quinoxalines has been researched along with Spinal Cord Injuries in 21 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 8 (38.10) | 18.2507 |
| 2000's | 10 (47.62) | 29.6817 |
| 2010's | 3 (14.29) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Bethea, JR; Bracchi-Ricard, V; Gao, H; Jayakumar, AR; Johnstone, AL; Johnstone, JT; Morton, PD; Norenberg, MD; Pearse, DD | 1 |
| Han, X; Liu, H; Lu, M; Lv, D; Wang, S | 1 |
| Beattie, MS; Bresnahan, JC; Gensel, JC; Tovar, CA | 1 |
| Azbill, RD; Mu, X; Springer, JE | 1 |
| Hughes, MG; Hulsebosch, CE; McAdoo, DJ; Xu, GY; Ye, Z | 1 |
| Miranda, A; Peles, S; Sengupta, JN; Shaker, R | 1 |
| Brumley, MR; Robinson, SR | 1 |
| Gwak, YS; Hulsebosch, CE; Kang, J; Leem, JW | 1 |
| Choiniere, D; Teng, YD; Wrathall, JR | 2 |
| Teng, YD; Wrathall, JR | 1 |
| Hao, JX; Xu, XJ | 1 |
| Marriott, R; Teng, YD; Wrathall, JR | 1 |
| Follesa, P; Mocchetti, I; Wrathall, JR | 1 |
| Rosenberg, LJ; Teng, YD; Wrathall, JR | 1 |
| Bennett, AD; Everhart, AW; Hulsebosch, CE | 1 |
| Li, S; Tator, CH | 1 |
| Cobanoğlu, S; Görgülü, A; Kiriş, T; Küçük, M; Turkoğlu, U; Unal, F | 1 |
| Abraham, KE; Brewer, KL; McGinty, JF | 1 |
| Pikov, V; Wrathall, JR | 1 |
| Choiniere, D; Mundt, DJ; Teng, YD; Wrathall, JR | 1 |
21 other study(ies) available for quinoxalines and Spinal Cord Injuries
| Article | Year |
|---|---|
Inhibition of NADPH oxidase activation in oligodendrocytes reduces cytotoxicity following trauma.
Topics: Acetophenones; Animals; Animals, Newborn; Astrocytes; Axons; Cell Death; Culture Media, Conditioned; Enzyme Activation; Enzyme Inhibitors; Excitatory Amino Acid Antagonists; Female; Locomotion; Membrane Glycoproteins; Mice; NADPH Oxidase 2; NADPH Oxidases; Oligodendroglia; Oxidative Stress; Primary Cell Culture; Quinoxalines; Receptors, AMPA; Spinal Cord Injuries | 2013 |
Targeting IKK/NF-κB pathway reduces infiltration of inflammatory cells and apoptosis after spinal cord injury in rats.
Topics: Animals; Anti-Inflammatory Agents, Non-Steroidal; Apoptosis; bcl-2-Associated X Protein; Caspase 3; Enzyme Activation; Female; I-kappa B Kinase; Imidazoles; In Situ Nick-End Labeling; Intercellular Adhesion Molecule-1; Myelitis; Neuroprotective Agents; Neutrophil Infiltration; NF-kappa B; Proto-Oncogene Proteins c-bcl-2; Quinoxalines; Rats; Rats, Sprague-Dawley; Signal Transduction; Spinal Cord Injuries | 2012 |
Topiramate treatment is neuroprotective and reduces oligodendrocyte loss after cervical spinal cord injury.
Topics: Analysis of Variance; Animals; Apoptosis; Cervical Vertebrae; Excitatory Amino Acid Antagonists; Female; Fructose; Grooming; Histological Techniques; Movement; Neuroprotective Agents; Oligodendroglia; Quinoxalines; Rats; Rats, Long-Evans; Spinal Cord Injuries; Topiramate | 2012 |
NBQX treatment improves mitochondrial function and reduces oxidative events after spinal cord injury.
Topics: Animals; Dizocilpine Maleate; Female; Glucose; Glutamic Acid; Injections, Spinal; Lipid Peroxidation; Mitochondria; Neuroprotective Agents; Quinoxalines; Rats; Rats, Long-Evans; Reactive Oxygen Species; Receptors, AMPA; Receptors, Kainic Acid; Receptors, N-Methyl-D-Aspartate; Spinal Cord Injuries; Synaptosomes; Thiobarbituric Acid Reactive Substances; Thoracic Vertebrae | 2002 |
Concentrations of glutamate released following spinal cord injury kill oligodendrocytes in the spinal cord.
