Page last updated: 2024-09-05

sb 203580 and Cicatrix

sb 203580 has been researched along with Cicatrix in 2 studies

Research

Studies (2)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	0 (0.00)	18.2507
2000's	1 (50.00)	29.6817
2010's	1 (50.00)	24.3611
2020's	0 (0.00)	2.80

Authors

Authors	Studies
Chang, Y; Shi, L; Wu, X; Yang, Y; Yu, FS; Zhang, Y	1
Engel, FB; Hsieh, PC; Keating, MT; Lee, RT	1

Other Studies

2 other study(ies) available for sb 203580 and Cicatrix

Article	Year
Activation of JNK signaling mediates connective tissue growth factor expression and scar formation in corneal wound healing. PloS one, 2012, Volume: 7, Issue:2 Topics: Animals; Anthracenes; Cell Line, Transformed; Cicatrix; Collagen Type I; Connective Tissue Growth Factor; Cornea; Corneal Stroma; Enzyme Activation; Eye Injuries, Penetrating; Fibroblasts; Fibronectins; Flavonoids; Gene Expression Regulation; Humans; Imidazoles; JNK Mitogen-Activated Protein Kinases; Male; MAP Kinase Signaling System; Phosphorylation; Pyridines; Rats; Rats, Wistar; Telomerase; Transforming Growth Factor beta1; Wound Healing	2012
FGF1/p38 MAP kinase inhibitor therapy induces cardiomyocyte mitosis, reduces scarring, and rescues function after myocardial infarction. Proceedings of the National Academy of Sciences of the United States of America, 2006, Oct-17, Volume: 103, Issue:42 Topics: Animals; Cicatrix; Enzyme Inhibitors; Fibroblast Growth Factor 1; Imidazoles; Male; Mitosis; Myocardial Infarction; Myocytes, Cardiac; p38 Mitogen-Activated Protein Kinases; Pyridines; Rats; Rats, Sprague-Dawley; Regeneration	2006

Article

Year

Activation of JNK signaling mediates connective tissue growth factor expression and scar formation in corneal wound healing.

PloS one, 2012, Volume: 7, Issue:2

Topics: Animals; Anthracenes; Cell Line, Transformed; Cicatrix; Collagen Type I; Connective Tissue Growth Factor; Cornea; Corneal Stroma; Enzyme Activation; Eye Injuries, Penetrating; Fibroblasts; Fibronectins; Flavonoids; Gene Expression Regulation; Humans; Imidazoles; JNK Mitogen-Activated Protein Kinases; Male; MAP Kinase Signaling System; Phosphorylation; Pyridines; Rats; Rats, Wistar; Telomerase; Transforming Growth Factor beta1; Wound Healing

2012

FGF1/p38 MAP kinase inhibitor therapy induces cardiomyocyte mitosis, reduces scarring, and rescues function after myocardial infarction.

Proceedings of the National Academy of Sciences of the United States of America, 2006, Oct-17, Volume: 103, Issue:42

Topics: Animals; Cicatrix; Enzyme Inhibitors; Fibroblast Growth Factor 1; Imidazoles; Male; Mitosis; Myocardial Infarction; Myocytes, Cardiac; p38 Mitogen-Activated Protein Kinases; Pyridines; Rats; Rats, Sprague-Dawley; Regeneration

2006