ro 31-8425 and Necrosis

ro 31-8425 has been researched along with Necrosis in 1 studies

Research

Studies (1)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's1 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Gaarde, WA; Han, D; Johnson, HS; Kaplowitz, N; Saberi, B; Ybanez, MD1

Other Studies

1 other study(ies) available for ro 31-8425 and Necrosis

ArticleYear
Protein kinase C (PKC) participates in acetaminophen hepatotoxicity through c-jun-N-terminal kinase (JNK)-dependent and -independent signaling pathways.
    Hepatology (Baltimore, Md.), 2014, Volume: 59, Issue:4

    Topics: Acetaminophen; AMP-Activated Protein Kinases; Animals; Cells, Cultured; Chemical and Drug Induced Liver Injury; Disease Models, Animal; Hepatocytes; In Vitro Techniques; Indoles; JNK Mitogen-Activated Protein Kinases; Male; Maleimides; Mice; Mice, Inbred C57BL; Mitochondria, Liver; Necrosis; Protein Kinase C; Protein Kinase Inhibitors; Signal Transduction

2014