rhodioloside has been researched along with Infarction, Middle Cerebral Artery in 11 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 5 (45.45) | 24.3611 |
| 2020's | 6 (54.55) | 2.80 |
| Authors | Studies |
|---|---|
| Jia, Y; Li, F; Lv, X; Mao, Q; Wang, J; Wu, B; Yan, T; Zhang, X | 1 |
| Chi, J; Dai, H; Li, C; Liang, M; Tian, S; Wang, Y; Xu, H; Zhu, H | 1 |
| Cui, Q; Ding, X; Lin, G; Sang, N; Tan, J; Wang, F; Xu, J; Xu, W; You, S; Zhan, C; Zhang, J; Zhang, P; Zhu, Y | 1 |
| Brown, J; Chu, K; Hong, G; Huang, X; Lai, W; Su, Y; Wang, Y | 1 |
| Ding, D; Hua, X; Li, S; Lu, Y; Ma, Z; Xing, X; Xu, J | 1 |
| Chen, Q; Chen, W; Dong, C; Dong, W; Gong, T; Han, P; Liu, W; Liu, X; Sun, S; Wen, S; Zhao, S | 1 |
| Brown, J; Chen, L; Chu, K; Hong, G; Lai, W; Wang, Y; Xie, X; Zhang, X | 1 |
| Hu, X; Mei, D; Yan, F; Zhang, B; Zuo, W | 1 |
| Brown, J; Chen, L; Chu, K; Hong, G; Lai, W; Xu, L; Ying, X; Zhang, X | 1 |
| Brown, J; Chen, L; Chu, K; Hong, G; Lai, W; Wei, Y; Zhang, X; Zheng, Z | 1 |
| Feng, SF; Li, XQ; Liu, SB; Shi, TY; Tian, Z; Wu, YM; Xing, JH; Zhang, N; Zhao, MG | 1 |
11 other study(ies) available for rhodioloside and Infarction, Middle Cerebral Artery
| Article | Year |
|---|---|
Salidroside inhibited cerebral ischemia/reperfusion-induced oxidative stress and apoptosis via Nrf2/Trx1 signaling pathway.
Topics: Animals; Antioxidants; Apoptosis; Brain Ischemia; Humans; Infarction, Middle Cerebral Artery; Mitogen-Activated Protein Kinases; NF-E2-Related Factor 2; Oxidative Stress; Rats; Reperfusion; Reperfusion Injury; Signal Transduction; Thioredoxins | 2022 |
Salidroside attenuates cerebral ischemia/reperfusion injury by regulating TSC2-induced autophagy.
Topics: AMP-Activated Protein Kinases; Animals; Apoptosis; Autophagy; Brain Ischemia; Infarction, Middle Cerebral Artery; Mammals; Rats; Reperfusion Injury; TOR Serine-Threonine Kinases | 2023 |
Multi-pathway neuroprotective effects of a novel salidroside derivative SHPL-49 against acute cerebral ischemic injury.
Topics: Animals; Apoptosis; bcl-2-Associated X Protein; Brain Injuries; Brain Ischemia; Calcium; Infarction, Middle Cerebral Artery; Ischemia; Neuroprotective Agents; Oxidative Stress; Proto-Oncogene Proteins c-bcl-2; Rats; Reperfusion Injury | 2023 |
Salidroside Restores an Anti-inflammatory Endothelial Phenotype by Selectively Inhibiting Endothelial Complement After Oxidative Stress.
Topics: Animals; Anti-Inflammatory Agents; Apoptosis Regulatory Proteins; Brain; Cell Line; Coculture Techniques; Complement Activation; Complement C3; Complement Inactivating Agents; Disease Models, Animal; Endothelial Cells; Glucosides; Human Umbilical Vein Endothelial Cells; Humans; Infarction, Middle Cerebral Artery; Inflammation Mediators; Male; Mice; Oxidative Stress; Phenols; Phenotype; Rats, Sprague-Dawley; Reperfusion Injury; Signal Transduction | 2020 |
Fibroblast growth factor 2 contributes to the effect of salidroside on dendritic and synaptic plasticity after cerebral ischemia/reperfusion injury.
