pyrazolanthrone has been researched along with Asthma in 6 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 2 (33.33) | 29.6817 |
2010's | 3 (50.00) | 24.3611 |
2020's | 1 (16.67) | 2.80 |
Authors | Studies |
---|---|
Chen, BC; Chen, CL; Cheng, WH; Kao, SY; Lin, CH; Lin, LY; Yuliani, FS | 1 |
Fang, L; Liu, RY; Shen, QY; Wu, HM | 1 |
Chen, B; Fan, XY; Wu, HM; Wu, SS; Zhang, LL; Zhang, SQ | 1 |
Dakhama, A; Domenico, J; Gelfand, EW; Joetham, A; Ohnishi, H; Okamoto, M; Schedel, M; Takeda, K | 1 |
Adcock, IM; Bennett, BL; Chung, KF; Eynott, PR; Groneberg, DA; Leung, SY; Nath, P; Noble, A; Xu, L | 1 |
Adcock, IM; Bennett, BL; Chung, KF; Eynott, P; Leung, SY; Nath, P | 1 |
6 other study(ies) available for pyrazolanthrone and Asthma
Article | Year |
---|---|
Amphiregulin induces CCN2 and fibronectin expression by TGF-β through EGFR-dependent pathway in lung epithelial cells.
Topics: Amphiregulin; Animals; Asthma; Epithelial Cells; Epithelial-Mesenchymal Transition; ErbB Receptors; Fibronectins; Fibrosis; Humans; Lung; Mice; Ovalbumin; RNA, Small Interfering; Transcription Factor AP-1; Transforming Growth Factor beta; Transforming Growth Factor beta1 | 2022 |
SP600125 promotes resolution of allergic airway inflammation via TLR9 in an OVA-induced murine acute asthma model.
Topics: Acute Disease; Animals; Anthracenes; Asthma; Bronchoalveolar Lavage Fluid; Cell Count; Cytokines; Disease Models, Animal; Female; Hypersensitivity; Immunoglobulin E; Inflammation; JNK Mitogen-Activated Protein Kinases; Lung; Mice, Inbred C57BL; Mucus; Ovalbumin; Phosphorylation; Toll-Like Receptor 9 | 2015 |
LPS Cooperates with Poly-L-Arginine to Promote IL-6 and IL-8 Release via the JNK Signaling Pathway in NCI-H292 Cells.
Topics: Anthracenes; Asthma; Cell Line, Tumor; Drug Synergism; Enzyme-Linked Immunosorbent Assay; Humans; Interleukin-6; Interleukin-8; JNK Mitogen-Activated Protein Kinases; Lipopolysaccharides; MAP Kinase Signaling System; Peptides; Phosphorylation | 2016 |
Loss of T regulatory cell suppression following signaling through glucocorticoid-induced tumor necrosis receptor (GITR) is dependent on c-Jun N-terminal kinase activation.
Topics: Animals; Anthracenes; Asthma; Autoimmune Diseases; Cell Proliferation; Enzyme Activation; Female; Glucocorticoid-Induced TNFR-Related Protein; Immune Tolerance; Lung; MAP Kinase Kinase 4; MAP Kinase Kinase 7; MAP Kinase Signaling System; Mice; Mice, Knockout; NF-kappa B; p38 Mitogen-Activated Protein Kinases; Phosphorylation; Protein Kinase Inhibitors; T-Lymphocytes, Regulatory | 2012 |
Effect of an inhibitor of Jun N-terminal protein kinase, SP600125, in single allergen challenge in sensitized rats.
Topics: Allergens; Animals; Anthracenes; Asthma; Bronchoalveolar Lavage Fluid; Cell Count; Cytokines; Enzyme Activation; Eosinophils; JNK Mitogen-Activated Protein Kinases; Leukocyte Count; Leukocytes, Mononuclear; Lung; Macrophages; Male; MAP Kinase Kinase 4; Mitogen-Activated Protein Kinase Kinases; Mitogen-Activated Protein Kinases; Models, Animal; Neutrophils; Ovalbumin; p38 Mitogen-Activated Protein Kinases; Rats; Rats, Inbred BN | 2004 |
Potential role of c-Jun NH2-terminal kinase in allergic airway inflammation and remodelling: effects of SP600125.
Topics: Animals; Anthracenes; Asthma; Bronchial Hyperreactivity; Dose-Response Relationship, Drug; JNK Mitogen-Activated Protein Kinases; Lung; Male; Mice; Mice, Inbred BALB C | 2005 |