pyrimidine dimers has been researched along with Li-Fraumeni Syndrome in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 3 (100.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Ford, JM; Hanawalt, PC | 1 |
| Barley, RD; Enns, L; Mirzayans, R; Paterson, MC | 1 |
| Baril, C; Drobetsky, EA; Drouin, R; Therrien, JP | 1 |
3 other study(ies) available for pyrimidine dimers and Li-Fraumeni Syndrome
| Article | Year |
|---|---|
Li-Fraumeni syndrome fibroblasts homozygous for p53 mutations are deficient in global DNA repair but exhibit normal transcription-coupled repair and enhanced UV resistance.
Topics: Apoptosis; Cell Survival; Clone Cells; DNA Repair; Dose-Response Relationship, Radiation; Fibroblasts; Homozygote; Humans; Li-Fraumeni Syndrome; Microscopy, Fluorescence; Mutation; Neoplasms; Pyrimidine Dimers; Radiation Tolerance; Tetrahydrofolate Dehydrogenase; Transcription, Genetic; Tumor Suppressor Protein p53; Ultraviolet Rays | 1995 |
Aberrant p21WAF1-dependent growth arrest as the possible mechanism of abnormal resistance to ultraviolet light cytotoxicity in Li-Fraumeni syndrome fibroblast strains heterozygous for TP53 mutations.
Topics: Apoptosis; Cell Division; Cells, Cultured; Cyclin-Dependent Kinase Inhibitor p21; Cyclins; DNA Damage; Fibroblasts; Heterozygote; Humans; Li-Fraumeni Syndrome; Mutation; Pyrimidine Dimers; RNA; Tumor Suppressor Protein p53; Ultraviolet Rays; Up-Regulation | 1998 |
Human cells compromised for p53 function exhibit defective global and transcription-coupled nucleotide excision repair, whereas cells compromised for pRb function are defective only in global repair.
Topics: DNA Repair; Fibroblasts; Humans; Li-Fraumeni Syndrome; Lung; Oncogene Proteins, Viral; Pyrimidine Dimers; Retinoblastoma Protein; Skin; Transcription, Genetic; Tumor Suppressor Protein p53; Ultraviolet Rays | 1999 |