Page last updated: 2024-11-04

sb 202190 and Bleb

sb 202190 has been researched along with Bleb in 2 studies

4-(4-fluorophenyl)-2-(4-hydroxyphenyl)-5-(4-pyridyl)imidazole: structure given in first source; inhibits p38 MAP kinase

Research Excerpts

ExcerptRelevanceReference
"Pemphigus vulgaris (PV) is a skin blistering disease caused by autoantibodies targeting the desmosomal adhesion proteins desmoglein (Dsg) 3 and 1."1.46Inhibition of p38MAPK signalling prevents epidermal blistering and alterations of desmosome structure induced by pemphigus autoantibodies in human epidermis. ( Egu, DT; Spindler, V; Walter, E; Waschke, J, 2017)

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's2 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Egu, DT1
Walter, E1
Spindler, V1
Waschke, J1
Saito, M1
Stahley, SN1
Caughman, CY1
Mao, X1
Tucker, DK1
Payne, AS1
Amagai, M1
Kowalczyk, AP1

Other Studies

2 other studies available for sb 202190 and Bleb

ArticleYear
Inhibition of p38MAPK signalling prevents epidermal blistering and alterations of desmosome structure induced by pemphigus autoantibodies in human epidermis.
    The British journal of dermatology, 2017, Volume: 177, Issue:6

    Topics: Antibodies, Monoclonal; Autoantibodies; Blister; Desmosomes; Epidermis; Humans; Imidazoles; Immunogl

2017
Signaling dependent and independent mechanisms in pemphigus vulgaris blister formation.
    PloS one, 2012, Volume: 7, Issue:12

    Topics: Animals; Antibodies, Monoclonal; Blister; Cell Adhesion; Cluster Analysis; Desmoglein 3; Desmosomes;

2012