ro 31-8425 has been researched along with Necrosis in 1 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 1 (100.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Gaarde, WA; Han, D; Johnson, HS; Kaplowitz, N; Saberi, B; Ybanez, MD | 1 |
1 other study(ies) available for ro 31-8425 and Necrosis
Article | Year |
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Protein kinase C (PKC) participates in acetaminophen hepatotoxicity through c-jun-N-terminal kinase (JNK)-dependent and -independent signaling pathways.
Topics: Acetaminophen; AMP-Activated Protein Kinases; Animals; Cells, Cultured; Chemical and Drug Induced Liver Injury; Disease Models, Animal; Hepatocytes; In Vitro Techniques; Indoles; JNK Mitogen-Activated Protein Kinases; Male; Maleimides; Mice; Mice, Inbred C57BL; Mitochondria, Liver; Necrosis; Protein Kinase C; Protein Kinase Inhibitors; Signal Transduction | 2014 |