rosiglitazone has been researched along with Precancerous Conditions in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 3 (100.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Herrigel, DJ; Moss, RA | 1 |
| Andrisani, OM; Bi, P; Bidwell, CA; Castro, B; Durkes, A; Elzey, BD; Freeman, JL; Karki, A; Konieczny, SF; Kuang, S; Wang, C; Wirbisky, SE; Yue, F | 1 |
| Balkwill, FR; Bossard, M; Candido, JB; Cook, N; Emami-Shahri, N; Hagemann, T; Maniati, E; Nedospasov, SA; Tuveson, DA | 1 |
1 review(s) available for rosiglitazone and Precancerous Conditions
| Article | Year |
|---|---|
Diabetes mellitus as a novel risk factor for gastrointestinal malignancies.
Topics: Adenocarcinoma; Carcinoma, Hepatocellular; Colorectal Neoplasms; Diabetes Complications; Humans; Hypoglycemic Agents; Insulin; Liver Neoplasms; Metformin; Non-alcoholic Fatty Liver Disease; Pancreatic Neoplasms; Pioglitazone; Precancerous Conditions; Receptors, Somatomedin; Risk Factors; Rosiglitazone; Thiazolidinediones | 2014 |
2 other study(ies) available for rosiglitazone and Precancerous Conditions
| Article | Year |
|---|---|
Notch activation drives adipocyte dedifferentiation and tumorigenic transformation in mice.
Topics: Adipocytes; Animals; Biomarkers, Tumor; Cell Dedifferentiation; Cell Differentiation; Cell Lineage; Cell Proliferation; Cell Transformation, Neoplastic; Diabetes Mellitus, Experimental; Diamines; Dibenzazepines; Gene Deletion; Gene Expression Profiling; Gene Expression Regulation, Neoplastic; Humans; Ligands; Lipid Metabolism; Liposarcoma; Metabolic Syndrome; Mice, Inbred C57BL; PPAR gamma; Precancerous Conditions; PTEN Phosphohydrolase; Receptors, Notch; Rosiglitazone; Signal Transduction; Thiazoles; Thiazolidinediones; Xenograft Model Antitumor Assays | 2016 |
Crosstalk between the canonical NF-κB and Notch signaling pathways inhibits Pparγ expression and promotes pancreatic cancer progression in mice.
Topics: Animals; Basic Helix-Loop-Helix Transcription Factors; Carcinoma, Pancreatic Ductal; Disease Progression; Down-Regulation; Gene Expression Regulation, Neoplastic; Genes, ras; Histones; Homeodomain Proteins; I-kappa B Kinase; Inflammation; Mice; Mice, Inbred C57BL; Neoplasm Proteins; NF-kappa B; Pancreatic Diseases; Pancreatic Neoplasms; Phosphorylation; PPAR gamma; Precancerous Conditions; Protein Processing, Post-Translational; Proto-Oncogene Mas; Receptors, Notch; RNA, Small Interfering; Rosiglitazone; Signal Transduction; Thiazolidinediones; Transcription Factor HES-1; Tumor Necrosis Factor-alpha | 2011 |