pyridoxine has been researched along with Nephrocalcinosis in 10 studies
4,5-bis(hydroxymethyl)-2-methylpyridin-3-ol: structure in first source
vitamin B6 : Any member of the group of pyridines that exhibit biological activity against vitamin B6 deficiency. Vitamin B6 deficiency is associated with microcytic anemia, electroencephalographic abnormalities, dermatitis with cheilosis (scaling on the lips and cracks at the corners of the mouth) and glossitis (swollen tongue), depression and confusion, and weakened immune function. Vitamin B6 consists of the vitamers pyridoxine, pyridoxal, and pyridoxamine and their respective 5'-phosphate esters (and includes their corresponding ionized and salt forms).
Nephrocalcinosis: A condition characterized by calcification of the renal tissue itself. It is usually seen in distal RENAL TUBULAR ACIDOSIS with calcium deposition in the DISTAL KIDNEY TUBULES and the surrounding interstitium. Nephrocalcinosis causes RENAL INSUFFICIENCY.
| Excerpt | Relevance | Reference |
|---|---|---|
| "Pyridoxine-responsive seizures (PRS) and the role of pyridoxine (PN, vitamin B(6)) in hypophosphatasia (HPP) are incompletely understood." | 7.74 | Pyridoxine-responsive seizures as the first symptom of infantile hypophosphatasia caused by two novel missense mutations (c.677T>C, p.M226T; c.1112C>T, p.T371I) of the tissue-nonspecific alkaline phosphatase gene. ( Baumgartner-Sigl, S; Ericson, KL; Haberlandt, E; Högler, W; Mumm, S; Ryan, L; Scholl-Bürgi, S; Sergi, C; Whyte, MP, 2007) |
| "Pyridoxine-responsive seizures (PRS) and the role of pyridoxine (PN, vitamin B(6)) in hypophosphatasia (HPP) are incompletely understood." | 3.74 | Pyridoxine-responsive seizures as the first symptom of infantile hypophosphatasia caused by two novel missense mutations (c.677T>C, p.M226T; c.1112C>T, p.T371I) of the tissue-nonspecific alkaline phosphatase gene. ( Baumgartner-Sigl, S; Ericson, KL; Haberlandt, E; Högler, W; Mumm, S; Ryan, L; Scholl-Bürgi, S; Sergi, C; Whyte, MP, 2007) |
| "Primary hyperoxaluria is a rare genetic disorder characterized by oxalate overproduction, leading to kidney failure due to nephrocalcinosis, and is eventually responsible for systemic oxalosis." | 1.48 | Type 1 primary hyperoxaluria: A case report and focus on bone impairment of systemic oxalosis. ( Boivin, G; Caillard, S; Javier, RM; Pijnenburg, L; Rizzo, S, 2018) |
| "Primary hyperoxalurias are rare inborn errors of metabolism resulting in increased endogenous production of oxalate that leads to excessive urinary oxalate excretion." | 1.46 | Clinical spectrum of primary hyperoxaluria type 1: Experience of a tertiary center. ( Abdelaziz, H; Abdelrahman, SM; Badr, AM; Bazaraa, HM; Eweeda, KM; Fayez, A; Ghanim, K; Helmy, R; Kotb, MA; Nabhan, MM; Soliman, NA; Tolba, OA, 2017) |
| "Primary hyperoxaluria type I (PH I) is characterized by an excessive endogenous production and excretion of oxalic and glycolic acid." | 1.28 | Vitamin B6 resistant primary hyperoxaluria type I. Report of 5 cases. ( Donckerwolcke, RA; Streefland, M, 1989) |
| "Significant progress of nephrolithiasis and nephrocalcinosis was observed in 80% of patients with permanent hypomagnesemia and in 4% of patients with normalization of serum magnesium level." | 1.27 | Prognosis of urolithiasis and nephrocalcinosis in hypomagnesemia. ( Karlíková, L; Revúsová, V; Suchánek, B; Zvara, V, 1985) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 6 (60.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (10.00) | 29.6817 |
