quinazolines has been researched along with Hepatolenticular Degeneration in 2 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 2 (100.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Borchard, S; Concilli, M; Crispino, R; De Cegli, R; Di Schiavi, E; Einer, C; Franco, B; Ilyechova, EY; Indrieri, A; Iorio, R; Kroemer, G; Lichtmannegger, J; Mariniello, M; Merolla, A; Petruzzelli, R; Pietrocola, F; Polishchuk, EV; Polishchuk, RS; Puchkova, LV; Ranucci, G; Romano, A; Schmidt, HH; Zibert, A; Zischka, H | 1 |
Bertele', V; Garattini, S; Gerardi, C; Joppi, R | 1 |
1 review(s) available for quinazolines and Hepatolenticular Degeneration
Article | Year |
---|---|
Letting post-marketing bridge the evidence gap: the case of orphan drugs.
Topics: Adrenal Cortex Neoplasms; Adrenocortical Carcinoma; Anti-Inflammatory Agents, Non-Steroidal; Antineoplastic Agents; Antineoplastic Agents, Hormonal; Barrett Esophagus; Cladribine; Dihematoporphyrin Ether; Ductus Arteriosus, Patent; Hepatolenticular Degeneration; Humans; Ibuprofen; Leukemia, Hairy Cell; Mitotane; Orphan Drug Production; Platelet Aggregation Inhibitors; Product Surveillance, Postmarketing; Quinazolines; Thrombocythemia, Essential; Zinc Acetate | 2016 |
1 other study(ies) available for quinazolines and Hepatolenticular Degeneration
Article | Year |
---|---|
Activation of Autophagy, Observed in Liver Tissues From Patients With Wilson Disease and From ATP7B-Deficient Animals, Protects Hepatocytes From Copper-Induced Apoptosis.
Topics: Animals; Apoptosis; Autophagosomes; Autophagy; Basic Helix-Loop-Helix Leucine Zipper Transcription Factors; Benzylamines; Cell Survival; Copper; Copper-Transporting ATPases; Female; Hep G2 Cells; Hepatocytes; Hepatolenticular Degeneration; Humans; Liver; Male; Mice; Mice, Knockout; Microscopy, Confocal; Microscopy, Electron; Mitochondria; Protein Transport; Quinazolines; Rats; Signal Transduction; TOR Serine-Threonine Kinases | 2019 |