ro 31-8220 has been researched along with Congenital Myotonic Dystrophy in 2 studies
Ro 31-8220: a protein kinase C inhibitor
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 2 (100.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Ketley, A | 1 |
Chen, CZ | 1 |
Li, X | 1 |
Arya, S | 1 |
Robinson, TE | 1 |
Granados-Riveron, J | 1 |
Udosen, I | 1 |
Morris, GE | 1 |
Holt, I | 1 |
Furling, D | 1 |
Chaouch, S | 1 |
Haworth, B | 1 |
Southall, N | 1 |
Shinn, P | 1 |
Zheng, W | 1 |
Austin, CP | 1 |
Hayes, CJ | 1 |
Brook, JD | 1 |
Zhao, Y | 1 |
Koebis, M | 1 |
Suo, S | 1 |
Ohno, S | 1 |
Ishiura, S | 1 |
2 other studies available for ro 31-8220 and Congenital Myotonic Dystrophy
Article | Year |
---|---|
High-content screening identifies small molecules that remove nuclear foci, affect MBNL distribution and CELF1 protein levels via a PKC-independent pathway in myotonic dystrophy cell lines.
Topics: Alternative Splicing; Animals; CELF1 Protein; Cell Nucleus; Cells, Cultured; Chromomycin A3; Disease | 2014 |
Regulation of the alternative splicing of sarcoplasmic reticulum Ca²⁺-ATPase1 (SERCA1) by phorbol 12-myristate 13-acetate (PMA) via a PKC pathway.
Topics: Alternative Splicing; Exons; HEK293 Cells; Humans; Indoles; Myotonic Dystrophy; Protein Kinase C; Pr | 2012 |