Page last updated: 2024-08-17

quinoxalines and Encephalopathy, Toxic

quinoxalines has been researched along with Encephalopathy, Toxic in 5 studies

Research

Studies (5)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's3 (60.00)29.6817
2010's1 (20.00)24.3611
2020's1 (20.00)2.80

Authors

AuthorsStudies
Guo, SY; Li, CQ; Li, J; Wu, LR; Zhang, Y; Zhou, JL; Zhu, XY1
Aihara, M; Araie, M; Chen, YN; Lee, KY; Nakayama, M1
Akaike, A; Kaneko, S; Katsuki, H; Kume, T; Nishikawa, H; Sugimoto, H; Taguchi, R; Terauchi, T; Yonaga, M1
Heimann, K; Hoernchen, H; Merz, U; Peschgens, T; Wenzl, T1
Laverty, R; Russell, BR1

Other Studies

5 other study(ies) available for quinoxalines and Encephalopathy, Toxic

ArticleYear
Developmental neurotoxicity and toxic mechanisms induced by olaquindox in zebrafish.
    Journal of applied toxicology : JAT, 2021, Volume: 41, Issue:4

    Topics: Animals; China; Embryo, Nonmammalian; Embryonic Development; Genetic Variation; Genotype; Neurotoxicity Syndromes; Quinoxalines; Water Pollutants, Chemical; Zebrafish

2021
Brimonidine is neuroprotective against glutamate-induced neurotoxicity, oxidative stress, and hypoxia in purified rat retinal ganglion cells.
    Molecular vision, 2010, Feb-17, Volume: 16

    Topics: Animals; Brimonidine Tartrate; Cell Hypoxia; Cell Separation; Cell Survival; Glutamic Acid; Neuroprotective Agents; Neurotoxicity Syndromes; Oxidative Stress; Quinoxalines; Rats; Rats, Wistar; Retinal Ganglion Cells

2010
Serofendic acid prevents acute glutamate neurotoxicity in cultured cortical neurons.
    European journal of pharmacology, 2003, Sep-23, Volume: 477, Issue:3

    Topics: Animals; Calcium; Cattle; Cell Survival; Cells, Cultured; Cerebral Cortex; Cysteine; Diterpenes; Dizocilpine Maleate; Dose-Response Relationship, Drug; Drug Combinations; Fetal Blood; Fetus; Ionomycin; Neurons; Neurotoxicity Syndromes; Nitric Oxide; Quinoxalines; Rats; S-Nitrosothiols; Sodium Glutamate; Time Factors; Valine

2003
[Depression of respiration via toxic effects on the central nervous system following use of topical brimonidine in an infant with congenital glaucoma].
    Der Ophthalmologe : Zeitschrift der Deutschen Ophthalmologischen Gesellschaft, 2007, Volume: 104, Issue:6

    Topics: Antihypertensive Agents; Apnea; Brimonidine Tartrate; Cataract; Female; Glaucoma; Humans; Infant, Newborn; Infant, Premature, Diseases; Neurotoxicity Syndromes; Ophthalmic Solutions; Postoperative Complications; Quinoxalines; Respiratory Insufficiency; Trabeculectomy

2007
The effect of (R)-HA966 or ACEA 1021 on dexfenfluramine or (S)-MDMA-induced changes in temperature, activity, and neurotoxicity.
    Pharmacology, biochemistry, and behavior, 2001, Volume: 68, Issue:3

    Topics: Animals; Autoradiography; Body Temperature; Brain Chemistry; Carrier Proteins; Chromatography, High Pressure Liquid; Dexfenfluramine; Dose-Response Relationship, Drug; Excitatory Amino Acid Antagonists; Male; Membrane Glycoproteins; Membrane Transport Proteins; Motor Activity; N-Methyl-3,4-methylenedioxyamphetamine; Nerve Tissue Proteins; Neurotoxicity Syndromes; Pyrrolidinones; Quinoxalines; Rats; Rats, Wistar; Receptors, N-Methyl-D-Aspartate; Serotonin; Serotonin Plasma Membrane Transport Proteins; Serotonin Receptor Agonists; Stereoisomerism; Telemetry

2001