Topics: Animals; Apoptosis; Cyclic AMP-Dependent Protein Kinases; Dendrites; Fibroblast Growth Factor 2; Glucosides; Infarction, Middle Cerebral Artery; Inflammation; Male; Neuronal Plasticity; Neuroprotective Agents; Phenols; Rats; Rats, Sprague-Dawley; Reperfusion Injury; Signal Transduction | 2020 |
Salidroside Inhibits Reactive Astrogliosis and Glial Scar Formation in Late Cerebral Ischemia via the Akt/GSK-3β Pathway.
Topics: Animals; Astrocytes; Brain; Brain Ischemia; Cell Proliferation; Gliosis; Glucosides; Glycogen Synthase Kinase 3 beta; Infarction, Middle Cerebral Artery; Male; Mice, Inbred C57BL; Neuroprotective Agents; Phenols; Proto-Oncogene Proteins c-akt; Signal Transduction | 2021 |
Inhibition of Complement Drives Increase in Early Growth Response Proteins and Neuroprotection Mediated by Salidroside After Cerebral Ischemia.
Topics: Animals; Brain Ischemia; Complement C3; Complement Inactivator Proteins; Complement System Proteins; Early Growth Response Transcription Factors; Glucosides; Infarction, Middle Cerebral Artery; Neuroprotection; Phenols; Rats; Reperfusion Injury; Time Factors | 2018 |
Salidroside improves brain ischemic injury by activating PI3K/Akt pathway and reduces complications induced by delayed tPA treatment.
Topics: Animals; Brain; Cell Survival; Cells, Cultured; Endothelial Cells; Glucosides; Humans; Infarction, Middle Cerebral Artery; Male; Neuroprotective Agents; Phenols; Phosphatidylinositol 3-Kinases; Proto-Oncogene Proteins c-akt; Rats, Sprague-Dawley; Reactive Oxygen Species; Signal Transduction; Tissue Plasminogen Activator | 2018 |
Salidroside Reduces Inflammation and Brain Injury After Permanent Middle Cerebral Artery Occlusion in Rats by Regulating PI3K/PKB/Nrf2/NFκB Signaling Rather than Complement C3 Activity.
Topics: Animals; Brain Injuries; Brain Ischemia; Complement C3; Glucosides; Infarction, Middle Cerebral Artery; Inflammation; Neuroprotection; NF-E2-Related Factor 2; Phenols; Phosphatidylinositol 3-Kinases; Rats; Signal Transduction | 2019 |
Salidroside-Mediated Neuroprotection is Associated with Induction of Early Growth Response Genes (Egrs) Across a Wide Therapeutic Window.
Topics: Animals; Apoptosis; bcl-2-Associated X Protein; bcl-X Protein; Brain; Brain Ischemia; Caspase 3; Disease Models, Animal; Dose-Response Relationship, Drug; Early Growth Response Transcription Factors; Glucosides; Infarction, Middle Cerebral Artery; Male; Neuroprotective Agents; PC12 Cells; Phenols; Random Allocation; Rats; Rats, Sprague-Dawley; Reperfusion Injury; Stroke; Time Factors | 2015 |
Neuroprotective effects of Salidroside and its analogue tyrosol galactoside against focal cerebral ischemia in vivo and H2O2-induced neurotoxicity in vitro.
Topics: Animals; Apoptosis Regulatory Proteins; bcl-2-Associated X Protein; Brain Ischemia; Cell Survival; Cerebral Cortex; Disease Models, Animal; Drug Administration Schedule; Galactosides; Glucosides; Hydrogen Peroxide; Infarction, Middle Cerebral Artery; Male; Neurons; Neuroprotective Agents; Oxidants; Phenols; Phenylethyl Alcohol; Primary Cell Culture; Rats; Rats, Sprague-Dawley; Reactive Oxygen Species; Reperfusion Injury | 2012 |