Page last updated: 2024-09-05

sb 203580 and Airflow Obstruction, Chronic

sb 203580 has been researched along with Airflow Obstruction, Chronic in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's3 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Al-Jahdali, H; Al-Kufaidy, R; Al-Muhsen, S; Alnassar, SA; BaHammam, AS; Halwani, R; Hamid, Q; Pureza, MA; Vazquez-Tello, A1
Chen, X; Dong, L; Gong, Y; Huang, Y; Jin, X; Li, S; Li, W; Long, F; Xia, J; Yang, H; Yu, R; Zhang, P; Zhang, Y; Zhu, N1
Kopec-Harding, K; Li, J; Meng, A; Wu, J; Wu, M; Wu, S; Yan, X; Zhang, X1

Other Studies

3 other study(ies) available for sb 203580 and Airflow Obstruction, Chronic

ArticleYear
IL-17 Enhances Chemotaxis of Primary Human B Cells during Asthma.
    PloS one, 2014, Volume: 9, Issue:12

    Topics: Adult; Asthma; B-Lymphocytes; Bronchi; Chemokine CXCL13; Chemotaxis; Enzyme Activation; Female; Humans; Imidazoles; Inflammation; Interleukin-17; Interleukin-8; Male; p38 Mitogen-Activated Protein Kinases; Pulmonary Disease, Chronic Obstructive; Pyridines; Receptors, Interleukin; Receptors, Interleukin-17; Th17 Cells

2014
1α,25-Dihydroxyvitamin D3 Induces Neutrophil Apoptosis through the p38 MAPK Signaling Pathway in Chronic Obstructive Pulmonary Disease Patients.
    PloS one, 2015, Volume: 10, Issue:4

    Topics: Aged; Apoptosis; Case-Control Studies; Female; Humans; Imidazoles; Male; Neutrophils; p38 Mitogen-Activated Protein Kinases; Phosphorylation; Pulmonary Disease, Chronic Obstructive; Pyridines; Signal Transduction; Vitamin D

2015
In vitro modeling of COPD inflammation and limitation of p38 inhibitor - SB203580.
    International journal of chronic obstructive pulmonary disease, 2016, Volume: 11

    Topics: Aged; Anti-Inflammatory Agents; Case-Control Studies; Cell Line; Chemokine CCL5; Cytokines; Female; Humans; Imidazoles; Inflammation Mediators; Interleukin-10; Macrophages, Alveolar; Male; Middle Aged; p38 Mitogen-Activated Protein Kinases; Phosphorylation; Protein Kinase Inhibitors; Pulmonary Disease, Chronic Obstructive; Pyridines; Signal Transduction; Tumor Necrosis Factor-alpha

2016