Page last updated: 2024-11-04

s-methylisothiopseudouronium and Brain Edema

s-methylisothiopseudouronium has been researched along with Brain Edema in 1 studies

S-methylisothiopseudouronium: inhibits nitric oxide synthase; structure in first source

Brain Edema: Increased intracellular or extracellular fluid in brain tissue. Cytotoxic brain edema (swelling due to increased intracellular fluid) is indicative of a disturbance in cell metabolism, and is commonly associated with hypoxic or ischemic injuries (see HYPOXIA, BRAIN). An increase in extracellular fluid may be caused by increased brain capillary permeability (vasogenic edema), an osmotic gradient, local blockages in interstitial fluid pathways, or by obstruction of CSF flow (e.g., obstructive HYDROCEPHALUS). (From Childs Nerv Syst 1992 Sep; 8(6):301-6)

Research

Studies (1)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (100.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Takemori, K1
Ito, H1
Suzuki, T1

Other Studies

1 other study available for s-methylisothiopseudouronium and Brain Edema

ArticleYear
Effects of inducible nitric oxide synthase inhibition on cerebral edema in severe hypertension.
    Acta neurochirurgica. Supplement, 2000, Volume: 76

    Topics: Animals; Brain Edema; Cerebral Cortex; Fibrinogen; Glial Fibrillary Acidic Protein; Hypertension; In

2000