sapropterin has been researched along with Diabetic Cardiomyopathies in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 2 (66.67) | 24.3611 |
| 2020's | 1 (33.33) | 2.80 |
| Authors | Studies |
|---|---|
| Cho, KI; Cho, SW; Ha, NC; Han, J; Heo, HJ; Jeong, SH; Jeong, YJ; Kha, PT; Kim, HK; Kim, M; Kim, N; Kim, SW; Ko, KS; Ko, TH; Lee, SR; Minamino, T; Nilius, B; Park, NM; Park, YS; Rhee, BD; Seo, DY; Shimizu, I; Song, IS; Won, JC; Youm, JB | 1 |
| Baumgardt, SL; Bosnjak, ZJ; Fang, J; Ge, ZD; Kersten, JR; Leucker, TM; Paterson, M; Warltier, DC; Zhang, DX | 1 |
| Fujita, M; Iwasaka, T; Jo, F; Jo, H; Kosaki, A; Okazaki, T; Otani, H; Shimazu, T; Yoshioka, K | 1 |
3 other study(ies) available for sapropterin and Diabetic Cardiomyopathies
| Article | Year |
|---|---|
BH4 activates CaMKK2 and rescues the cardiomyopathic phenotype in rodent models of diabetes.
Topics: AMP-Activated Protein Kinases; Animals; Biopterins; Calcium-Calmodulin-Dependent Protein Kinase Kinase; Cyclic AMP Response Element-Binding Protein; Diabetes Mellitus; Diabetic Cardiomyopathies; Heart; Male; Mice; Mice, Inbred C57BL; Mitochondria; Myocardial Contraction; Myocardium; Myocytes, Cardiac; Organelle Biogenesis; Oxidative Phosphorylation; Rats; Rats, Long-Evans; Signal Transduction | 2020 |
Chronic Co-Administration of Sepiapterin and L-Citrulline Ameliorates Diabetic Cardiomyopathy and Myocardial Ischemia/Reperfusion Injury in Obese Type 2 Diabetic Mice.
Topics: Age Factors; Animals; Biopterins; Cardiotonic Agents; Cells, Cultured; Citrulline; Coronary Circulation; Coronary Vessels; Diabetes Mellitus, Type 2; Diabetic Cardiomyopathies; Disease Models, Animal; Drug Administration Schedule; Drug Therapy, Combination; Endothelial Cells; Isolated Heart Preparation; Mice, Inbred C57BL; Mice, Obese; Myocardial Infarction; Myocardial Reperfusion Injury; Myocardium; Nitric Oxide; Nitric Oxide Synthase Type III; Obesity; Phosphorylation; Protein Multimerization; Pterins; Time Factors; Vasodilation; Ventricular Function, Left | 2016 |
Inhibition of nitric oxide synthase uncoupling by sepiapterin improves left ventricular function in streptozotocin-induced diabetic mice.
Topics: Animals; Biopterins; Cardiotonic Agents; Coenzymes; Diabetes Mellitus, Experimental; Diabetic Cardiomyopathies; Enzyme Inhibitors; Lipid Peroxidation; Male; Mice; Mice, Inbred C57BL; Mice, Knockout; Nitric Oxide; Nitric Oxide Synthase; Pterins; Tyrosine; Ventricular Dysfunction, Left; Ventricular Function, Left | 2011 |