rosiglitazone has been researched along with Dunnigan Syndrome in 4 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (25.00) | 29.6817 |
| 2010's | 3 (75.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Adams, C; Agostini, M; Beig, J; Blom, DJ; Chatterjee, K; Fairall, L; Majithia, AR; Marais, AD; Murphy, R; Muskett, FW; Nagy, L; O'Rahilly, S; Rajanayagam, O; Savage, DB; Schoenmakers, E; Schwabe, JWR; Semple, RK; Szatmari, I | 1 |
| Hodnett, PA; Maher, MM; McLaughlin, PD; O'Halloran, D; Ryan, J | 1 |
| Auclair, M; Boccara, F; Capeau, J; Capel, E; Caron-Debarle, M; Guerci, B; Lascols, O; Vigeral, C; Vigouroux, C | 1 |
| Buettner, J; Lüdtke, A; Schmidt, HH; Worman, HJ | 1 |
4 other study(ies) available for rosiglitazone and Dunnigan Syndrome
| Article | Year |
|---|---|
A Pharmacogenetic Approach to the Treatment of Patients With
Topics: Adolescent; Adult; Amino Acid Substitution; Binding Sites; Female; Gene Expression Regulation; Genes, Reporter; HEK293 Cells; Humans; Hypoglycemic Agents; Ligands; Lipodystrophy, Familial Partial; Models, Molecular; Molecular Conformation; Molecular Docking Simulation; Mutation, Missense; Pharmacogenetics; Pioglitazone; PPAR gamma; Protein Conformation; Recombinant Proteins; Rosiglitazone; Thiazolidinediones; Young Adult | 2018 |
Quantitative whole-body MRI in familial partial lipodystrophy type 2: changes in adipose tissue distribution coincide with biochemical improvement.
Topics: Adipose Tissue; Adolescent; Female; Humans; Hypoglycemic Agents; Insulin Resistance; Lipodystrophy, Familial Partial; Magnetic Resonance Imaging; Rosiglitazone; Thiazolidinediones; Whole Body Imaging | 2012 |
Peroxisome proliferator-activated receptor-γ mutations responsible for lipodystrophy with severe hypertension activate the cellular renin-angiotensin system.
Topics: Adult; Aged; Amino Acid Sequence; Angiotensin II; Anilides; Animals; Case-Control Studies; Enzyme Activation; Female; Fibroblasts; Genetic Predisposition to Disease; HEK293 Cells; Humans; Hypertension; Inflammation Mediators; Leukocytes, Mononuclear; Lipodystrophy, Familial Partial; Male; Mitogen-Activated Protein Kinases; Molecular Sequence Data; Muscle, Smooth, Vascular; Mutation; Myocytes, Smooth Muscle; Oxidative Stress; Phenotype; PPAR gamma; Receptor, Angiotensin, Type 1; Renin-Angiotensin System; RNA Interference; Rosiglitazone; Severity of Illness Index; Skin; Thiazolidinediones; Transfection; Young Adult | 2013 |
New PPARG mutation leads to lipodystrophy and loss of protein function that is partially restored by a synthetic ligand.
Topics: Adolescent; Adult; Amino Acid Substitution; Animals; Cell Line; Female; Genes, Reporter; Humans; Lipodystrophy, Familial Partial; Metabolic Syndrome; Mutation, Missense; Pedigree; Point Mutation; PPAR gamma; Protein Structure, Tertiary; Rosiglitazone; Thiazolidinediones; Transcription, Genetic; Transfection | 2007 |