Page last updated: 2024-11-02

quinone and Necrosis

quinone has been researched along with Necrosis in 4 studies

benzoquinone : The simplest members of the class of benzoquinones, consisting of cyclohexadiene which is substituted by two oxo groups.
1,4-benzoquinone : The simplest member of the class of 1,4-benzoquinones, obtained by the formal oxidation of hydroquinone to the corresponding diketone. It is a metabolite of benzene.
quinone : Compounds having a fully conjugated cyclic dione structure, such as that of benzoquinones, derived from aromatic compounds by conversion of an even number of -CH= groups into -C(=O)- groups with any necessary rearrangement of double bonds (polycyclic and heterocyclic analogues are included).

Necrosis: The death of cells in an organ or tissue due to disease, injury or failure of the blood supply.

Research Excerpts

ExcerptRelevanceReference
"The resistance of BQ-treated cells to necrosis could be due to increased cellular GSH and formation of BQ-GSH conjugates which are less toxic than free BQ, minimal toxicity being provided by GS4-BHQ."5.35Apoptosis/necrosis switch in two different cancer cell lines: influence of benzoquinone- and hydrogen peroxide-induced oxidative stress intensity, and glutathione. ( Bagrel, D; Baigi, MG; Beley, M; Brault, L; Gaüzère, F; Hilali, RE; Néguesque, A, 2008)
" We also monitored the nuclear envelope in living cells after induction of apoptosis or necrosis using 1 microM staurosporine or 100 microM p-benzoquinone, respectively."3.70Noninvasive monitoring of apoptosis versus necrosis in a neuroblastoma cell line expressing a nuclear pore protein tagged with the green fluorescent protein. ( Beckman, M; Hallberg, E; Imreh, G; Iverfeldt, K, 1998)
"The mechanism(s) of liver damage initiated by ingestion of toxic components of thermally oxidized lipids was compared in a rat model with the documented mechanisms of hepatic failure and necrosis initiated by acetaminophen."3.69Comparison of thermally oxidized lipids and acetaminophen with concurrent consumption of ethanol as inducers of liver cirrhosis. ( Fouad, FM; Mamer, OA; Shahidi, F, 1995)
"The resistance of BQ-treated cells to necrosis could be due to increased cellular GSH and formation of BQ-GSH conjugates which are less toxic than free BQ, minimal toxicity being provided by GS4-BHQ."1.35Apoptosis/necrosis switch in two different cancer cell lines: influence of benzoquinone- and hydrogen peroxide-induced oxidative stress intensity, and glutathione. ( Bagrel, D; Baigi, MG; Beley, M; Brault, L; Gaüzère, F; Hilali, RE; Néguesque, A, 2008)

Research

Studies (4)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's2 (50.00)18.2507
2000's2 (50.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Baigi, MG1
Brault, L1
Néguesque, A1
Beley, M1
Hilali, RE1
Gaüzère, F1
Bagrel, D1
Fouad, FM1
Shahidi, F1
Mamer, OA1
Imreh, G1
Beckman, M1
Iverfeldt, K1
Hallberg, E1
Arriagada, C1
Paris, I1
Sanchez de las Matas, MJ1
Martinez-Alvarado, P1
Cardenas, S1
Castañeda, P1
Graumann, R1
Perez-Pastene, C1
Olea-Azar, C1
Couve, E1
Herrero, MT1
Caviedes, P1
Segura-Aguilar, J1

Other Studies

4 other studies available for quinone and Necrosis

ArticleYear
Apoptosis/necrosis switch in two different cancer cell lines: influence of benzoquinone- and hydrogen peroxide-induced oxidative stress intensity, and glutathione.
    Toxicology in vitro : an international journal published in association with BIBRA, 2008, Volume: 22, Issue:6

    Topics: Animals; Apoptosis; Benzoquinones; Breast Neoplasms; Carcinoma, Hepatocellular; Cell Line, Tumor; Fe

2008
Comparison of thermally oxidized lipids and acetaminophen with concurrent consumption of ethanol as inducers of liver cirrhosis.
    Journal of toxicology and environmental health, 1995, Volume: 46, Issue:2

    Topics: Acetaminophen; Acute-Phase Reaction; Analysis of Variance; Animals; Benzoquinones; DNA; Drug Interac

1995
Noninvasive monitoring of apoptosis versus necrosis in a neuroblastoma cell line expressing a nuclear pore protein tagged with the green fluorescent protein.
    Experimental cell research, 1998, Feb-01, Volume: 238, Issue:2

    Topics: Apoptosis; Benzoquinones; Biomarkers; Chromatin; DNA Fragmentation; Green Fluorescent Proteins; Huma

1998
On the neurotoxicity mechanism of leukoaminochrome o-semiquinone radical derived from dopamine oxidation: mitochondria damage, necrosis, and hydroxyl radical formation.
    Neurobiology of disease, 2004, Volume: 16, Issue:2

    Topics: Animals; Benzoquinones; Cell Line; Dopamine; Hydroxyl Radical; Membrane Potentials; Mitochondria; Ne

2004