Topics: Animals; Cell Count; Disease Models, Animal; Excitatory Amino Acid Antagonists; Glutamic Acid; Male; Microdialysis; Neuroprotective Agents; Oligodendroglia; Quinoxalines; Rats; Rats, Sprague-Dawley; Receptors, AMPA; Spinal Cord; Spinal Cord Injuries; Time Factors | 2004 |
Acute nociceptive somatic stimulus sensitizes neurones in the spinal cord to colonic distension in the rat.
Topics: Acids; Animals; Catheterization; Colon; Excitatory Amino Acid Antagonists; Lumbar Vertebrae; Male; Neurons, Afferent; Nociceptors; Pipecolic Acids; Quinoxalines; Rats; Rats, Sprague-Dawley; Receptors, AMPA; Receptors, N-Methyl-D-Aspartate; Spinal Cord; Spinal Cord Injuries; Stimulation, Chemical | 2004 |
The serotonergic agonists quipazine, CGS-12066A, and alpha-methylserotonin alter motor activity and induce hindlimb stepping in the intact and spinal rat fetus.
Topics: Analysis of Variance; Animals; Behavior, Animal; Dose-Response Relationship, Drug; Embryo, Mammalian; Female; Hindlimb; Male; Motor Activity; Pregnancy; Psychomotor Performance; Quinoxalines; Quipazine; Rats; Rats, Sprague-Dawley; Serotonin; Serotonin Receptor Agonists; Spinal Cord Injuries | 2005 |
Spinal AMPA receptor inhibition attenuates mechanical allodynia and neuronal hyperexcitability following spinal cord injury in rats.
Topics: Animals; Axotomy; Excitatory Amino Acid Antagonists; Hindlimb; Injections, Spinal; Male; Neuralgia; Physical Stimulation; Posterior Horn Cells; Quinoxalines; Rats; Rats, Sprague-Dawley; Receptors, AMPA; Recovery of Function; Spinal Cord Injuries | 2007 |
Dose-dependent reduction of tissue loss and functional impairment after spinal cord trauma with the AMPA/kainate antagonist NBQX.
Topics: Animals; Dose-Response Relationship, Drug; Female; Kainic Acid; Osmolar Concentration; Quinoxalines; Rats; Rats, Sprague-Dawley; Receptors, AMPA; Spinal Cord; Spinal Cord Injuries | 1994 |
Amelioration of functional deficits from spinal cord trauma with systemically administered NBQX, an antagonist of non-N-methyl-D-aspartate receptors.
Topics: alpha-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic Acid; Animals; Behavior, Animal; Female; Quinoxalines; Rats; Rats, Sprague-Dawley; Receptors, N-Methyl-D-Aspartate; Spinal Cord; Spinal Cord Injuries; Time Factors | 1996 |
Evaluation of cardiorespiratory parameters in rats after spinal cord trauma and treatment with NBQX, an antagonist of excitatory amino acid receptors.
Topics: Animals; Blood Pressure; Carbon Dioxide; Excitatory Amino Acid Antagonists; Female; Heart Rate; Oxygen; Partial Pressure; Quinoxalines; Rats; Rats, Sprague-Dawley; Receptors, Glutamate; Reference Values; Respiration; Spinal Cord Injuries | 1996 |
Treatment of a chronic allodynia-like response in spinally injured rats: effects of systemically administered excitatory amino acid receptor antagonists.
Topics: Animals; Behavior, Animal; Chronic Disease; Dextromethorphan; Dizocilpine Maleate; Dose-Response Relationship, Drug; Excitatory Amino Acid Antagonists; Female; Hyperalgesia; Ischemia; Motor Activity; Pipecolic Acids; Quinoxalines; Rats; Rats, Sprague-Dawley; Receptors, AMPA; Receptors, N-Methyl-D-Aspartate; Regional Blood Flow; Spinal Cord; Spinal Cord Injuries | 1996 |
Delayed antagonism of AMPA/kainate receptors reduces long-term functional deficits resulting from spinal cord trauma.
