salsoline has been researched along with Neuroblastoma in 11 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 4 (36.36) | 18.2507 |
| 2000's | 6 (54.55) | 29.6817 |
| 2010's | 1 (9.09) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Das, JR; Tizabi, Y | 1 |
| Ramlochansingh, C; Taylor, RE; Tizabi, Y | 1 |
| Akao, Y; Maruyama, W; Nakagawa, Y; Naoi, M; Shamoto-Nagai, M; Shimizu, S; Tsujimoto, Y; Yi, H; Youdim, MB | 1 |
| Iwasa, K; Maruyama, W; Naoi, M; Ohde, H; Shimazu, S; Takahashi, T; Yi, H; Yoneda, F | 1 |
| Akao, Y; Chen, K; Maruyama, W; Naoi, M; Shih, J; Yi, H | 1 |
| Akao, Y; Maruyama, W; Naoi, M; Yamaoka, Y; Yi, H | 1 |
| Dostert, P; Hashizume, Y; Kasamatsu, T; Kohda, K; Maruyama, W; Naoi, M; Takahashi, T | 1 |
| Dostert, P; Kasamatsu, T; Maruyama, W; Naoi, M | 1 |
| Deng, Y; Kawai, M; Maruyama, W; Minami, C; Nakahara, D; Naoi, M; Takahashi, T | 1 |
| Maruyama, W; Naoi, M | 1 |
| Boulton, AA; Davis, BA; Dostert, P; Maruyama, W; Naoi, M | 1 |
11 other study(ies) available for salsoline and Neuroblastoma
| Article | Year |
|---|---|
Additive protective effects of donepezil and nicotine against salsolinol-induced cytotoxicity in SH-SY5Y cells.
Topics: Analysis of Variance; Annexin A5; Apoptosis; Atropine; Cell Cycle; Cell Line, Tumor; Cell Survival; Cholinesterase Inhibitors; Donepezil; Dose-Response Relationship, Drug; Drug Combinations; Drug Interactions; Flow Cytometry; Gene Expression Regulation, Neoplastic; Humans; Indans; Mecamylamine; Neuroblastoma; Nicotine; Nicotinic Agonists; Nicotinic Antagonists; Piperidines; Propidium; Salsoline Alkaloids | 2009 |
Toxic effects of low alcohol and nicotine combinations in SH-SY5Y cells are apoptotically mediated.
Topics: Analysis of Variance; Animals; Apoptosis; Caspase 3; Cell Line, Tumor; Cell Survival; Central Nervous System Depressants; Dose-Response Relationship, Drug; Drug Combinations; Ethanol; Humans; Neuroblastoma; Nicotine; Nicotinic Agonists; Salsoline Alkaloids; Tetrazolium Salts; Thiazoles | 2011 |
Mitochondrial permeability transition mediates apoptosis induced by N-methyl(R)salsolinol, an endogenous neurotoxin, and is inhibited by Bcl-2 and rasagiline, N-propargyl-1(R)-aminoindan.
Topics: Animals; Apoptosis; Cyclosporine; Enzyme Inhibitors; Humans; Indans; Ion Channels; Mitochondria, Liver; Mitochondrial Membrane Transport Proteins; Mitochondrial Permeability Transition Pore; Neuroblastoma; Neuroprotective Agents; Neurotoxins; Permeability; Proto-Oncogene Proteins c-bcl-2; Rats; Salsoline Alkaloids; Tetrahydroisoquinolines; Tumor Cells, Cultured | 2002 |
Neuroprotective function of R-(-)-1-(benzofuran-2-yl)-2-propylaminopentane, [R-(-)-BPAP], against apoptosis induced by N-methyl(R)salsolinol, an endogenous dopaminergic neurotoxin, in human dopaminergic neuroblastoma SH-SY5Y cells.
Topics: Apoptosis; Benzofurans; Electrophoresis, Polyacrylamide Gel; Flow Cytometry; Humans; Neuroblastoma; Neuroprotective Agents; Neurotoxins; Proto-Oncogene Proteins c-bcl-2; Receptors, Dopamine; Salsoline Alkaloids; Structure-Activity Relationship; Tetrahydroisoquinolines; Tumor Cells, Cultured | 2004 |
Type A monoamine oxidase is the target of an endogenous dopaminergic neurotoxin, N-methyl(R)salsolinol, leading to apoptosis in SH-SY5Y cells.
