sarkosyl has been researched along with Dementias, Transmissible in 3 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 3 (100.00) | 29.6817 |
2010's | 0 (0.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Li, L; Park, KW | 1 |
Bauman, PA; Biesert, L; Cai, K; Dichtelmüller, H; Fabbrizzi, F; Gajardo, R; Gröner, A; Jorquera, JI; Kempf, C; Kreil, TR; Lawrence, LA; Petteway, SR; Pifat, DY; von Hoegen, I | 1 |
Gilch, S; Groschup, MH; Lucassen, R; Muranyi, W; Nunziante, M; Riesner, D; Schätzl, HM; Spielhaupter, C; Tatzelt, J; Winklhofer, KF | 1 |
3 other study(ies) available for sarkosyl and Dementias, Transmissible
Article | Year |
---|---|
Cytoplasmic expression of mouse prion protein causes severe toxicity in Caenorhabditis elegans.
Topics: Animals; Animals, Genetically Modified; Caenorhabditis elegans; Cell Movement; Cytoplasm; Detergents; Disease Models, Animal; Endopeptidase K; Mice; Muscle Cells; Mutation; Prion Diseases; Prion Proteins; Prions; Protein Folding; Sarcosine; Solubility | 2008 |
Critical factors influencing prion inactivation by sodium hydroxide.
Topics: Animals; Biological Assay; Cricetinae; Decontamination; Endopeptidase K; Prion Diseases; PrPSc Proteins; Sarcosine; Sodium Hydroxide | 2006 |
Intracellular re-routing of prion protein prevents propagation of PrP(Sc) and delays onset of prion disease.
Topics: Acids; Amidohydrolases; Animals; Cell Compartmentation; Detergents; Golgi Apparatus; Intracellular Fluid; Mice; Mice, Transgenic; Peptide-N4-(N-acetyl-beta-glucosaminyl) Asparagine Amidase; Prion Diseases; Prions; Protein Folding; Protein Structure, Secondary; PrPSc Proteins; Sarcosine; Suramin; trans-Golgi Network; Tumor Cells, Cultured | 2001 |