sb 203580 has been researched along with Cell Transformation, Neoplastic in 9 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 7 (77.78) | 29.6817 |
| 2010's | 2 (22.22) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Bowman, L; Ding, M; Hou, L; Meighan, TG; Shi, X | 1 |
| Chen, X; Duan, L; He, T; Luo, J; Shi, Q; Tang, M; Wang, H; Weng, Y; Wu, R; Yang, X; Ye, L; Zhang, Y; Zhou, L | 1 |
| Fabra, A; Guerrero, J; Martínez, J; Quintanilla, M; Santibáñez, JF | 1 |
| Abbott, RT; Bohling, SD; Buckstein, R; Coupland, RW; Dube, ID; Elenitoba-Johnson, KS; Jenson, SD; Lim, MS; Lin, Z; Palais, RA; Perez-Ordonez, B; Perkins, SL; Shami, PJ; Tripp, S; Wang, LY | 1 |
| Carpenter, G; Tikhomirov, O | 1 |
| Shapiro, DJ; Zhang, CC | 1 |
| Okajima, E; Thorgeirsson, UP | 1 |
| Eliazer, S; Ilaria, RL; Silvany, RE; Wolff, NC | 1 |
| Bilter, GK; Der, CJ; Pruitt, K; Pruitt, WM; Westwick, JK | 1 |
9 other study(ies) available for sb 203580 and Cell Transformation, Neoplastic
| Article | Year |
|---|---|
Induction of miR-21-PDCD4 signaling by tungsten carbide-cobalt nanoparticles in JB6 cells involves ROS-mediated MAPK pathways.
Topics: Animals; Apoptosis Regulatory Proteins; Butadienes; Cell Line; Cell Transformation, Neoplastic; Cobalt; Disease Models, Animal; Enzyme Inhibitors; Epidermis; Hydrogen Peroxide; Imidazoles; In Vitro Techniques; MAP Kinase Signaling System; Mice; MicroRNAs; Mitogen-Activated Protein Kinase Kinases; Nanoparticles; Nitriles; Occupational Exposure; Pyridines; Reactive Oxygen Species; RNA-Binding Proteins; Signal Transduction; Tungsten Compounds | 2013 |
S100A9 promotes the proliferation and invasion of HepG2 hepatocellular carcinoma cells via the activation of the MAPK signaling pathway.
Topics: Animals; Calgranulin B; Carcinoma, Hepatocellular; Cell Line, Tumor; Cell Proliferation; Cell Transformation, Neoplastic; Enzyme Inhibitors; Extracellular Signal-Regulated MAP Kinases; Female; Flavonoids; Hep G2 Cells; Humans; Imidazoles; Liver Neoplasms; MAP Kinase Signaling System; Mice; Mice, Nude; Mitogen-Activated Protein Kinases; Neoplasm Invasiveness; Neoplasm Transplantation; p38 Mitogen-Activated Protein Kinases; Phosphorylation; Protein Kinase Inhibitors; Pyridines; Random Allocation; Tetrazolium Salts; Thiazoles; Xenograft Model Antitumor Assays | 2013 |
Transforming growth factor-beta1 modulates matrix metalloproteinase-9 production through the Ras/MAPK signaling pathway in transformed keratinocytes.
Topics: Animals; Cell Line, Transformed; Cell Transformation, Neoplastic; Curcumin; Dipeptides; Enzyme Inhibitors; Flavonoids; Genes, Reporter; Humans; Imidazoles; Keratinocytes; MAP Kinase Signaling System; Matrix Metalloproteinase 9; Matrix Metalloproteinase Inhibitors; Mice; Oligodeoxyribonucleotides, Antisense; Promoter Regions, Genetic; Pyridines; ras Proteins; Recombinant Proteins; Transforming Growth Factor beta; Transforming Growth Factor beta1; Tumor Cells, Cultured | 2002 |
Involvement of multiple signaling pathways in follicular lymphoma transformation: p38-mitogen-activated protein kinase as a target for therapy.
Topics: Apoptosis; Cell Transformation, Neoplastic; Chromosomes, Human, Pair 14; Chromosomes, Human, Pair 18; Enzyme Inhibitors; Gene Expression Profiling; Humans; Imidazoles; Lymphoma, B-Cell; Lymphoma, Follicular; Lymphoma, Large B-Cell, Diffuse; MAP Kinase Signaling System; Mitogen-Activated Protein Kinases; Oligonucleotide Array Sequence Analysis; p38 Mitogen-Activated Protein Kinases; Pyridines; Receptors, Cytokine; Receptors, Growth Factor; Translocation, Genetic; Tumor Cells, Cultured | 2003 |
Ligand-induced, p38-dependent apoptosis in cells expressing high levels of epidermal growth factor receptor and ErbB-2.
