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ro 31-8220 and Congenital Myotonic Dystrophy

ro 31-8220 has been researched along with Congenital Myotonic Dystrophy in 2 studies

Ro 31-8220: a protein kinase C inhibitor

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's2 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Ketley, A1
Chen, CZ1
Li, X1
Arya, S1
Robinson, TE1
Granados-Riveron, J1
Udosen, I1
Morris, GE1
Holt, I1
Furling, D1
Chaouch, S1
Haworth, B1
Southall, N1
Shinn, P1
Zheng, W1
Austin, CP1
Hayes, CJ1
Brook, JD1
Zhao, Y1
Koebis, M1
Suo, S1
Ohno, S1
Ishiura, S1

Other Studies

2 other studies available for ro 31-8220 and Congenital Myotonic Dystrophy

ArticleYear
High-content screening identifies small molecules that remove nuclear foci, affect MBNL distribution and CELF1 protein levels via a PKC-independent pathway in myotonic dystrophy cell lines.
    Human molecular genetics, 2014, Mar-15, Volume: 23, Issue:6

    Topics: Alternative Splicing; Animals; CELF1 Protein; Cell Nucleus; Cells, Cultured; Chromomycin A3; Disease

2014
Regulation of the alternative splicing of sarcoplasmic reticulum Ca²⁺-ATPase1 (SERCA1) by phorbol 12-myristate 13-acetate (PMA) via a PKC pathway.
    Biochemical and biophysical research communications, 2012, Jun-29, Volume: 423, Issue:2

    Topics: Alternative Splicing; Exons; HEK293 Cells; Humans; Indoles; Myotonic Dystrophy; Protein Kinase C; Pr

2012