Page last updated: 2024-09-05

sb 203580 and Acrocephalosyndactylia

sb 203580 has been researched along with Acrocephalosyndactylia in 2 studies

Research

Studies (2)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	0 (0.00)	18.2507
2000's	1 (50.00)	29.6817
2010's	1 (50.00)	24.3611
2020's	0 (0.00)	2.80

Authors

Authors	Studies
Chang, M; Chen, P; Li, Y; Sun, S; Weng, T; Zhang, B; Zhang, L; Zhang, S	1
Delannoy, P; Lemonnier, J; Lomri, A; Marie, PJ	1

Other Studies

2 other study(ies) available for sb 203580 and Acrocephalosyndactylia

Article	Year
A Ser252Trp mutation in fibroblast growth factor receptor 2 (FGFR2) mimicking human Apert syndrome reveals an essential role for FGF signaling in the regulation of endochondral bone formation. PloS one, 2014, Volume: 9, Issue:1 Topics: Acrocephalosyndactylia; Animals; Cell Differentiation; Embryonic Development; Flavonoids; Gene Knock-In Techniques; Humans; Imidazoles; Mesenchymal Stem Cells; Mice; Mice, Inbred C57BL; Mutation; Osteogenesis; Phosphorylation; Pyridines; Receptor, Fibroblast Growth Factor, Type 2; Signal Transduction; Up-Regulation	2014
Increased expression of protein kinase Calpha, interleukin-1alpha, and RhoA guanosine 5'-triphosphatase in osteoblasts expressing the Ser252Trp fibroblast growth factor 2 receptor Apert mutation: identification by analysis of complementary DNA microarray. Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research, 2001, Volume: 16, Issue:4 Topics: Acrocephalosyndactylia; Amino Acid Substitution; Cell Line, Transformed; DNA, Complementary; Enzyme Induction; Enzyme Inhibitors; Fetus; Flavonoids; Gene Expression Regulation; Humans; Imidazoles; Interleukin-1; Isoenzymes; MAP Kinase Kinase Kinase 1; MAP Kinase Signaling System; Mitogen-Activated Protein Kinases; Naphthalenes; Oligonucleotide Array Sequence Analysis; Osteoblasts; p38 Mitogen-Activated Protein Kinases; Point Mutation; Protein Kinase C; Protein Kinase C-alpha; Protein Serine-Threonine Kinases; Pyridines; Receptor Protein-Tyrosine Kinases; Receptor, Fibroblast Growth Factor, Type 2; Receptors, Fibroblast Growth Factor; rhoA GTP-Binding Protein; Skull; Subtraction Technique	2001

Article

Year

A Ser252Trp mutation in fibroblast growth factor receptor 2 (FGFR2) mimicking human Apert syndrome reveals an essential role for FGF signaling in the regulation of endochondral bone formation.

PloS one, 2014, Volume: 9, Issue:1

Topics: Acrocephalosyndactylia; Animals; Cell Differentiation; Embryonic Development; Flavonoids; Gene Knock-In Techniques; Humans; Imidazoles; Mesenchymal Stem Cells; Mice; Mice, Inbred C57BL; Mutation; Osteogenesis; Phosphorylation; Pyridines; Receptor, Fibroblast Growth Factor, Type 2; Signal Transduction; Up-Regulation

2014

Increased expression of protein kinase Calpha, interleukin-1alpha, and RhoA guanosine 5'-triphosphatase in osteoblasts expressing the Ser252Trp fibroblast growth factor 2 receptor Apert mutation: identification by analysis of complementary DNA microarray.

Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research, 2001, Volume: 16, Issue:4

Topics: Acrocephalosyndactylia; Amino Acid Substitution; Cell Line, Transformed; DNA, Complementary; Enzyme Induction; Enzyme Inhibitors; Fetus; Flavonoids; Gene Expression Regulation; Humans; Imidazoles; Interleukin-1; Isoenzymes; MAP Kinase Kinase Kinase 1; MAP Kinase Signaling System; Mitogen-Activated Protein Kinases; Naphthalenes; Oligonucleotide Array Sequence Analysis; Osteoblasts; p38 Mitogen-Activated Protein Kinases; Point Mutation; Protein Kinase C; Protein Kinase C-alpha; Protein Serine-Threonine Kinases; Pyridines; Receptor Protein-Tyrosine Kinases; Receptor, Fibroblast Growth Factor, Type 2; Receptors, Fibroblast Growth Factor; rhoA GTP-Binding Protein; Skull; Subtraction Technique

2001