| 2010's | 3 (30.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Pijnenburg, L | 1 |
| Caillard, S | 1 |
| Boivin, G | 1 |
| Rizzo, S | 1 |
| Javier, RM | 1 |
| Sas, DJ | 1 |
| Harris, PC | 1 |
| Milliner, DS | 1 |
| Soliman, NA | 1 |
| Nabhan, MM | 1 |
| Abdelrahman, SM | 1 |
| Abdelaziz, H | 1 |
| Helmy, R | 1 |
| Ghanim, K | 1 |
| Bazaraa, HM | 1 |
| Badr, AM | 1 |
| Tolba, OA | 1 |
| Kotb, MA | 1 |
| Eweeda, KM | 1 |
| Fayez, A | 1 |
| Baumgartner-Sigl, S | 1 |
| Haberlandt, E | 1 |
| Mumm, S | 1 |
| Scholl-Bürgi, S | 1 |
| Sergi, C | 1 |
| Ryan, L | 1 |
| Ericson, KL | 1 |
| Whyte, MP | 1 |
| Högler, W | 1 |
| Streefland, M | 1 |
| Donckerwolcke, RA | 1 |
| Vigeral, P | 1 |
| Kenouch, S | 1 |
| Chauveau, D | 1 |
| Mougenot, B | 1 |
| Méry, JP | 1 |
| Revúsová, V | 1 |
| Zvara, V | 1 |
| Karlíková, L | 1 |
| Suchánek, B | 1 |
| Kurbanova, EG | 1 |
| Mishchenko, BP | 1 |
| McCombs, HL | 1 |
| Gershoff, SN | 1 |
| Broyer, M | 1 |
| Berger, R | 1 |
1 review available for pyridoxine and Nephrocalcinosis
| Article | Year |
|---|---|
Recent advances in the identification and management of inherited hyperoxalurias.
Topics: Alcohol Oxidoreductases; Fluid Therapy; Genetic Testing; Humans; Hyperoxaluria, Primary; Kidney Calc | 2019 |
9 other studies available for pyridoxine and Nephrocalcinosis
| Article | Year |
|---|---|
Type 1 primary hyperoxaluria: A case report and focus on bone impairment of systemic oxalosis.
Topics: Adult; Biopsy; Bone Density; Calcium Oxalate; Humans; Hyperoxaluria, Primary; Ilium; Kidney Failure, | 2018 |
Clinical spectrum of primary hyperoxaluria type 1: Experience of a tertiary center.
Topics: Adult; Child; Child, Preschool; Cohort Studies; Consanguinity; Egypt; Female; Humans; Hyperoxaluria, | 2017 |
Pyridoxine-responsive seizures as the first symptom of infantile hypophosphatasia caused by two novel missense mutations (c.677T>C, p.M226T; c.1112C>T, p.T371I) of the tissue-nonspecific alkaline phosphatase gene.
Topics: Alkaline Phosphatase; Ethanolamines; Exons; Fatal Outcome; Female; Humans; Hypercalcemia; Hypercalci | 2007 |
Vitamin B6 resistant primary hyperoxaluria type I. Report of 5 cases.
Topics: Child; Child, Preschool; Drug Resistance; Female; Follow-Up Studies; Humans; Hyperoxaluria; Hyperoxa | 1989 |
Piridoxilate-associated nephrocalcinosis: a new form of chronic oxalate nephropathy.
Topics: Aged; Calcium Oxalate; Glyoxylates; Humans; Kidney Calculi; Male; Nephrocalcinosis; Pyridoxine | 1987 |
Prognosis of urolithiasis and nephrocalcinosis in hypomagnesemia.
Topics: Adolescent; Adult; Aged; Allopurinol; Cation Exchange Resins; Cellulose; Child; Child, Preschool; Ci | 1985 |
[Pyridoxine levels in the bodies of patients with dysmetabolic nephropathy].
Topics: Adolescent; Calcium; Child; Child, Preschool; Humans; Nephrocalcinosis; Oxalates; Oxalic Acid; Pyrid | 1985 |
Effect of vitamin B 6 deficiency on the rat kidney.
Topics: Animals; Avitaminosis; Calcium; Chromatin; Cytoplasm; Endoplasmic Reticulum; Epithelial Cells; Golgi | 1972 |
[Oxalosis].
Topics: Acidosis, Renal Tubular; Alcohol Oxidoreductases; Allopurinol; Biopsy; Blood Urea Nitrogen; Calcium | 1968 |