Topics: Animals; Apoptosis; Blotting, Western; Cell Cycle; Cell Death; Cell Division; Cell Line; Cell Line, Tumor; Cell Survival; Cell Transformation, Neoplastic; DNA; DNA Fragmentation; Electrophoresis, Polyacrylamide Gel; Enzyme Activation; Enzyme Inhibitors; ErbB Receptors; Fibroblasts; Genetic Vectors; Green Fluorescent Proteins; Humans; Imidazoles; Immunoblotting; Ligands; Luminescent Proteins; Mice; Mitochondria; Mitogen-Activated Protein Kinases; NIH 3T3 Cells; p38 Mitogen-Activated Protein Kinases; Protein Structure, Tertiary; Pyridines; Receptor, ErbB-2; Time Factors | 2004 |
Activation of the p38 mitogen-activated protein kinase pathway by estrogen or by 4-hydroxytamoxifen is coupled to estrogen receptor-induced apoptosis.
Topics: Apoptosis; Breast Neoplasms; Cell Transformation, Neoplastic; Enzyme Induction; Estradiol; Female; HeLa Cells; Humans; Imidazoles; MAP Kinase Kinase 3; Mitogen-Activated Protein Kinase Kinases; Mitogen-Activated Protein Kinases; p38 Mitogen-Activated Protein Kinases; Protein-Tyrosine Kinases; Pyridines; Receptors, Estrogen; Recombinant Proteins; Tamoxifen | 2000 |
Different regulation of vascular endothelial growth factor expression by the ERK and p38 kinase pathways in v-ras, v-raf, and v-myc transformed cells.
Topics: Animals; Breast Neoplasms; Calcium-Calmodulin-Dependent Protein Kinases; Carcinoma; Cell Line; Cell Transformation, Neoplastic; Endothelial Growth Factors; Epithelial Cells; Flavonoids; Genes, myc; Genes, ras; Imidazoles; Liver; Lymphokines; MAP Kinase Signaling System; Mitogen-Activated Protein Kinases; Neovascularization, Pathologic; Oncogene Proteins v-raf; p38 Mitogen-Activated Protein Kinases; Pyridines; Rats; Retroviridae Proteins, Oncogenic; Vascular Endothelial Growth Factor A; Vascular Endothelial Growth Factors | 2000 |
Interference with the constitutive activation of ERK1 and ERK2 impairs EWS/FLI-1-dependent transformation.
Topics: 3T3 Cells; Animals; Binding Sites; Bone Neoplasms; Butadienes; Cell Transformation, Neoplastic; Colony-Forming Units Assay; DNA; Enzyme Activation; Enzyme Inhibitors; Flavonoids; Genes, ras; Humans; Imidazoles; MAP Kinase Kinase 1; Mice; Mitogen-Activated Protein Kinase 1; Mitogen-Activated Protein Kinase 3; Mitogen-Activated Protein Kinase Kinases; Mitogen-Activated Protein Kinases; Mutation; Nitriles; Oncogene Proteins, Fusion; Protein Serine-Threonine Kinases; Proto-Oncogene Protein c-fli-1; Pyridines; RNA-Binding Protein EWS; Sarcoma, Ewing; Transcription Factors; Tumor Cells, Cultured | 2000 |
Raf-independent deregulation of p38 and JNK mitogen-activated protein kinases are critical for Ras transformation.
Topics: Animals; Cell Cycle; Cell Division; Cell Line; Cell Transformation, Neoplastic; Cloning, Molecular; Culture Media, Conditioned; Cyclin D1; Cyclins; Enzyme Inhibitors; Flavonoids; Genes, ras; Imidazoles; Intestinal Mucosa; JNK Mitogen-Activated Protein Kinases; Mitogen-Activated Protein Kinases; Mutagenesis, Site-Directed; p38 Mitogen-Activated Protein Kinases; Phosphorylation; Proto-Oncogene Proteins c-raf; Proto-Oncogene Proteins p21(ras); Pyridines; Rats; Recombinant Proteins; Transforming Growth Factor alpha | 2002 |