Topics: Animals; Disease Models, Animal; Dose-Response Relationship, Drug; Excitatory Amino Acid Antagonists; Female; Immunohistochemistry; Microinjections; Neurons; Quinoxalines; Rats; Rats, Sprague-Dawley; Receptors, AMPA; Receptors, Kainic Acid; Serotonin; Spinal Cord Injuries; Time Factors | 1997 |
2,3-Dihydroxy-6-nitro-7-sulfamoyl-benzo(F)-quinoxaline (NBQX) increases fibroblast growth factor mRNA levels after contusive spinal cord injury.
Topics: Animals; Contusions; Excitatory Amino Acid Antagonists; Fibroblast Growth Factors; Laminectomy; Quinoxalines; Rats; RNA, Messenger; Spinal Cord Injuries; Tissue Distribution | 1998 |
2,3-Dihydroxy-6-nitro-7-sulfamoyl-benzo(f)quinoxaline reduces glial loss and acute white matter pathology after experimental spinal cord contusion.
Topics: Animals; Brain; Cell Death; Contusions; Excitatory Amino Acid Antagonists; Female; Hindlimb; Immunohistochemistry; Neuroglia; Quinoxalines; Rats; Rats, Sprague-Dawley; Spinal Cord Injuries | 1999 |
Intrathecal administration of an NMDA or a non-NMDA receptor antagonist reduces mechanical but not thermal allodynia in a rodent model of chronic central pain after spinal cord injury.
Topics: Animals; Chronic Disease; Disease Models, Animal; Dose-Response Relationship, Drug; Excitatory Amino Acid Antagonists; Hyperalgesia; Injections, Spinal; Male; Motor Activity; Pain; Physical Stimulation; Quinoxalines; Rats; Rats, Sprague-Dawley; Reaction Time; Receptors, N-Methyl-D-Aspartate; Spinal Cord; Spinal Cord Injuries; Valine | 2000 |
Action of locally administered NMDA and AMPA/kainate receptor antagonists in spinal cord injury.
Topics: Animals; Cerebellum; Dizocilpine Maleate; Evoked Potentials; Evoked Potentials, Motor; Evoked Potentials, Somatosensory; Excitatory Amino Acid Antagonists; Male; Quinoxalines; Rats; Rats, Wistar; Receptors, AMPA; Receptors, Kainic Acid; Receptors, N-Methyl-D-Aspartate; Regional Blood Flow; Spinal Cord; Spinal Cord Injuries | 2000 |
Superoxide dismutase activity and the effects of NBQX and CPP on lipid peroxidation in experimental spinal cord injury.
Topics: Animals; Excitatory Amino Acid Antagonists; Lipid Peroxidation; Male; Malondialdehyde; N-Methylaspartate; Piperazines; Quinoxalines; Rats; Rats, Sprague-Dawley; Spinal Cord; Spinal Cord Injuries; Superoxide Dismutase | 2000 |
The role of kainic acid/AMPA and metabotropic glutamate receptors in the regulation of opioid mRNA expression and the onset of pain-related behavior following excitotoxic spinal cord injury.
Topics: Animals; Behavior, Animal; Dynorphins; Enkephalins; Excitatory Amino Acid Agonists; Excitatory Amino Acid Antagonists; Grooming; Indans; Male; Nerve Degeneration; Neurons; Neurotoxins; Opioid Peptides; Pain; Pain Measurement; Protein Precursors; Quinoxalines; Rats; Rats, Long-Evans; Receptors, AMPA; Receptors, Glutamate; Receptors, Kainic Acid; Receptors, Metabotropic Glutamate; RNA, Messenger; Spinal Cord; Spinal Cord Injuries | 2001 |
Altered glutamate receptor function during recovery of bladder detrusor-external urethral sphincter coordination in a rat model of spinal cord injury.
Topics: Animals; Excitatory Amino Acid Antagonists; Female; Hindlimb; Injections, Spinal; Muscle Contraction; Muscle, Smooth; Piperazines; Quinoxalines; Rats; Rats, Sprague-Dawley; Receptors, AMPA; Receptors, Glutamate; Receptors, N-Methyl-D-Aspartate; Spinal Cord Injuries; Urethra; Urinary Bladder; Urodynamics | 2002 |
Evidence that local non-NMDA receptors contribute to functional deficits in contusive spinal cord injury.
Topics: Animals; Contusions; Female; Hindlimb; Motor Activity; Quinoxalines; Rats; Rats, Inbred Strains; Receptors, Amino Acid; Receptors, Cell Surface; Spinal Cord Injuries | 1992 |