s-nitroso-n-acetylpenicillamine has been researched along with Acute Confusional Senile Dementia in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 1 (33.33) | 18.2507 |
| 2000's | 1 (33.33) | 29.6817 |
| 2010's | 1 (33.33) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Chakroborty, S; Kim, J; Schneider, C; Stutzmann, GE; West, AR | 1 |
| Anrather, J; Carlson, GA; Frys, K; Iadecola, C; Park, L; Pitstick, R; Younkin, S; Zhou, P | 1 |
| Alberici, AC; Benussi, L; Binetti, G; Gasparini, L; Ghidoni, R; Growdon, JH; Moratto, D; Nitsch, RM; Trabucchi, M | 1 |
3 other study(ies) available for s-nitroso-n-acetylpenicillamine and Acute Confusional Senile Dementia
| Article | Year |
|---|---|
Nitric oxide signaling is recruited as a compensatory mechanism for sustaining synaptic plasticity in Alzheimer's disease mice.
Topics: Alzheimer Disease; Amyloid beta-Protein Precursor; Animals; Disease Models, Animal; Enzyme Inhibitors; Excitatory Postsynaptic Potentials; Gene Expression Regulation; Hippocampus; Humans; In Vitro Techniques; Mice; Mice, Transgenic; Neuronal Plasticity; NG-Nitroarginine Methyl Ester; Nitric Oxide; Nitric Oxide Donors; Presenilin-1; Ryanodine Receptor Calcium Release Channel; S-Nitroso-N-Acetylpenicillamine; Signal Transduction; tau Proteins | 2015 |
NADPH-oxidase-derived reactive oxygen species mediate the cerebrovascular dysfunction induced by the amyloid beta peptide.
Topics: Acetylcholine; Adenosine; Alzheimer Disease; Amino Acid Sequence; Amyloid beta-Peptides; Amyloid beta-Protein Precursor; Animals; Bradykinin; Calcimycin; Cerebrovascular Circulation; Female; Glycoproteins; Humans; Hyperemia; Laser-Doppler Flowmetry; Male; Membrane Glycoproteins; Mice; Mice, Inbred C57BL; Mice, Transgenic; Molecular Sequence Data; NADPH Oxidase 2; NADPH Oxidases; Nerve Tissue Proteins; Nitric Oxide Donors; Oxidative Stress; Peptide Fragments; Phenanthridines; Reactive Oxygen Species; S-Nitroso-N-Acetylpenicillamine; Vibrissae; X Chromosome | 2005 |
Modulation of presenilin-1 processing by nitric oxide during apoptosis induced by serum withdrawal and glucose deprivation.
Topics: Alzheimer Disease; Apoptosis; Benzoates; Culture Media, Serum-Free; Cytochrome c Group; Deoxyglucose; Glioma; Glucose; Humans; Hypoglycemia; Imidazoles; Membrane Proteins; Mitochondria; Nitric Oxide; Nitric Oxide Donors; Nitroprusside; Penicillamine; Presenilin-1; Protein Processing, Post-Translational; S-Nitroso-N-Acetylpenicillamine; Tumor Cells, Cultured | 1999 |