Page last updated: 2024-08-21

sapropterin and Cardiomyopathies, Primary

sapropterin has been researched along with Cardiomyopathies, Primary in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's2 (66.67)29.6817
2010's0 (0.00)24.3611
2020's1 (33.33)2.80

Authors

AuthorsStudies
Abdel-Rahman, AA; Yao, F1
Arkenbout, EK; Barker, N; Barouch, LA; Bedja, D; Champion, HC; Channon, KM; Chen, J; Cingolani, O; Gabrielson, KL; Hale, AB; Halushka, MK; Kaminski, PM; Kass, DA; Leyton-Mange, JS; Moens, AL; Niu, X; Wolin, MS; Wuyts, FL; Xia, Y; Yang, R1
Anversa, P; Duan, J; Gao, F; Kajstura, J; Leri, A; Ren, J; Sowers, JR; Sreejayan, N; Thomas, DP; Yang, X1

Other Studies

3 other study(ies) available for sapropterin and Cardiomyopathies, Primary

ArticleYear
Tetrahydrobiopterin paradoxically mediates cardiac oxidative stress and mitigates ethanol-evoked cardiac dysfunction in conscious female rats.
    European journal of pharmacology, 2021, Oct-15, Volume: 909

    Topics: Animals; Biopterins; Cardiomyopathies; Disease Models, Animal; Ethanol; Female; Heart; Humans; Myocardium; Nitric Oxide Synthase Type III; Oxidative Stress; Rats; Reactive Oxygen Species; Sugar Acids

2021
Adverse ventricular remodeling and exacerbated NOS uncoupling from pressure-overload in mice lacking the beta3-adrenoreceptor.
    Journal of molecular and cellular cardiology, 2009, Volume: 47, Issue:5

    Topics: Age Factors; Animals; Biopterins; Blotting, Western; Cardiomyopathies; Echocardiography; Hypertrophy, Left Ventricular; Male; Mice; Mice, Mutant Strains; Nitric Oxide Synthase; Oxidative Stress; Receptors, Adrenergic, beta-3; Superoxides; Vasoconstriction; Ventricular Remodeling

2009
IGF-I alleviates diabetes-induced RhoA activation, eNOS uncoupling, and myocardial dysfunction.
    American journal of physiology. Regulatory, integrative and comparative physiology, 2008, Volume: 294, Issue:3

    Topics: Animals; Biopterins; Cardiomyopathies; Cell Separation; Diabetes Mellitus, Experimental; Folic Acid Antagonists; Insulin-Like Growth Factor I; Kv1.2 Potassium Channel; Mice; Mice, Transgenic; Mitogen-Activated Protein Kinases; Myocardial Contraction; Myocytes, Cardiac; Myosin Heavy Chains; Nitric Oxide Synthase Type III; Oncogene Protein v-akt; Oxidation-Reduction; p38 Mitogen-Activated Protein Kinases; Phosphorylation; rho GTP-Binding Proteins; rho-Associated Kinases; rhoA GTP-Binding Protein; Tetrahydrofolate Dehydrogenase; Ultrasonography

2008