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ro 31-8220 and Myotonic Dystrophy

ro 31-8220 has been researched along with Myotonic Dystrophy in 2 studies

Ro 31-8220: a protein kinase C inhibitor

Myotonic Dystrophy: Neuromuscular disorder characterized by PROGRESSIVE MUSCULAR ATROPHY; MYOTONIA, and various multisystem atrophies. Mild INTELLECTUAL DISABILITY may also occur. Abnormal TRINUCLEOTIDE REPEAT EXPANSION in the 3' UNTRANSLATED REGIONS of DMPK PROTEIN gene is associated with Myotonic Dystrophy 1. DNA REPEAT EXPANSION of zinc finger protein-9 gene intron is associated with Myotonic Dystrophy 2.

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's2 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Ketley, A1
Chen, CZ1
Li, X1
Arya, S1
Robinson, TE1
Granados-Riveron, J1
Udosen, I1
Morris, GE1
Holt, I1
Furling, D1
Chaouch, S1
Haworth, B1
Southall, N1
Shinn, P1
Zheng, W1
Austin, CP1
Hayes, CJ1
Brook, JD1
Zhao, Y1
Koebis, M1
Suo, S1
Ohno, S1
Ishiura, S1

Other Studies

2 other studies available for ro 31-8220 and Myotonic Dystrophy

ArticleYear
High-content screening identifies small molecules that remove nuclear foci, affect MBNL distribution and CELF1 protein levels via a PKC-independent pathway in myotonic dystrophy cell lines.
    Human molecular genetics, 2014, Mar-15, Volume: 23, Issue:6

    Topics: Alternative Splicing; Animals; CELF1 Protein; Cell Nucleus; Cells, Cultured; Chromomycin A3; Disease

2014
Regulation of the alternative splicing of sarcoplasmic reticulum Ca²⁺-ATPase1 (SERCA1) by phorbol 12-myristate 13-acetate (PMA) via a PKC pathway.
    Biochemical and biophysical research communications, 2012, Jun-29, Volume: 423, Issue:2

    Topics: Alternative Splicing; Exons; HEK293 Cells; Humans; Indoles; Myotonic Dystrophy; Protein Kinase C; Pr

2012