Topics: Apoptosis; Binding Sites; Cell Line, Tumor; DNA; Gene Silencing; Humans; Kinetics; Membrane Potentials; Mitochondria; Monoamine Oxidase; Monoamine Oxidase Inhibitors; Neuroblastoma; Neurotoxins; RNA, Small Interfering; Salsoline Alkaloids; Tetrahydroisoquinolines; Transfection | 2006 |
Involvement of type A monoamine oxidase in neurodegeneration: regulation of mitochondrial signaling leading to cell death or neuroprotection.
Topics: Cell Death; Cell Line, Tumor; Dopamine; Dose-Response Relationship, Drug; Drug Interactions; Enzyme Activation; Enzyme Inhibitors; Gene Expression Regulation; Humans; Indans; Mitochondria; Mitochondrial Membranes; Monoamine Oxidase; Nerve Degeneration; Neuroblastoma; Proto-Oncogene Proteins c-bcl-2; RNA, Small Interfering; Salsoline Alkaloids; Serotonin; Signal Transduction; Tetrahydroisoquinolines; Transfection | 2006 |
An endogenous dopaminergic neurotoxin, N-methyl-(R)-salsolinol, induces DNA damage in human dopaminergic neuroblastoma SH-SY5Y cells.
Topics: Antioxidants; Apoptosis; Carcinogens; Catalase; Cycloheximide; DNA Damage; Dopamine; Free Radicals; Humans; Microscopy, Fluorescence; Neuroblastoma; Neuroprotective Agents; Neurotoxins; Oxidation-Reduction; Parkinson Disease; Protein Synthesis Inhibitors; Salsoline Alkaloids; Selegiline; Semicarbazides; Stereoisomerism; Tetrahydroisoquinolines; Tumor Cells, Cultured | 1997 |
Oxidation of N-methyl(R)salsolinol: involvement to neurotoxicity and neuroprotection by endogenous catechol isoquinolines.
Topics: 3,4-Dihydroxyphenylacetic Acid; Animals; Antioxidants; Corpus Striatum; Cycloheximide; Disease Models, Animal; DNA Damage; Dopamine; Homovanillic Acid; Humans; Hydroxyl Radical; Isoquinolines; Male; Necrosis; Neuroblastoma; Neurons; Neurotoxins; Oxidative Stress; Parkinson Disease, Secondary; Posture; Rats; Rats, Wistar; Salsoline Alkaloids; Stereoisomerism; Substantia Nigra; Tetrahydroisoquinolines; Tumor Cells, Cultured | 1998 |
Generation of reactive oxygen species accounts for cytotoxicity of an endogenous dopaminergic neurotoxin, (R)-N-methylsalsolinol, to differentiated dopaminergic SH-SY5Y cells.
Topics: Cell Differentiation; Cell Survival; Dopamine; Humans; Hydroxyl Radical; Isoquinolines; Neuroblastoma; Neurotoxins; Oxidation-Reduction; Reactive Oxygen Species; Salsoline Alkaloids; Tetrahydroisoquinolines; Tumor Cells, Cultured | 1998 |
Cell death of dopamine neurons in aging and Parkinson's disease.
Topics: Adult; Aged; Aging; Apoptosis; Brain; Cerebrospinal Fluid; DNA Damage; DNA, Neoplasm; Female; Humans; Male; Methyltransferases; Middle Aged; Neuroblastoma; Neurons; Neurotoxins; Oxidative Stress; Parkinson Disease; Reactive Oxygen Species; Receptors, Dopamine; Salsoline Alkaloids; Tetrahydroisoquinolines; Tumor Cells, Cultured | 1999 |
Enantio-specific induction of apoptosis by an endogenous neurotoxin, N-methyl(R)salsolinol, in dopaminergic SH-SY5Y cells: suppression of apoptosis by N-(2-heptyl)-N-methylpropargylamine.
Topics: Alkynes; Apoptosis; Dopamine; Drug Interactions; Fluorescent Dyes; Humans; Membrane Potentials; Mitochondria; Monoamine Oxidase Inhibitors; Neuroblastoma; Neurons; Neuroprotective Agents; Neurotoxins; Parkinsonian Disorders; Salsoline Alkaloids; Signal Transduction; Stereoisomerism; Substantia Nigra; Tetrahydroisoquinolines; Tumor Cells, Cultured | 2001 |