salicylates has been researched along with Sleep-Wake-Disorders* in 3 studies
2 review(s) available for salicylates and Sleep-Wake-Disorders
Article | Year |
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The difficult asthmatic.
A poor therapeutic response may be explained by incomplete or erroneous diagnostic assessment, by failure to employ optimal drug doses and combinations, or by inadequate attention to the non-pharmacologic aspects of management. Poor compliance and counterproductive patient attitudes may need to be addressed. These problems and the approach to asthma concomitant with other diseases are discussed. Topics: Asthma; Diabetes Complications; Diagnosis, Differential; Dyspnea, Paroxysmal; Female; Gastroesophageal Reflux; Heart Diseases; Humans; Hyperventilation; Occupational Diseases; Patient Compliance; Physician-Patient Relations; Pregnancy; Pregnancy Complications; Respiratory Tract Diseases; Respiratory Tract Infections; Salicylates; Sleep Wake Disorders; Surgical Procedures, Operative; Sympathomimetics; Theophylline; Thyroid Diseases | 1984 |
The treatment of teething in infants. A review.
Topics: Acetaminophen; Alcohols; Aspirin; Feeding and Eating Disorders; Gels; Humans; Hypnotics and Sedatives; Infant; Lidocaine; Mouth Mucosa; Salicylates; Sleep Wake Disorders; Tooth Eruption | 1972 |
1 other study(ies) available for salicylates and Sleep-Wake-Disorders
Article | Year |
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The effect of some bacterial products on temperature and sleep in rat.
The lipopolysaccharides from P. aeruginosa, S. minnesota and mucopeptide from Streptococcus group A injected intravenously into rats induce a dose-dependent changes of temperature. Simultaneously, a profound disturbance of sleep occurs. The administration of salicylate, which markedly suppressed the fever does not influence the mucopeptide-caused sleep disturbance. The most prominent change in the sleep pattern is a marked decrease of the total time of paradoxical sleep. The measurement of turnover rates of 5-hydroxytryptamine (5-HT) and noradrenaline (NA) in hypothalamus and midbrain, areas involved in temperature and sleep control, after injection of streptococcal mucopeptide demonstrated a significant increase of 5-HT turnover in both areas during fever and paradoxical sleep deprivation. Small electrolytic lesions of the dorsal raphe nuclei which are the largest collection of neural cells containing 5-HT completely eliminated the pyrogenic potency of mucopeptide. The findings suggest that some bacterial products might increase the body temperature through the interference with activity of 5-HT-containing neurons of the raphe complex. Topics: Body Temperature; Fever; Humans; Hypothalamus; Lipopolysaccharides; Mucoproteins; Neurons; Norepinephrine; Pseudomonas aeruginosa; Salicylates; Salmonella; Serotonin; Sleep Stages; Sleep Wake Disorders; Streptococcus pyogenes | 1975 |