pyrazolanthrone has been researched along with Carcinoma, Anaplastic in 5 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 3 (60.00) | 29.6817 |
2010's | 2 (40.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Huang, HY; Lai, CH; Lee, CL; Leung, PC; Soong, YK; Wang, HS; Wu, HM | 1 |
Behren, A; Iftner, T; Mühlen, S; Plinkert, PK; Simon, C | 1 |
Abid, K; Aroui, S; Brahim, S; Kenani, A | 1 |
Feng, G; Guan, Z; Li, D; Ma, J; Sun, T; Wang, L; Xia, L; Zhu, X | 1 |
Miyamoto-Yamasaki, Y; Tachibana, H; Yamada, K; Yamasaki, M | 1 |
5 other study(ies) available for pyrazolanthrone and Carcinoma, Anaplastic
Article | Year |
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Gonadotropin-releasing hormone type II (GnRH-II) agonist regulates the invasiveness of endometrial cancer cells through the GnRH-I receptor and mitogen-activated protein kinase (MAPK)-dependent activation of matrix metalloproteinase (MMP)-2.
Topics: Anthracenes; Butadienes; Carcinoma; Cell Line, Tumor; Cell Movement; Endometrial Neoplasms; Enzyme Activation; Female; Gene Knockdown Techniques; Gonadotropin-Releasing Hormone; Humans; Hydroxamic Acids; MAP Kinase Signaling System; Matrix Metalloproteinase 2; Mitogen-Activated Protein Kinases; Neoplasm Invasiveness; Nitriles; Phosphorylation; Receptors, LHRH | 2013 |
AP-1 and ERK1 but not p38 nor JNK is required for CRPV early protein 2-dependent MMP-9 promoter activation in rabbit epithelial cells.
Topics: Animals; Anthracenes; Binding Sites; Carcinoma; Cell Line; Cell Line, Tumor; Cottontail rabbit papillomavirus; Disease Models, Animal; DNA Mutational Analysis; Enzyme Inhibitors; Epithelial Cells; Flavonoids; Humans; Imidazoles; JNK Mitogen-Activated Protein Kinases; Matrix Metalloproteinase 9; Mitogen-Activated Protein Kinase 3; Molecular Sequence Data; p38 Mitogen-Activated Protein Kinases; Promoter Regions, Genetic; Pyridines; Rabbits; Signal Transduction; Transcription Factor AP-1; Transcription Factors; Viral Proteins | 2009 |
Involvement of C-jun NH2-terminal kinase and apoptosis induced factor in apoptosis induced by deglycosylated bleomycin in laryngeal carcinoma cells.
Topics: Anthracenes; Antibiotics, Antineoplastic; Apoptosis; Apoptosis Inducing Factor; Bleomycin; Carcinoma; Cell Line, Tumor; Colonic Neoplasms; Fas Ligand Protein; Humans; JNK Mitogen-Activated Protein Kinases; Laryngeal Neoplasms; Leukemia; Mitochondria; TNF-Related Apoptosis-Inducing Ligand | 2009 |
c-Jun NH2-terminal kinase activation is essential for up-regulation of LC3 during ceramide-induced autophagy in human nasopharyngeal carcinoma cells.
Topics: Anthracenes; Autophagy; Carcinoma; Cell Line, Tumor; Ceramides; Enzyme Activation; Gene Expression Regulation, Neoplastic; Humans; JNK Mitogen-Activated Protein Kinases; Microtubule-Associated Proteins; Nasopharyngeal Carcinoma; Nasopharyngeal Neoplasms; Reactive Oxygen Species; RNA, Small Interfering; Signal Transduction; Transcription, Genetic; Up-Regulation | 2011 |
Induction of endoreduplication by a JNK inhibitor SP600125 in human lung carcinoma A 549 cells.
Topics: Anthracenes; Carcinoma; Cell Cycle Proteins; Cell Division; Cell Line, Tumor; Cell Proliferation; Cyclin-Dependent Kinase Inhibitor p21; DNA; DNA Replication; G2 Phase; Gene Expression Regulation; Humans; JNK Mitogen-Activated Protein Kinases; Lung Neoplasms; Polyploidy; Protein Binding; Protein Processing, Post-Translational; Proto-Oncogene Proteins c-bcl-2; Proto-Oncogene Proteins c-mdm2; Signal Transduction; Tumor Suppressor Protein p53 | 2007 |