1
Akasaki, T; Arai, A; Kanda, E; Miura, O; Miyasaka, N; Nosaka, Y; Sumimoto, H1

Other Studies

5 other study(ies) available for sb 203580 and Fibrosarcoma

ArticleYear
Differential inhibition of single and cluster type tumor cell migration.
    Anticancer research, 2009, Volume: 29, Issue:8

    Topics: Adolescent; Bone Neoplasms; Cell Culture Techniques; Cell Movement; Cell Proliferation; Chromones; Deoxyuridine; Enzyme Inhibitors; Extracellular Signal-Regulated MAP Kinases; Fibrosarcoma; Flavonoids; Humans; Imidazoles; Male; Morpholines; Okadaic Acid; Osteosarcoma; p38 Mitogen-Activated Protein Kinases; Paclitaxel; Phosphatidylinositol 3-Kinases; Phosphoinositide-3 Kinase Inhibitors; Pyridines; Signal Transduction; Tumor Cells, Cultured

2009
Trientine, a copper-chelating agent, induced apoptosis in murine fibrosarcoma cells by activation of the p38 MAPK pathway.
    The Journal of veterinary medical science, 2009, Volume: 71, Issue:11

    Topics: Animals; Apoptosis; Cell Line, Tumor; Chelating Agents; Copper; Enzyme Activation; Fibrosarcoma; Imidazoles; Mice; p38 Mitogen-Activated Protein Kinases; Pyridines; Signal Transduction; Trientine

2009
Bcl-2 up-regulation and P-p53 down-regulation account for the low sensitivity of murine L929 fibrosarcoma cells to oridonin-induced apoptosis.
    Biological & pharmaceutical bulletin, 2005, Volume: 28, Issue:11

    Topics: Animals; Antineoplastic Agents, Phytogenic; Apoptosis; bcl-2-Associated X Protein; Bisbenzimidazole; Blotting, Western; Caspases; Cell Cycle; Cell Line, Tumor; Clone Cells; Diterpenes; Diterpenes, Kaurane; DNA Fragmentation; Drug Resistance, Neoplasm; Enzyme Inhibitors; Fibrosarcoma; Flow Cytometry; Gene Expression Regulation, Neoplastic; Genes, bcl-2; Genes, p53; Imidazoles; Mice; Phosphorylation; Pyridines; Tumor Suppressor Protein p53

2005
Beta-sitosterol-induced-apoptosis is mediated by the activation of ERK and the downregulation of Akt in MCA-102 murine fibrosarcoma cells.
    International immunopharmacology, 2007, Volume: 7, Issue:8

    Topics: Animals; Anthracenes; Apoptosis; Apoptosis Regulatory Proteins; Caspase Inhibitors; Caspases; Cell Line, Tumor; Cell Survival; Chromones; Dose-Response Relationship, Drug; Down-Regulation; Extracellular Signal-Regulated MAP Kinases; Fibrosarcoma; G1 Phase; Hypolipidemic Agents; Imidazoles; Mice; Models, Biological; Morpholines; Oligopeptides; Phosphatidylinositol 3-Kinases; Phosphoinositide-3 Kinase Inhibitors; Proto-Oncogene Proteins; Proto-Oncogene Proteins c-akt; Proto-Oncogene Proteins c-bcl-2; Pyridines; Signal Transduction; Sitosterols; Tumor Suppressor Protein p53

2007
Rac is activated by tumor necrosis factor alpha and is involved in activation of Erk.
    Biochemical and biophysical research communications, 2001, Jul-20, Volume: 285, Issue:3

    Topics: Animals; Cell Line; Chromones; Enzyme Activation; Enzyme Inhibitors; Fibrosarcoma; Genes, Dominant; Imidazoles; Mice; Mitogen-Activated Protein Kinases; Morpholines; Mutagenesis, Site-Directed; p38 Mitogen-Activated Protein Kinases; Phosphatidylinositol 3-Kinases; Phosphoinositide-3 Kinase Inhibitors; Protein Serine-Threonine Kinases; Proto-Oncogene Proteins; Proto-Oncogene Proteins c-akt; Pyridines; rac GTP-Binding Proteins; Transfection; Tumor Necrosis Factor-alpha

2001