Page last updated: 2024-08-18

pyrazolanthrone and Carcinoma, Ductal, Pancreatic

pyrazolanthrone has been researched along with Carcinoma, Ductal, Pancreatic in 2 studies

Research

Studies (2)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	0 (0.00)	18.2507
2000's	1 (50.00)	29.6817
2010's	1 (50.00)	24.3611
2020's	0 (0.00)	2.80

Authors

Authors	Studies
Alt, E; Heinemann, ML; Ilmer, M; Kettlun, C; Recio-Boiles, A; Rhea, PR; Ruetering, J; Vykoukal, J	1
Ammerpohl, O; Griep, U; Grutzmann, R; Janssen, O; Kalthoff, H; Kloppel, G; Pilarsky, C; Saeger, HD; Schniewind, B; Sipos, B; Trauzold, A	1

Other Studies

2 other study(ies) available for pyrazolanthrone and Carcinoma, Ductal, Pancreatic

Article	Year
JNK pathway inhibition selectively primes pancreatic cancer stem cells to TRAIL-induced apoptosis without affecting the physiology of normal tissue resident stem cells. Oncotarget, 2016, Mar-01, Volume: 7, Issue:9 Topics: Adenocarcinoma; Animals; Anthracenes; Antineoplastic Combined Chemotherapy Protocols; Apoptosis; Carcinoma, Pancreatic Ductal; Cell Line, Tumor; Cells, Cultured; Flow Cytometry; Humans; JNK Mitogen-Activated Protein Kinases; Male; Mice, Nude; Microscopy, Fluorescence; Neoplastic Stem Cells; Pancreatic Neoplasms; RNA Interference; Signal Transduction; Stem Cells; TNF-Related Apoptosis-Inducing Ligand; Tumor Burden; Xenograft Model Antitumor Assays	2016
Complementary effects of HDAC inhibitor 4-PB on gap junction communication and cellular export mechanisms support restoration of chemosensitivity of PDAC cells. British journal of cancer, 2007, Jan-15, Volume: 96, Issue:1 Topics: Anthracenes; Antineoplastic Combined Chemotherapy Protocols; Apoptosis; Carcinoma, Pancreatic Ductal; Caspase 8; Cell Communication; Cell Cycle; Cell Line, Tumor; Cell Proliferation; Deoxycytidine; Dose-Response Relationship, Drug; Drug Resistance, Neoplasm; Drug Screening Assays, Antitumor; Drug Synergism; Enzyme Activation; Gap Junctions; Gemcitabine; Histone Deacetylase Inhibitors; Humans; Pancreatic Neoplasms; Phenylbutyrates; Time Factors; Tumor Cells, Cultured	2007

Article

Year

JNK pathway inhibition selectively primes pancreatic cancer stem cells to TRAIL-induced apoptosis without affecting the physiology of normal tissue resident stem cells.

Oncotarget, 2016, Mar-01, Volume: 7, Issue:9

Topics: Adenocarcinoma; Animals; Anthracenes; Antineoplastic Combined Chemotherapy Protocols; Apoptosis; Carcinoma, Pancreatic Ductal; Cell Line, Tumor; Cells, Cultured; Flow Cytometry; Humans; JNK Mitogen-Activated Protein Kinases; Male; Mice, Nude; Microscopy, Fluorescence; Neoplastic Stem Cells; Pancreatic Neoplasms; RNA Interference; Signal Transduction; Stem Cells; TNF-Related Apoptosis-Inducing Ligand; Tumor Burden; Xenograft Model Antitumor Assays

2016

Complementary effects of HDAC inhibitor 4-PB on gap junction communication and cellular export mechanisms support restoration of chemosensitivity of PDAC cells.

British journal of cancer, 2007, Jan-15, Volume: 96, Issue:1

Topics: Anthracenes; Antineoplastic Combined Chemotherapy Protocols; Apoptosis; Carcinoma, Pancreatic Ductal; Caspase 8; Cell Communication; Cell Cycle; Cell Line, Tumor; Cell Proliferation; Deoxycytidine; Dose-Response Relationship, Drug; Drug Resistance, Neoplasm; Drug Screening Assays, Antitumor; Drug Synergism; Enzyme Activation; Gap Junctions; Gemcitabine; Histone Deacetylase Inhibitors; Humans; Pancreatic Neoplasms; Phenylbutyrates; Time Factors; Tumor Cells, Cultured

2007