sb 203580 has been researched along with Bone Loss, Osteoclastic in 3 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 1 (33.33) | 18.2507 |
2000's | 2 (66.67) | 29.6817 |
2010's | 0 (0.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Falany, ML; Larsen, KI; Ponomareva, LV; Wang, W; Williams, JP | 1 |
Ariyoshi, W; Ichimiya, H; Kanno, T; Koseki, T; Nishihara, T; Takahashi, T; Takano, H | 1 |
Adams, JL; Badger, AM; Bradbeer, JN; Griswold, DE; Lee, JC; Votta, B | 1 |
3 other study(ies) available for sb 203580 and Bone Loss, Osteoclastic
Article | Year |
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Glucose-dependent regulation of osteoclast H(+)-ATPase expression: potential role of p38 MAP-kinase.
Topics: Animals; Biological Transport; Birds; Blotting, Northern; Blotting, Western; Bone Resorption; Calmodulin; Dose-Response Relationship, Drug; Enzyme Activation; Enzyme Inhibitors; Glucose; Imidazoles; Kinetics; Microscopy, Confocal; Mitogen-Activated Protein Kinases; Osteoclasts; p38 Mitogen-Activated Protein Kinases; Precipitin Tests; Protein Structure, Tertiary; Proton-Translocating ATPases; Pyridines; Rats; RNA, Messenger; Time Factors; Up-Regulation | 2002 |
Mechanisms involved in enhancement of osteoclast formation and function by low molecular weight hyaluronic acid.
Topics: Acid Phosphatase; Animals; Antibodies, Monoclonal; Blotting, Western; Bone Resorption; Carrier Proteins; Cell Differentiation; Cell Line; Cell Survival; CSK Tyrosine-Protein Kinase; Dose-Response Relationship, Drug; Enzyme Inhibitors; Extracellular Matrix; Hyaluronan Receptors; Hyaluronic Acid; Imidazoles; Isoenzymes; Membrane Glycoproteins; Mice; Molecular Weight; Monocytes; Osteoclasts; p38 Mitogen-Activated Protein Kinases; Phosphorylation; Protein-Tyrosine Kinases; Pyridines; RANK Ligand; Receptor Activator of Nuclear Factor-kappa B; Signal Transduction; src-Family Kinases; Tartrate-Resistant Acid Phosphatase; Time Factors; Up-Regulation | 2005 |
Pharmacological profile of SB 203580, a selective inhibitor of cytokine suppressive binding protein/p38 kinase, in animal models of arthritis, bone resorption, endotoxin shock and immune function.
Topics: Animals; Arachidonic Acid; Arthritis; Bone Resorption; Calcium-Calmodulin-Dependent Protein Kinases; Collagen; Disease Models, Animal; Enzyme Inhibitors; Female; Imidazoles; Male; Mice; Mice, Inbred Strains; Mitogen-Activated Protein Kinases; p38 Mitogen-Activated Protein Kinases; Pyridines; Rats; Rats, Inbred Lew; Shock, Septic; Tumor Necrosis Factor-alpha | 1996 |