ramelteon has been researched along with Cardiovascular Stroke in 3 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 1 (33.33) | 24.3611 |
2020's | 2 (66.67) | 2.80 |
Authors | Studies |
---|---|
Jin, Y; Li, S; Zhang, H | 1 |
Bunte, S; Goetze, L; Heinen, A; Hollmann, MW; Huhn, R; Mathes, A; Raupach, A; Stroethoff, M; Torregroza, C | 1 |
Behmenburg, F; Bunte, S; Christoph, I; Heinen, A; Hollmann, MW; Huhn, R; Mathes, A; Raupach, A; Senpolat, S; Stroethoff, M | 1 |
3 other study(ies) available for ramelteon and Cardiovascular Stroke
Article | Year |
---|---|
Remote ischemic preconditioning-induced late cardioprotection: possible role of melatonin-mitoKATP-H2S signaling pathway.
Topics: Animals; Ischemia; Ischemic Preconditioning; Ischemic Preconditioning, Myocardial; KATP Channels; Melatonin; Myocardial Infarction; Myocardial Reperfusion Injury; Rats; Rats, Wistar; Signal Transduction; Tumor Necrosis Factor-alpha | 2023 |
The Melatonin Receptor Agonist Ramelteon Induces Cardioprotection that Requires MT2 Receptor Activation and Release of Reactive Oxygen Species.
Topics: Animals; Cardiovascular Agents; Disease Models, Animal; Indenes; Isolated Heart Preparation; Male; Mitochondria, Heart; Mitochondrial Membrane Transport Proteins; Mitochondrial Permeability Transition Pore; Myocardial Infarction; Myocardial Reperfusion Injury; Myocytes, Cardiac; Rats, Wistar; Reactive Oxygen Species; Receptor, Melatonin, MT2; Signal Transduction; Ventricular Function, Left | 2020 |
Melatonin Receptor Agonist Ramelteon Reduces Ischemia-Reperfusion Injury Through Activation of Mitochondrial Potassium Channels.
Topics: Animals; Cardiovascular Agents; Hemodynamics; Indenes; Isolated Heart Preparation; Male; Mitochondria, Heart; Myocardial Infarction; Myocardial Reperfusion Injury; Myocytes, Cardiac; Potassium Channels; Potassium Channels, Calcium-Activated; Rats, Wistar; Receptors, Melatonin; Signal Transduction; Ventricular Function, Left | 2018 |