Page last updated: 2024-08-18

pyrazolanthrone and Thyroid Neoplasms

pyrazolanthrone has been researched along with Thyroid Neoplasms in 6 studies

Research

Studies (6)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (16.67)29.6817
2010's5 (83.33)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Cui, D; Xu, J; Zhao, Y1
Chen, X; Chen, XJ; Gong, XH; Jiang, ZC; Wu, WJ; Zhou, Q1
Fugazzola, L; Grassi, ES; Lábadi, Á; Negri, I; Persani, L; Vezzoli, V; Vitale, G1
Bosari, S; Cirello, V; Colombo, C; Ferrero, S; Fugazzola, L; Grassi, ES; Persani, L; Ricca, D; Vaira, V; Vezzoli, V; Vicentini, L1
Charpentier, E; El Btaouri, H; Greffe, Y; Martiny, L; Morjani, H1
Bulgin, D; Mitsutake, N; Namba, H; Ohtsuru, A; Palona, I; Podtcheko, A; Rogounovitch, T; Saenko, V; Takakura, S; Yamashita, S1

Other Studies

6 other study(ies) available for pyrazolanthrone and Thyroid Neoplasms

ArticleYear
Activation of CXCL5-CXCR2 axis promotes proliferation and accelerates G1 to S phase transition of papillary thyroid carcinoma cells and activates JNK and p38 pathways.
    Cancer biology & therapy, 2019, Volume: 20, Issue:5

    Topics: Animals; Anthracenes; Cell Line, Tumor; Cell Proliferation; Chemokine CXCL5; G1 Phase Cell Cycle Checkpoints; Humans; Imidazoles; MAP Kinase Signaling System; Mice; Mice, Nude; Phenylurea Compounds; Phosphorylation; Pyridines; Receptors, Interleukin-8B; Recombinant Proteins; Thyroid Cancer, Papillary; Thyroid Neoplasms; Xenograft Model Antitumor Assays

2019
Downregulated LRRK2 gene expression inhibits proliferation and migration while promoting the apoptosis of thyroid cancer cells by inhibiting activation of the JNK signaling pathway.
    International journal of oncology, 2019, Volume: 55, Issue:1

    Topics: Anthracenes; Apoptosis; Cell Cycle Checkpoints; Cell Line, Tumor; Cell Movement; Cell Proliferation; Down-Regulation; Enzyme Activation; Humans; Leucine-Rich Repeat Serine-Threonine Protein Kinase-2; MAP Kinase Kinase 4; MAP Kinase Signaling System; Thyroid Neoplasms; Transfection

2019
SP600125 has a remarkable anticancer potential against undifferentiated thyroid cancer through selective action on ROCK and p53 pathways.
    Oncotarget, 2015, Nov-03, Volume: 6, Issue:34

    Topics: Anthracenes; Cell Line, Tumor; Cell Proliferation; Histone Deacetylase 6; Histone Deacetylases; Humans; Protein Kinase Inhibitors; rho-Associated Kinases; Signal Transduction; Thyroid Neoplasms; Tumor Suppressor Protein p53

2015
Multicellular spheroids from normal and neoplastic thyroid tissues as a suitable model to test the effects of multikinase inhibitors.
    Oncotarget, 2017, Feb-07, Volume: 8, Issue:6

    Topics: Adenocarcinoma, Follicular; Adult; Aged; Aged, 80 and over; Anthracenes; Antineoplastic Agents; beta Catenin; Carcinoma, Papillary; Case-Control Studies; Cell Movement; Cell Proliferation; Female; Humans; Male; Middle Aged; Molecular Targeted Therapy; Phenotype; Protein Kinase Inhibitors; Protein Kinases; rho-Associated Kinases; Signal Transduction; Spheroids, Cellular; Thyroid Cancer, Papillary; Thyroid Gland; Thyroid Neoplasms; Tumor Cells, Cultured

2017
Role of JNK/ATF-2 pathway in inhibition of thrombospondin-1 (TSP-1) expression and apoptosis mediated by doxorubicin and camptothecin in FTC-133 cells.
    Biochimica et biophysica acta, 2011, Volume: 1813, Issue:5

    Topics: Activating Transcription Factor 2; Anthracenes; Apoptosis; Camptothecin; Caspase 3; Cell Line, Tumor; Cell Survival; Doxorubicin; Enzyme Activation; Gene Expression Regulation, Neoplastic; Humans; JNK Mitogen-Activated Protein Kinases; Phosphorylation; Signal Transduction; Sphingosine; Thrombospondin 1; Thyroid Neoplasms

2011
Selective pharmacologic inhibition of c-Jun NH2-terminal kinase radiosensitizes thyroid anaplastic cancer cell lines via induction of terminal growth arrest.
    Thyroid : official journal of the American Thyroid Association, 2006, Volume: 16, Issue:3

    Topics: Activating Transcription Factor 2; Anthracenes; Antineoplastic Agents; Apoptosis; Cell Line, Tumor; Cell Proliferation; Cellular Senescence; Combined Modality Therapy; DNA Repair; Humans; JNK Mitogen-Activated Protein Kinases; Radiation-Sensitizing Agents; Signal Transduction; Thyroid Neoplasms

2006