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pyridoxine and Ischemic Attack, Transient

pyridoxine has been researched along with Ischemic Attack, Transient in 6 studies

4,5-bis(hydroxymethyl)-2-methylpyridin-3-ol: structure in first source
vitamin B6 : Any member of the group of pyridines that exhibit biological activity against vitamin B6 deficiency. Vitamin B6 deficiency is associated with microcytic anemia, electroencephalographic abnormalities, dermatitis with cheilosis (scaling on the lips and cracks at the corners of the mouth) and glossitis (swollen tongue), depression and confusion, and weakened immune function. Vitamin B6 consists of the vitamers pyridoxine, pyridoxal, and pyridoxamine and their respective 5'-phosphate esters (and includes their corresponding ionized and salt forms).

Ischemic Attack, Transient: Brief reversible episodes of focal, nonconvulsive ischemic dysfunction of the brain having a duration of less than 24 hours, and usually less than one hour, caused by transient thrombotic or embolic blood vessel occlusion or stenosis. Events may be classified by arterial distribution, temporal pattern, or etiology (e.g., embolic vs. thrombotic). (From Adams et al., Principles of Neurology, 6th ed, pp814-6)

Research Excerpts

ExcerptRelevanceReference
"Epidemiological and laboratory studies suggest that increasing concentrations of plasma homocysteine (total homocysteine [tHcy]) accelerate cardiovascular disease by promoting vascular inflammation, endothelial dysfunction, and hypercoagulability."9.11Homocysteine-lowering treatment with folic acid, cobalamin, and pyridoxine does not reduce blood markers of inflammation, endothelial dysfunction, or hypercoagulability in patients with previous transient ischemic attack or stroke: a randomized substudy o ( Baker, R; Dusitanond, P; Eikelboom, JW; Gilmore, G; Hankey, GJ; Jamrozik, K; Klijn, CJ; Langton, P; Loh, K; Thom, J; van Bockxmeer, FM; Yi, Q, 2005)
"Epidemiological and laboratory studies suggest that increasing concentrations of plasma homocysteine (total homocysteine [tHcy]) accelerate cardiovascular disease by promoting vascular inflammation, endothelial dysfunction, and hypercoagulability."5.11Homocysteine-lowering treatment with folic acid, cobalamin, and pyridoxine does not reduce blood markers of inflammation, endothelial dysfunction, or hypercoagulability in patients with previous transient ischemic attack or stroke: a randomized substudy o ( Baker, R; Dusitanond, P; Eikelboom, JW; Gilmore, G; Hankey, GJ; Jamrozik, K; Klijn, CJ; Langton, P; Loh, K; Thom, J; van Bockxmeer, FM; Yi, Q, 2005)

Research

Studies (6)

TimeframeStudies, this research(%)All Research%
pre-19903 (50.00)18.7374
1990's1 (16.67)18.2507
2000's2 (33.33)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Dusitanond, P1
Eikelboom, JW1
Hankey, GJ1
Thom, J1
Gilmore, G1
Loh, K1
Yi, Q1
Klijn, CJ1
Langton, P1
van Bockxmeer, FM1
Baker, R1
Jamrozik, K1
Vermeulen, EG1
Rauwerda, JA1
Erix, P1
de Jong, SC1
Twisk, JW1
Jakobs, C1
Witjes, RJ1
Stehouwer, CD1
Lantos, J1
Sárosi, I1
Temes, G1
Török, B1
Zervas, NT1
Hori, H1
Rosoff, CB1
Clerens, J1
Ballesteros Martínez, L1
Manera Oleo, JM1
Olba Miralles, L1

Trials

1 trial available for pyridoxine and Ischemic Attack, Transient

ArticleYear
Homocysteine-lowering treatment with folic acid, cobalamin, and pyridoxine does not reduce blood markers of inflammation, endothelial dysfunction, or hypercoagulability in patients with previous transient ischemic attack or stroke: a randomized substudy o
    Stroke, 2005, Volume: 36, Issue:1

    Topics: Biomarkers; Blood Coagulation; Cardiovascular Diseases; Endothelium, Vascular; Folic Acid; Homocyste

2005

Other Studies

5 other studies available for pyridoxine and Ischemic Attack, Transient

ArticleYear
Normohomocysteinaemia and vitamin-treated hyperhomocysteinaemia are associated with similar risks of cardiovascular events in patients with premature atherothrombotic cerebrovascular disease. A prospective cohort study.
    The Netherlands journal of medicine, 2000, Volume: 56, Issue:4

    Topics: Adult; Arteriosclerosis; Female; Folic Acid; Follow-Up Studies; Hematinics; Humans; Hyperhomocystein

2000
Influence of protective drugs on the EEG during short-term transient ischaemia.
    Acta physiologica Hungarica, 1990, Volume: 75, Issue:3

    Topics: Animals; Dogs; Electroencephalography; Ischemic Attack, Transient; Lidocaine; Phenytoin; Piracetam;

1990
Experimental inhibition of serotonin by antibiotic: prevention of cerebral vasospasm.
    Journal of neurosurgery, 1974, Volume: 41, Issue:1

    Topics: Animals; Cerebral Angiography; Disease Models, Animal; Evaluation Studies as Topic; Haplorhini; Isch

1974
[Problem in psychogeriatrics: quantitative and qualitative cerebral circulation insufficiencies in geriatric medicine].
    Zeitschrift fur Alternsforschung, 1971, Volume: 24, Issue:1

    Topics: Aged; Aging; Cerebrovascular Disorders; Dietary Proteins; Glucose; Hemodynamics; Humans; Infections;

1971
[Clinical trial with a new antilipemic agent in chronic cerebrovascular insufficiency].
    Revista clinica espanola, 1969, Aug-31, Volume: 114, Issue:4

    Topics: Adult; Aged; Anticholesteremic Agents; Cerebrovascular Disorders; Cholesterol; Dextrans; Female; Hum

1969