quinone has been researched along with Necrosis in 4 studies
benzoquinone : The simplest members of the class of benzoquinones, consisting of cyclohexadiene which is substituted by two oxo groups.
1,4-benzoquinone : The simplest member of the class of 1,4-benzoquinones, obtained by the formal oxidation of hydroquinone to the corresponding diketone. It is a metabolite of benzene.
quinone : Compounds having a fully conjugated cyclic dione structure, such as that of benzoquinones, derived from aromatic compounds by conversion of an even number of -CH= groups into -C(=O)- groups with any necessary rearrangement of double bonds (polycyclic and heterocyclic analogues are included).
Necrosis: The death of cells in an organ or tissue due to disease, injury or failure of the blood supply.
| Excerpt | Relevance | Reference |
|---|---|---|
| "The resistance of BQ-treated cells to necrosis could be due to increased cellular GSH and formation of BQ-GSH conjugates which are less toxic than free BQ, minimal toxicity being provided by GS4-BHQ." | 5.35 | Apoptosis/necrosis switch in two different cancer cell lines: influence of benzoquinone- and hydrogen peroxide-induced oxidative stress intensity, and glutathione. ( Bagrel, D; Baigi, MG; Beley, M; Brault, L; Gaüzère, F; Hilali, RE; Néguesque, A, 2008) |
| " We also monitored the nuclear envelope in living cells after induction of apoptosis or necrosis using 1 microM staurosporine or 100 microM p-benzoquinone, respectively." | 3.70 | Noninvasive monitoring of apoptosis versus necrosis in a neuroblastoma cell line expressing a nuclear pore protein tagged with the green fluorescent protein. ( Beckman, M; Hallberg, E; Imreh, G; Iverfeldt, K, 1998) |
| "The mechanism(s) of liver damage initiated by ingestion of toxic components of thermally oxidized lipids was compared in a rat model with the documented mechanisms of hepatic failure and necrosis initiated by acetaminophen." | 3.69 | Comparison of thermally oxidized lipids and acetaminophen with concurrent consumption of ethanol as inducers of liver cirrhosis. ( Fouad, FM; Mamer, OA; Shahidi, F, 1995) |
| "The resistance of BQ-treated cells to necrosis could be due to increased cellular GSH and formation of BQ-GSH conjugates which are less toxic than free BQ, minimal toxicity being provided by GS4-BHQ." | 1.35 | Apoptosis/necrosis switch in two different cancer cell lines: influence of benzoquinone- and hydrogen peroxide-induced oxidative stress intensity, and glutathione. ( Bagrel, D; Baigi, MG; Beley, M; Brault, L; Gaüzère, F; Hilali, RE; Néguesque, A, 2008) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 2 (50.00) | 18.2507 |
| 2000's | 2 (50.00) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Baigi, MG | 1 |
| Brault, L | 1 |
| Néguesque, A | 1 |
| Beley, M | 1 |
| Hilali, RE | 1 |
| Gaüzère, F | 1 |
| Bagrel, D | 1 |
| Fouad, FM | 1 |
| Shahidi, F | 1 |
| Mamer, OA | 1 |
| Imreh, G | 1 |
| Beckman, M | 1 |
| Iverfeldt, K | 1 |
| Hallberg, E | 1 |
| Arriagada, C | 1 |
| Paris, I | 1 |
| Sanchez de las Matas, MJ | 1 |
| Martinez-Alvarado, P | 1 |
| Cardenas, S | 1 |
| Castañeda, P | 1 |
| Graumann, R | 1 |
| Perez-Pastene, C | 1 |
| Olea-Azar, C | 1 |
| Couve, E | 1 |
| Herrero, MT | 1 |
| Caviedes, P | 1 |
| Segura-Aguilar, J | 1 |
4 other studies available for quinone and Necrosis
| Article | Year |
|---|---|
Apoptosis/necrosis switch in two different cancer cell lines: influence of benzoquinone- and hydrogen peroxide-induced oxidative stress intensity, and glutathione.
Topics: Animals; Apoptosis; Benzoquinones; Breast Neoplasms; Carcinoma, Hepatocellular; Cell Line, Tumor; Fe | 2008 |
Comparison of thermally oxidized lipids and acetaminophen with concurrent consumption of ethanol as inducers of liver cirrhosis.
Topics: Acetaminophen; Acute-Phase Reaction; Analysis of Variance; Animals; Benzoquinones; DNA; Drug Interac | 1995 |
Noninvasive monitoring of apoptosis versus necrosis in a neuroblastoma cell line expressing a nuclear pore protein tagged with the green fluorescent protein.
Topics: Apoptosis; Benzoquinones; Biomarkers; Chromatin; DNA Fragmentation; Green Fluorescent Proteins; Huma | 1998 |
On the neurotoxicity mechanism of leukoaminochrome o-semiquinone radical derived from dopamine oxidation: mitochondria damage, necrosis, and hydroxyl radical formation.
Topics: Animals; Benzoquinones; Cell Line; Dopamine; Hydroxyl Radical; Membrane Potentials; Mitochondria; Ne | 2004 |