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pyrazolanthrone and Encephalopathy, Toxic

pyrazolanthrone has been researched along with Encephalopathy, Toxic in 4 studies

Research

Studies (4)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's2 (50.00)24.3611
2020's2 (50.00)2.80

Authors

AuthorsStudies
Ji, Z; Liu, Z; Xia, Z; Xu, H; Xu, R; Xu, S; Zhao, W1
Chen, C; Jiang, X; Liu, X; Qin, X; Tang, Q; Wang, B; Zhang, J; Zhang, Y; Zou, Z1
Chao, X; Chen, M; Chen, X; Chen, Z; Gao, Y; Jin, M; Liu, P; Ouyang, Y; Pi, R; Ramassamy, C; Tan, M1
Chen, CH; Chen, KL; Chen, SJ; Huang, CF; Hung, DZ; Jinn, TR; Lee, kI; Su, CC; Su, YC; Tang, FC; Tseng, TJ; Yen, CC1

Other Studies

4 other study(ies) available for pyrazolanthrone and Encephalopathy, Toxic

ArticleYear
Mechanistic study of mtROS-JNK-SOD2 signaling in bupivacaine-induced neuron oxidative stress.
    Aging, 2020, 07-13, Volume: 12, Issue:13

    Topics: Acetylcysteine; Animals; Anthracenes; Antioxidants; Bupivacaine; Cell Line, Tumor; Cyclic N-Oxides; Disease Models, Animal; Gene Knockdown Techniques; Humans; Injections, Spinal; JNK Mitogen-Activated Protein Kinases; Male; MAP Kinase Signaling System; Mitochondria; Neurons; Neurotoxicity Syndromes; Oxidative Stress; Rats; Reactive Oxygen Species; RNA, Small Interfering; Superoxide Dismutase; Transcriptional Activation

2020
Zinc Oxide Nanoparticles Induce Ferroptotic Neuronal Cell Death in vitro and in vivo.
    International journal of nanomedicine, 2020, Volume: 15

    Topics: Administration, Inhalation; Animals; Anthracenes; Cell Death; Cerebral Cortex; Cyclohexylamines; Ferroptosis; Lipid Peroxidation; Lung; Male; MAP Kinase Signaling System; Metal Nanoparticles; Mice, Inbred C57BL; Neurons; Neurotoxicity Syndromes; PC12 Cells; Phenylenediamines; Rats; Zinc Oxide

2020
Downregulation of Nrf2/HO-1 pathway and activation of JNK/c-Jun pathway are involved in homocysteic acid-induced cytotoxicity in HT-22 cells.
    Toxicology letters, 2013, Oct-23, Volume: 223, Issue:1

    Topics: Animals; Anthracenes; Apoptosis; Cell Line; Cell Survival; Dose-Response Relationship, Drug; Down-Regulation; Heme Oxygenase-1; Homocysteine; MAP Kinase Signaling System; Membrane Proteins; Mice; Mitochondria; Neurons; Neurotoxicity Syndromes; NF-E2-Related Factor 2; Oxidative Stress; Protein Kinase Inhibitors; Proto-Oncogene Proteins c-bcl-2; Reactive Oxygen Species

2013
Chloroacetic acid induced neuronal cells death through oxidative stress-mediated p38-MAPK activation pathway regulated mitochondria-dependent apoptotic signals.
    Toxicology, 2013, Jan-07, Volume: 303

    Topics: Acetates; Animals; Anthracenes; Apoptosis; Cell Line, Tumor; Dose-Response Relationship, Drug; Glutathione; Imidazoles; Mice; Mitochondria; Neuroblastoma; Neurons; Neurotoxicity Syndromes; Oxidative Stress; p38 Mitogen-Activated Protein Kinases; Pyridines

2013