Page last updated: 2024-08-21

sapropterin and Left Ventricular Dysfunction

sapropterin has been researched along with Left Ventricular Dysfunction in 5 studies

Research

Studies (5)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (20.00)29.6817
2010's4 (80.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Fujita, M; Iwasaka, T; Otani, H; Shimazu, T; Shiojima, I; Yoshioka, K1
Bedja, D; Carnicer, R; Casadei, B; Channon, KM; Duglan, D; Hahn, VS; Hashimoto, T; Kass, DA; Recalde, A; Sivakumaran, V; Zhu, G1
Boontje, NM; de Crom, R; de Waard, MC; Dekkers, DH; Duncker, DJ; Kuster, DW; Lamers, JM; van der Velden, J; van Haperen, R1
Bernstein, KE; Boulden, BM; Dikalov, S; Dudley, SC; Fan, TH; Fredd, S; Harrison, DG; Jiao, Z; Liu, H; Lovelock, JD; Silberman, GA; Widder, J; Wolska, BM; Xiao, HD1
Fujita, M; Iwasaka, T; Jo, F; Jo, H; Kosaki, A; Okazaki, T; Otani, H; Shimazu, T; Yoshioka, K1

Other Studies

5 other study(ies) available for sapropterin and Left Ventricular Dysfunction

ArticleYear
Sepiapterin prevents left ventricular hypertrophy and dilatory remodeling induced by pressure overload in rats.
    American journal of physiology. Heart and circulatory physiology, 2015, Nov-15, Volume: 309, Issue:10

    Topics: Animals; Aorta; Biopterins; Capillaries; Cell Size; Constriction; Dilatation, Pathologic; Enzyme Inhibitors; Glycine; Heart; Hypertrophy, Left Ventricular; Male; Myocardium; Myocytes, Cardiac; NG-Nitroarginine Methyl Ester; Nitric Oxide; Nitric Oxide Synthase; Organ Size; Oxidative Stress; Pressure; Pterins; Rats; Rats, Sprague-Dawley; Sulfhydryl Compounds; Ultrasonography; Ventricular Dysfunction, Left; Ventricular Remodeling

2015
Tetrahydrobiopterin Protects Against Hypertrophic Heart Disease Independent of Myocardial Nitric Oxide Synthase Coupling.
    Journal of the American Heart Association, 2016, Mar-21, Volume: 5, Issue:3

    Topics: Animals; Anti-Inflammatory Agents; Biopterins; Cardiovascular Agents; Cytokines; Cytoprotection; Disease Models, Animal; GTP Cyclohydrolase; Humans; Hypertrophy, Left Ventricular; Inflammation Mediators; Macrophages; Mice, Transgenic; Myocytes, Cardiac; Nitric Oxide; Nitric Oxide Synthase; Oxidation-Reduction; Signal Transduction; Superoxides; Time Factors; Ventricular Dysfunction, Left; Ventricular Function, Left; Ventricular Remodeling

2016
Detrimental effect of combined exercise training and eNOS overexpression on cardiac function after myocardial infarction.
    American journal of physiology. Heart and circulatory physiology, 2009, Volume: 296, Issue:5

    Topics: Acetylcysteine; Actin Cytoskeleton; Animals; Antioxidants; Arginine; Biopterins; Disease Models, Animal; Exercise Therapy; Female; Humans; Male; Mice; Mice, Inbred C57BL; Mice, Transgenic; Myocardial Contraction; Myocardial Infarction; Myocardium; Nitric Oxide Synthase Type III; Oxidative Stress; Phosphorylation; Physical Exertion; Sarcoplasmic Reticulum Calcium-Transporting ATPases; Superoxides; Time Factors; Up-Regulation; Ventricular Dysfunction, Left; Ventricular Pressure; Ventricular Remodeling

2009
Uncoupled cardiac nitric oxide synthase mediates diastolic dysfunction.
    Circulation, 2010, Feb-02, Volume: 121, Issue:4

    Topics: Animals; Biopterins; Calcium-Binding Proteins; Desoxycorticosterone; Disease Models, Animal; Heart Failure, Diastolic; Hypertension; Male; Mice; Mice, Inbred C57BL; Myocardium; Nephrectomy; Nitric Oxide; Nitric Oxide Synthase; Oxygen; Peptidyl-Dipeptidase A; Superoxides; Ventricular Dysfunction, Left

2010
Inhibition of nitric oxide synthase uncoupling by sepiapterin improves left ventricular function in streptozotocin-induced diabetic mice.
    Clinical and experimental pharmacology & physiology, 2011, Volume: 38, Issue:8

    Topics: Animals; Biopterins; Cardiotonic Agents; Coenzymes; Diabetes Mellitus, Experimental; Diabetic Cardiomyopathies; Enzyme Inhibitors; Lipid Peroxidation; Male; Mice; Mice, Inbred C57BL; Mice, Knockout; Nitric Oxide; Nitric Oxide Synthase; Pterins; Tyrosine; Ventricular Dysfunction, Left; Ventricular Function, Left

2011