Page last updated: 2024-08-18

pyrazolanthrone and Carcinoma, Transitional Cell

pyrazolanthrone has been researched along with Carcinoma, Transitional Cell in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (33.33)29.6817
2010's0 (0.00)24.3611
2020's2 (66.67)2.80

Authors

AuthorsStudies
Chen, F; Chen, S; Gao, Y; Wang, TB1
Chen, XD; Gao, S; Li, P; Qin, YM; Wu, Q; Zhou, XQ1
Deng, J; Feng, X; Geng, L; Jiang, G; Liu, J; Qian, Y; Wang, Y; Xie, H; Ye, Z; Yin, S; Zheng, S; Zhou, L1

Other Studies

3 other study(ies) available for pyrazolanthrone and Carcinoma, Transitional Cell

ArticleYear
ANLN Serves as an Oncogene in Bladder Urothelial Carcinoma via Activating JNK Signaling Pathway.
    Urologia internationalis, 2023, Volume: 107, Issue:3

    Topics: Carcinoma, Transitional Cell; Cell Line, Tumor; Cell Proliferation; Humans; MAP Kinase Signaling System; Microfilament Proteins; Oncogenes; Urinary Bladder; Urinary Bladder Neoplasms

2023
Effects of Holliday Junction-Recognition Protein-Mediated C-Jun N-Terminal Kinase/ Signal Transducer and Activator of Transcription 3 Signaling Pathway on Cell Proliferation, Cell Cycle and Cell Apoptosis in Bladder Urothelial Carcinoma.
    The Tohoku journal of experimental medicine, 2023, Feb-17, Volume: 259, Issue:3

    Topics: Apoptosis; Carcinoma, Transitional Cell; Cell Cycle; Cell Proliferation; DNA, Cruciform; Humans; JNK Mitogen-Activated Protein Kinases; Proliferating Cell Nuclear Antigen; Protein C; Signal Transduction; STAT3 Transcription Factor; Urinary Bladder; Urinary Bladder Neoplasms

2023
TLR4 signaling induces B7-H1 expression through MAPK pathways in bladder cancer cells.
    Cancer investigation, 2008, Volume: 26, Issue:8

    Topics: Anthracenes; Antigens, CD; B7-H1 Antigen; Carcinoma, Transitional Cell; Cell Line, Tumor; Chromones; Extracellular Signal-Regulated MAP Kinases; Flavonoids; Humans; Imidazoles; JNK Mitogen-Activated Protein Kinases; Lipopolysaccharide Receptors; Lipopolysaccharides; MAP Kinase Signaling System; Morpholines; Neoplasm Proteins; Phosphatidylinositol 3-Kinases; Phosphoinositide-3 Kinase Inhibitors; Phosphorylation; Protein Kinase Inhibitors; Protein Processing, Post-Translational; Proto-Oncogene Proteins c-akt; Pyridines; Toll-Like Receptor 4; Up-Regulation; Urinary Bladder Neoplasms

2008