pyrazolanthrone has been researched along with Brain Ischemia in 15 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 6 (40.00) | 29.6817 |
2010's | 8 (53.33) | 24.3611 |
2020's | 1 (6.67) | 2.80 |
Authors | Studies |
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Ahmad, R; Ahmad, S; Ikram, M; Khan, A; Khan, MS; Kim, MO; Rehman, IUR | 1 |
Anfinogenova, YD; Atochin, DN; Schepetkin, IA; Shvedova, MV | 1 |
Chen, J; Ji, X; Jia, J; Leak, RK; Li, P; Ling, F; Liu, X; Luo, Y; Zhang, L; Zhao, H | 1 |
Cui, J; Li, L; Sun, J; Wu, X; Yang, Q | 1 |
Chen, CX; Li, JM; Li, SX; Zhang, P; Zhao, YN | 1 |
Dai, Z; Ge, M; Li, L; Li, Y; Ma, K; Si, J; Wang, S; Yao, S; Yin, J | 1 |
Arumugam, TV; Baik, SH; Chen, CL; Cheng, YL; Chunduri, P; Dheen, ST; Drummond, GR; Fann, DY; Jo, DG; Lim, YA; Lok, KZ; Sobey, CG | 1 |
Jelks, KA; O'Donnell, ME; Wallace, BK | 1 |
Arai, K; Fujiwara, N; Ji, X; Liu, X; Lo, EH; Luo, Y; Murata, Y; Seo, JH; Terasaki, Y; Yan, F | 1 |
Cao, G; Chen, J; Gao, Y; Graham, SH; Luo, Y; Signore, AP; Sun, F; Yin, W; Yin, XM | 1 |
Choi, JW; Choi, MS; Jeon, MJ; Ko, KH; Park, GH; Ryu, MK; Yoo, oK | 1 |
Guan, QH; Pei, DS; Xu, TL; Zhang, GY | 1 |
Guan, QH; Liu, XM; Pei, DS; Wang, XT; Xu, TL; Zhang, GY | 1 |
Badaut, J; Granziera, C; Hirt, L; Magistretti, PJ; Price, M; Thevenet, J; Wiegler, K | 1 |
Guan, QH; Liu, XM; Pei, DS; Sun, YF; Wang, XT; Xu, J; Zhang, GY | 1 |
1 review(s) available for pyrazolanthrone and Brain Ischemia
Article | Year |
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[JUN N-TERMINAL KINASES AND THEIR PHARMACOLOGICAL MODULATION OF ISCHE-MIC AND REPERFUSION INJURY OF THE BRAIN].
Topics: Acetonitriles; Animals; Anthracenes; Benzothiazoles; Brain; Brain Ischemia; Gene Expression Regulation; Ginsenosides; Humans; JNK Mitogen-Activated Protein Kinases; Mitogen-Activated Protein Kinase 10; Neuroprotective Agents; Oximes; Plant Extracts; Protein Kinase Inhibitors; Quinoxalines; Reperfusion Injury; Signal Transduction | 2017 |
14 other study(ies) available for pyrazolanthrone and Brain Ischemia
Article | Year |
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Inhibition of JNK Alleviates Chronic Hypoperfusion-Related Ischemia Induces Oxidative Stress and Brain Degeneration via Nrf2/HO-1 and NF-
Topics: Aging; Animals; Anthracenes; Apoptosis; Brain Ischemia; Heme Oxygenase-1; Hippocampus; JNK Mitogen-Activated Protein Kinases; Male; Memory Disorders; Mice, Inbred C57BL; Models, Biological; Neurodegenerative Diseases; Neurons; NF-E2-Related Factor 2; NF-kappa B; Oxidative Stress; Perfusion; Phosphorylation; Signal Transduction; Synapses; Up-Regulation | 2020 |
Peroxiredoxin 2 battles poly(ADP-ribose) polymerase 1- and p53-dependent prodeath pathways after ischemic injury.
