s-(1,2-dichlorovinyl)cysteine has been researched along with ADPKD in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 3 (100.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| 't Hoen, PAC; Balog, J; Formica, C; Malas, T; Peters, DJM; Verburg, L | 1 |
| Formica, C; Happé, H; McNeill, H; Peters, DJ; Scharpfenecker, M; Veraar, KA; Vortkamp, A | 1 |
| Breuning, MH; de Heer, E; Happé, H; Kunnen, SJ; Leonhard, WN; Peters, DJ; van der Wal, AM | 1 |
3 other study(ies) available for s-(1,2-dichlorovinyl)cysteine and ADPKD
| Article | Year |
|---|---|
Characterisation of transcription factor profiles in polycystic kidney disease (PKD): identification and validation of STAT3 and RUNX1 in the injury/repair response and PKD progression.
Topics: Animals; Chromatin Immunoprecipitation; Core Binding Factor Alpha 2 Subunit; Cysteine; Disease Models, Animal; Disease Progression; Epithelial Cells; Gene Expression Regulation; Humans; Kidney; Male; Mice; Mice, Transgenic; Polycystic Kidney, Autosomal Dominant; Promoter Regions, Genetic; Protein Binding; STAT3 Transcription Factor; Transcription Factors; TRPP Cation Channels | 2019 |
Four-jointed knock-out delays renal failure in an ADPKD model with kidney injury.
Topics: Acute Kidney Injury; Animals; Cysteine; Disease Models, Animal; Disease Progression; Fibrosis; Inflammation Mediators; Intercellular Signaling Peptides and Proteins; Kidney; Kidney Failure, Chronic; Male; Mice, Knockout; Mutation; Polycystic Kidney, Autosomal Dominant; Time Factors; TRPP Cation Channels; Wnt Signaling Pathway | 2019 |
Altered Hippo signalling in polycystic kidney disease.
Topics: Animals; Cell Nucleus; Cysteine; Disease Models, Animal; Disease Progression; Epithelial Cells; Humans; Kidney; Kidney Tubules; Mice; Mice, Knockout; Polycystic Kidney, Autosomal Dominant; Polycystic Kidney, Autosomal Recessive; Proto-Oncogene Proteins c-yes; Regeneration; Signal Transduction; Transcriptional Activation; TRPP Cation Channels; Up-Regulation | 2011 |