s-nitroso-n-acetylpenicillamine has been researched along with Hypertrophy in 4 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 2 (50.00) | 29.6817 |
2010's | 2 (50.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Chuang, LY; Guh, JY; Huang, JS; Hwang, JY; Kan, WC; Liu, YB; Yang, YL | 1 |
Chuang, LY; Guh, JY; Huang, JS; Huang, YJ | 1 |
Anderson, HD; Kardami, E; Louis, XL; Netticadan, T; Stringer, DM; Taylor, C; Thandapilly, SJ; Wigle, J; Yang, T; Yu, L; Zahradka, P; Zhang, S | 1 |
Bouallegue, A; Daou, GB; Srivastava, AK | 1 |
4 other study(ies) available for s-nitroso-n-acetylpenicillamine and Hypertrophy
Article | Year |
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Angiotensin-converting enzyme inhibitors attenuated advanced glycation end products-induced renal tubular hypertrophy via enhancing nitric oxide signaling.
Topics: Angiotensin-Converting Enzyme Inhibitors; Captopril; Cell Enlargement; Cell Line; Cyclic GMP; Cyclic GMP-Dependent Protein Kinases; Enalapril; Enzyme Activation; Glycation End Products, Advanced; Humans; Hypertrophy; Kidney Tubules, Proximal; MAP Kinase Signaling System; Nitric Oxide; Nitric Oxide Donors; Nitric Oxide Synthase Type II; S-Nitroso-N-Acetylpenicillamine; Signal Transduction; Thionucleotides | 2019 |
Effects of nitric oxide and antioxidants on advanced glycation end products-induced hypertrophic growth in human renal tubular cells.
Topics: Antioxidants; Blotting, Western; Cell Line; Cell Size; Cyclic GMP; Cyclic GMP-Dependent Protein Kinases; Fibronectins; Flow Cytometry; Glycation End Products, Advanced; Humans; Hypertrophy; Kidney Tubules; Nitric Oxide; Nitric Oxide Donors; Nitric Oxide Synthase Type II; Oncogene Protein p21(ras); Receptor for Advanced Glycation End Products; Receptors, Immunologic; S-Nitroso-N-Acetylpenicillamine | 2009 |
Resveratrol prevents norepinephrine induced hypertrophy in adult rat cardiomyocytes, by activating NO-AMPK pathway.
Topics: Adenylate Kinase; Animals; Cell Size; Hypertrophy; Male; Myocytes, Cardiac; NG-Nitroarginine Methyl Ester; Niacinamide; Nitric Oxide; Norepinephrine; Protein Biosynthesis; Pyrazoles; Pyrimidines; Rats; Rats, Inbred SHR; Rats, Sprague-Dawley; Resveratrol; S-Nitroso-N-Acetylpenicillamine; Signal Transduction; Stilbenes | 2011 |
Nitric oxide attenuates endothelin-1-induced activation of ERK1/2, PKB, and Pyk2 in vascular smooth muscle cells by a cGMP-dependent pathway.
Topics: Animals; Aorta, Thoracic; Cell Proliferation; Cells, Cultured; Cyclic GMP; Dose-Response Relationship, Drug; Endothelin-1; Enzyme Inhibitors; Focal Adhesion Kinase 2; Guanylate Cyclase; Hypertrophy; Leucine; Mitogen-Activated Protein Kinase 1; Mitogen-Activated Protein Kinase 3; Muscle, Smooth, Vascular; Myocytes, Smooth Muscle; NG-Nitroarginine Methyl Ester; Nitric Oxide; Nitric Oxide Donors; Nitric Oxide Synthase; Nitroprusside; Oxadiazoles; Phosphorylation; Protein Biosynthesis; Proto-Oncogene Proteins c-akt; Quinoxalines; Rats; Receptors, Cytoplasmic and Nuclear; S-Nitroso-N-Acetylpenicillamine; Signal Transduction; Soluble Guanylyl Cyclase | 2007 |