Topics: Animals; Anthracenes; Apoptosis; bcl-2-Associated X Protein; Brain Ischemia; Cells, Cultured; DNA Damage; Humans; MAP Kinase Kinase 4; Mice; Mice, Inbred C57BL; Mice, Knockout; Mice, Transgenic; Neurons; Peroxiredoxins; Poly(ADP-ribose) Polymerases; Rats; Rats, Sprague-Dawley; Reactive Oxygen Species; Reperfusion Injury; Signal Transduction; Tumor Suppressor Protein p53 | 2013 |
Epigenetic Features Induced by Ischemia-Hypoxia in Cultured Rat Astrocytes.
Topics: Acetylation; Amyloid beta-Protein Precursor; Amyloid Precursor Protein Secretases; Animals; Anthracenes; Aspartic Acid Endopeptidases; Astrocytes; Brain Ischemia; Cells, Cultured; DNA (Cytosine-5-)-Methyltransferase 1; DNA (Cytosine-5-)-Methyltransferases; DNA Methylation; Epigenesis, Genetic; Hypoxia; MAP Kinase Signaling System; Methyl-CpG-Binding Protein 2; Rats, Sprague-Dawley; S-Adenosylmethionine; Up-Regulation | 2016 |
Hypertension-mediated enhancement of JNK activation in association with endoplasmic reticulum stress in rat model hippocampus with cerebral ischemia-reperfusion.
Topics: Animals; Anthracenes; Brain Ischemia; CA1 Region, Hippocampal; Disease Models, Animal; Endoplasmic Reticulum Stress; Enzyme Activation; Heat-Shock Proteins; Hippocampus; Hypertension; JNK Mitogen-Activated Protein Kinases; Male; MAP Kinase Signaling System; Rats; Rats, Inbred SHR; Rats, Inbred WKY; Reperfusion Injury; Transcription Factor CHOP | 2015 |
Transforming growth-beta 1 contributes to isoflurane postconditioning against cerebral ischemia-reperfusion injury by regulating the c-Jun N-terminal kinase signaling pathway.
Topics: Animals; Anthracenes; Behavior, Animal; Brain Ischemia; CA1 Region, Hippocampal; In Situ Nick-End Labeling; Ischemic Postconditioning; Isoflurane; JNK Mitogen-Activated Protein Kinases; Male; MAP Kinase Signaling System; Phosphorylation; Pyrazoles; Quinolines; Rats, Sprague-Dawley; Reperfusion Injury; Transforming Growth Factor beta1 | 2016 |
Evidence that NF-κB and MAPK Signaling Promotes NLRP Inflammasome Activation in Neurons Following Ischemic Stroke.
Topics: Adaptor Proteins, Signal Transducing; Animals; Anthracenes; Apoptosis Regulatory Proteins; Brain; Brain Ischemia; Butadienes; Extracellular Signal-Regulated MAP Kinases; Imidazoles; Inflammasomes; Mice; Neurons; NF-kappa B; Nitriles; NLR Family, Pyrin Domain-Containing 3 Protein; Pyridines; Signal Transduction; Stroke; Sulfones | 2018 |
Ischemia-induced stimulation of cerebral microvascular endothelial cell Na-K-Cl cotransport involves p38 and JNK MAP kinases.
Topics: Animals; Anthracenes; Blood-Brain Barrier; Brain; Brain Ischemia; Bumetanide; Cattle; Cell Hypoxia; Cells, Cultured; Endothelial Cells; Imidazoles; Infarction, Middle Cerebral Artery; JNK Mitogen-Activated Protein Kinases; Microcirculation; Microvessels; p38 Mitogen-Activated Protein Kinases; Pyrimidines; Rubidium; Signal Transduction; Sodium-Potassium-Chloride Symporters; Stroke | 2012 |
Delayed inhibition of c-Jun N-terminal kinase worsens outcomes after focal cerebral ischemia.
Topics: Animals; Anthracenes; Brain Ischemia; Cells, Cultured; Cerebral Cortex; Disease Models, Animal; Drug Administration Schedule; Enzyme Inhibitors; Infarction, Middle Cerebral Artery; JNK Mitogen-Activated Protein Kinases; Male; Matrix Metalloproteinase 9; Neovascularization, Physiologic; Rats; Rats, Sprague-Dawley | 2012 |
Neuroprotection against focal ischemic brain injury by inhibition of c-Jun N-terminal kinase and attenuation of the mitochondrial apoptosis-signaling pathway.
