Page last updated: 2024-08-18

pyrazolanthrone and Bile Duct Cancer

pyrazolanthrone has been researched along with Bile Duct Cancer in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (33.33)29.6817
2010's2 (66.67)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Hong, HJ; Hwang, H; Kim, H; Lee, H1
Ai, X; Chen, X; Liang, H; Lin, Y; Lu, Y; Zhang, B1
Chayama, K; Kuwahara, K; Miyata, H; Sasaki, T; Serikawa, M1

Other Studies

3 other study(ies) available for pyrazolanthrone and Bile Duct Cancer

ArticleYear
L1 Cell Adhesion Molecule Promotes Migration and Invasion via JNK Activation in Extrahepatic Cholangiocarcinoma Cells with Activating
    Molecules and cells, 2017, May-31, Volume: 40, Issue:5

    Topics: Anthracenes; Bile Duct Neoplasms; Cell Line, Tumor; Cell Movement; Cell Proliferation; Cholangiocarcinoma; Enzyme Activation; Gene Knockdown Techniques; Humans; MAP Kinase Kinase 4; MAP Kinase Signaling System; Mutation; Neural Cell Adhesion Molecule L1; Protein Kinase Inhibitors; Proto-Oncogene Proteins p21(ras); RNA, Small Interfering

2017
JNK inhibitor SP600125 enhances TGF-β-induced apoptosis of RBE human cholangiocarcinoma cells in a Smad-dependent manner.
    Molecular medicine reports, 2013, Volume: 8, Issue:6

    Topics: Anthracenes; Apoptosis; Bile Duct Neoplasms; Bile Ducts, Intrahepatic; Caspases; Cell Line, Tumor; Cholangiocarcinoma; Enzyme Activation; Humans; JNK Mitogen-Activated Protein Kinases; Phosphorylation; Protein Kinase Inhibitors; Signal Transduction; Smad Proteins; Transforming Growth Factor beta

2013
The effects of ZD1839 (Iressa), a highly selective EGFR tyrosine kinase inhibitor, as a radiosensitiser in bile duct carcinoma cell lines.
    International journal of oncology, 2006, Volume: 28, Issue:4

    Topics: Anthracenes; Bile Duct Neoplasms; Butadienes; Cell Line, Tumor; Cell Proliferation; Cell Survival; Chromones; Clone Cells; Dose-Response Relationship, Drug; Enzyme Activation; Enzyme Inhibitors; ErbB Receptors; Gefitinib; Humans; Imidazoles; JNK Mitogen-Activated Protein Kinases; Mitogen-Activated Protein Kinases; Morpholines; Nitriles; Oncogene Protein v-akt; p38 Mitogen-Activated Protein Kinases; Phosphorylation; Protein Kinase Inhibitors; Pyridines; Quinazolines; Radiation-Sensitizing Agents

2006