rhodioloside has been researched along with Hyperglycemia in 4 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 3 (75.00) | 24.3611 |
2020's | 1 (25.00) | 2.80 |
Authors | Studies |
---|---|
Han, J; Kasim, V; Luo, L; Wang, Y; Wu, S | 1 |
Ariyanti, AD; Kasim, V; Marcelina, O; Nugrahaningrum, DA; Wang, G; Wu, S; Zhang, J | 1 |
Alameddine, A; Ayer, A; Bourreau, J; Custaud, MA; Derbre, S; Fajloun, Z; Gauguier, D; Gauquelin-Koch, G; Navasiolava, N; Yuan, M | 1 |
Guo, S; Jiang, K; Liu, X; Luo, L; Shen, N; Sun, C; Wang, C; Wang, M; Xu, M; Yang, Y; Yao, L | 1 |
4 other study(ies) available for rhodioloside and Hyperglycemia
Article | Year |
---|---|
Salidroside facilitates therapeutic angiogenesis in diabetic hindlimb ischemia by inhibiting ferroptosis.
Topics: Animals; Diabetes Mellitus; Disease Models, Animal; Ferroptosis; Hindlimb; Hyperglycemia; Ischemia; Mice; Mice, Inbred C57BL; Muscle, Skeletal; Neovascularization, Physiologic | 2023 |
Salidroside-Pretreated Mesenchymal Stem Cells Enhance Diabetic Wound Healing by Promoting Paracrine Function and Survival of Mesenchymal Stem Cells Under Hyperglycemia.
Topics: Animals; Cells, Cultured; Diabetes Mellitus, Experimental; Glucosides; Hyperglycemia; Male; Mesenchymal Stem Cell Transplantation; Mesenchymal Stem Cells; Mice; Mice, Inbred C57BL; Paracrine Communication; Phenols; Survival Rate; Wound Healing | 2019 |
The cardiovascular effects of salidroside in the Goto-Kakizaki diabetic rat model.
Topics: Animals; Aorta; Blood Pressure; Cardiotonic Agents; Cardiovascular Diseases; Cardiovascular System; Diabetes Mellitus, Type 2; Endothelium, Vascular; Glucose Intolerance; Glucosides; Guanylate Cyclase; Heart Rate; Hyperglycemia; Hypertension; Male; Myocytes, Smooth Muscle; Nitric Oxide Synthase Type III; Phenols; Rats; Rats, Inbred WKY; Receptors, Cytoplasmic and Nuclear; Soluble Guanylyl Cyclase; Vasodilation | 2015 |
Salidroside improves glucose homeostasis in obese mice by repressing inflammation in white adipose tissues and improving leptin sensitivity in hypothalamus.
Topics: Adipose Tissue, White; Animals; Body Weight; Eating; Epididymis; Glucose-6-Phosphatase; Glucosides; Hyperglycemia; Hypothalamus; Inflammation; Leptin; Liver; Male; Mice; Mice, Obese; Obesity; Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha; Phenols; Triglycerides | 2016 |