Page last updated: 2024-08-18

pyrazolanthrone and Disease Exacerbation

pyrazolanthrone has been researched along with Disease Exacerbation in 5 studies

Research

Studies (5)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (20.00)29.6817
2010's3 (60.00)24.3611
2020's1 (20.00)2.80

Authors

AuthorsStudies
Hu, F; Jing, Y; Liu, D; Lv, W; Xin, T1
Banerjee, D; Banerjee, S; Ghosh, M; Lahiry, L; Roy, A1
Gao, P; Hou, B; Li, H; Li, L; Liu, D; Sun, D; Wei, Y; Zeng, W; Zhang, C; Zhang, N; Zhang, Z1
Bedja, D; Chen, Y; Cohn, RD; Dietz, HC; Doyle, JJ; Habashi, JP; Holm, TM; Judge, DP; Kim, D; Lindsay, ME; Loeys, BL; Marugan, JJ; Patnaik, S; Schoenhoff, F; Thomas, CJ; van Erp, C1
Bilter, GK; Boluro-Ajayi, M; Ennis, BW; Fultz, KE; Smith, KA; Stein, B; Westwick, JK; Zhu, D1

Other Studies

5 other study(ies) available for pyrazolanthrone and Disease Exacerbation

ArticleYear
ROCK1 knockdown inhibits non-small-cell lung cancer progression by activating the LATS2-JNK signaling pathway.
    Aging, 2020, 06-17, Volume: 12, Issue:12

    Topics: A549 Cells; Anthracenes; Apoptosis; Carcinoma, Non-Small-Cell Lung; Cell Proliferation; Disease Progression; Gene Knockdown Techniques; Humans; JNK Mitogen-Activated Protein Kinases; Lung Neoplasms; MAP Kinase Signaling System; Mitochondria; Mitochondrial Dynamics; Protein Serine-Threonine Kinases; rho-Associated Kinases; RNA, Small Interfering; Tumor Suppressor Proteins

2020
Increased cytoplasmic localization of p27(kip1) and its modulation of RhoA activity during progression of chronic myeloid leukemia.
    PloS one, 2013, Volume: 8, Issue:10

    Topics: Anthracenes; Antigens, CD34; Apoptosis; Blast Crisis; Cyclin-Dependent Kinase Inhibitor p27; Cytoplasm; Disease Progression; Fusion Proteins, bcr-abl; Gene Expression Regulation, Leukemic; Guanosine Triphosphate; Humans; K562 Cells; Leukemia, Myeloid, Chronic-Phase; Lymphocytes; MAP Kinase Kinase 4; Phosphorylation; Primary Cell Culture; Protein Kinase Inhibitors; rhoA GTP-Binding Protein; Signal Transduction

2013
Synergistic effects of c-Jun and SP1 in the promotion of TGFβ1-mediated diabetic nephropathy progression.
    Experimental and molecular pathology, 2016, Volume: 100, Issue:3

    Topics: Adult; Aged; Animals; Anthracenes; Blotting, Western; Cell Line; Diabetic Nephropathies; Disease Progression; Female; Glucose; Humans; JNK Mitogen-Activated Protein Kinases; Male; Mesangial Cells; Mice, Inbred C57BL; Microscopy, Fluorescence; Middle Aged; Mutation; Phosphorylation; Promoter Regions, Genetic; Proto-Oncogene Proteins c-jun; Reverse Transcriptase Polymerase Chain Reaction; RNA Interference; Sp1 Transcription Factor; Transforming Growth Factor beta1

2016
Noncanonical TGFβ signaling contributes to aortic aneurysm progression in Marfan syndrome mice.
    Science (New York, N.Y.), 2011, Apr-15, Volume: 332, Issue:6027

    Topics: Animals; Anthracenes; Aorta; Aortic Aneurysm; Diphenylamine; Disease Models, Animal; Disease Progression; Enzyme Activation; Losartan; MAP Kinase Signaling System; Marfan Syndrome; Mice; Mitogen-Activated Protein Kinase 1; Mitogen-Activated Protein Kinase 3; Mitogen-Activated Protein Kinase 8; Protein Kinase Inhibitors; Smad2 Protein; Smad4 Protein; Sulfonamides; Transforming Growth Factor beta

2011
Inhibition of tumor growth, angiogenesis, and tumor cell proliferation by a small molecule inhibitor of c-Jun N-terminal kinase.
    The Journal of pharmacology and experimental therapeutics, 2005, Volume: 313, Issue:1

    Topics: Angiogenesis Inhibitors; Animals; Anthracenes; Antineoplastic Agents; Blotting, Western; Cell Line, Tumor; Cell Movement; Cell Proliferation; Disease Progression; Endothelial Cells; Female; Flow Cytometry; G2 Phase; Mice; Mice, Inbred C57BL; Neoplasm Transplantation; Neoplasms

2005