Topics: 14-3-3 Proteins; Animals; Anthracenes; Apoptosis; Apoptosis Regulatory Proteins; bcl-2-Associated X Protein; Bcl-2-Like Protein 11; Brain Ischemia; Carrier Proteins; Cerebral Infarction; Enzyme Inhibitors; JNK Mitogen-Activated Protein Kinases; Membrane Proteins; Mice; Mice, Inbred C57BL; Mitochondria; Neuroprotective Agents; Phosphorylation; Proto-Oncogene Proteins; Proto-Oncogene Proteins c-bcl-2; Reperfusion Injury; Signal Transduction | 2005 |
Mitogen-activated protein kinases (MAPKs) mediate SIN-1/ glucose deprivation-induced death in rat primary astrocytes.
Topics: Adenosine Triphosphate; Animals; Anthracenes; Astrocytes; Brain Ischemia; Cell Culture Techniques; Cell Death; Cells, Cultured; Enzyme Activation; Enzyme Inhibitors; Flavonoids; Glucose; Imidazoles; L-Lactate Dehydrogenase; MAP Kinase Signaling System; Mitogen-Activated Protein Kinase 1; Molsidomine; Nitric Oxide Donors; p38 Mitogen-Activated Protein Kinases; Peroxynitrous Acid; Pyridines; Rats; Rats, Sprague-Dawley | 2005 |
Brain ischemia/reperfusion-induced expression of DP5 and its interaction with Bcl-2, thus freeing Bax from Bcl-2/Bax dimmers are mediated by c-Jun N-terminal kinase (JNK) pathway.
Topics: Analysis of Variance; Animals; Anthracenes; Apoptosis Regulatory Proteins; bcl-2-Associated X Protein; Blotting, Western; Brain Ischemia; Disease Models, Animal; Enzyme Inhibitors; Gene Expression Regulation; Hippocampus; Immunoprecipitation; JNK Mitogen-Activated Protein Kinases; Male; Neuropeptides; Proto-Oncogene Proteins c-bcl-2; Rats; Rats, Sprague-Dawley; Reperfusion; Time Factors | 2006 |
Neuroprotection against ischemic brain injury by SP600125 via suppressing the extrinsic and intrinsic pathways of apoptosis.
Topics: Animals; Anthracenes; Apoptosis; bcl-2-Associated X Protein; Brain Ischemia; Caspase 3; Caspases; Cerebral Infarction; Cytochromes c; Enzyme Inhibitors; Fas Ligand Protein; Hippocampus; JNK Mitogen-Activated Protein Kinases; Male; Membrane Glycoproteins; Nerve Degeneration; Neuroprotective Agents; Proto-Oncogene Proteins c-jun; Rats; Rats, Sprague-Dawley; Signal Transduction; Tumor Necrosis Factors | 2006 |
Thrombin-induced ischemic tolerance is prevented by inhibiting c-jun N-terminal kinase.
Topics: Animals; Anthracenes; Brain; Brain Ischemia; Cell Death; Enzyme Inhibitors; Hippocampus; Infarction, Middle Cerebral Artery; Ischemic Preconditioning; JNK Mitogen-Activated Protein Kinases; Male; Mice; Mice, Inbred ICR; Organ Culture Techniques; Phosphorylation; Proto-Oncogene Proteins c-jun; Rats; Rats, Sprague-Dawley; Thrombin | 2007 |
Opposing effects of Bad phosphorylation at two distinct sites by Akt1 and JNK1/2 on ischemic brain injury.
Topics: 14-3-3 Proteins; Animals; Anthracenes; Apoptosis; bcl-Associated Death Protein; bcl-X Protein; Brain Ischemia; Enzyme Activation; Hippocampus; Male; Mitochondria; Mitogen-Activated Protein Kinase 8; Mitogen-Activated Protein Kinase 9; Neuroprotective Agents; Organometallic Compounds; Phosphorylation; Phosphoserine; Protein Binding; Proto-Oncogene Proteins c-akt; Rats; Rats, Sprague-Dawley | 2007 |