pyrazolanthrone has been researched along with Alloxan Diabetes in 7 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (14.29) | 29.6817 |
| 2010's | 5 (71.43) | 24.3611 |
| 2020's | 1 (14.29) | 2.80 |
| Authors | Studies |
|---|---|
| Jing, GC; Liu, D; Liu, YQ; Zhang, MR | 1 |
| Hein, TW; Kuo, L; Ren, Y; Thengchaisri, N; Tsai, SH; Xu, W; Xu, X | 1 |
| Fukuda, D; Kusunose, K; Pham, PT; Sata, M; Shimabukuro, M; Soeki, T; Yagi, S; Yamada, H | 1 |
| Cai, L; Li, X; Liang, G; Liu, Q; Pan, Y; Ren, L; Tang, L; Wang, J; Wang, Y; Zhang, X; Zhao, Y | 1 |
| Cai, L; Liang, G; Liu, Q; Liu, Y; Miao, X; Sun, J; Tan, Y; Wang, Y; Zheng, Y; Zhou, S | 1 |
| Bai, X; Geng, J; Li, X; Tian, J; Yang, F | 1 |
| Reece, EA; Yang, P; Zhao, Z | 1 |
7 other study(ies) available for pyrazolanthrone and Alloxan Diabetes
| Article | Year |
|---|---|
Nao-Fu-Cong ameliorates diabetic cognitive dysfunction by inhibition of JNK/CHOP/Bcl2-mediated apoptosis in vivo and in vitro.
Topics: Acetylcysteine; Animals; Anthracenes; Apoptosis; Cognitive Dysfunction; Diabetes Mellitus, Experimental; Dose-Response Relationship, Drug; Drugs, Chinese Herbal; Hippocampus; Male; MAP Kinase Signaling System; Membrane Potential, Mitochondrial; Neurons; Neuroprotective Agents; Proto-Oncogene Proteins c-bcl-2; Random Allocation; Rats; Transcription Factor CHOP | 2020 |
Requisite roles of LOX-1, JNK, and arginase in diabetes-induced endothelial vasodilator dysfunction of porcine coronary arterioles.
Topics: Animals; Anthracenes; Arginase; Arterioles; Coronary Vessels; Cyclic N-Oxides; Diabetes Mellitus, Experimental; Dioxanes; Imidazoles; In Vitro Techniques; JNK Mitogen-Activated Protein Kinases; Male; Nitric Oxide; Pyridines; Scavenger Receptors, Class E; Serotonin; Spin Labels; Swine; Thiazoles; Vasodilation | 2019 |
Rivaroxaban, a specific FXa inhibitor, improved endothelium-dependent relaxation of aortic segments in diabetic mice.
Topics: Animals; Anthracenes; Aorta; Cell Line; Diabetes Mellitus, Experimental; Diabetic Angiopathies; Endothelium, Vascular; Factor Xa; Factor Xa Inhibitors; Humans; JNK Mitogen-Activated Protein Kinases; Male; Mice; Mice, Knockout; Nitric Oxide Synthase Type III; Phosphorylation; Receptor, PAR-2; Rivaroxaban; Streptozocin; Vasodilation | 2019 |
Targeting JNK by a new curcumin analog to inhibit NF-kB-mediated expression of cell adhesion molecules attenuates renal macrophage infiltration and injury in diabetic mice.
Topics: Animals; Anthracenes; Cell Line; Chemokine CCL2; Curcumin; Diabetes Mellitus, Experimental; Diabetic Nephropathies; Enzyme Inhibitors; Gene Expression Regulation; Intercellular Adhesion Molecule-1; Male; MAP Kinase Kinase 4; MAP Kinase Signaling System; Mice; NF-kappa B; Phosphorylation; Vascular Cell Adhesion Molecule-1 | 2013 |
Inhibition of JNK by compound C66 prevents pathological changes of the aorta in STZ-induced diabetes.
Topics: Animals; Anthracenes; Anti-Inflammatory Agents, Non-Steroidal; Aorta; Apoptosis; Blotting, Western; Cell Proliferation; Cells, Cultured; Curcumin; Diabetes Mellitus, Experimental; Diabetic Cardiomyopathies; JNK Mitogen-Activated Protein Kinases; Male; Mast Cells; Mice; Mice, Inbred C57BL; NF-E2-Related Factor 2; Phosphorylation; Real-Time Polymerase Chain Reaction; Reverse Transcriptase Polymerase Chain Reaction; RNA, Messenger | 2014 |
VEGF-A inhibition ameliorates podocyte apoptosis via repression of activating protein 1 in diabetes.
Topics: Albuminuria; Angiogenesis Inhibitors; Animals; Anthracenes; Antibodies, Monoclonal, Humanized; Apoptosis; Bevacizumab; Blood Glucose; Creatinine; Diabetes Mellitus, Experimental; HEK293 Cells; Humans; Kidney; Podocytes; Proto-Oncogene Proteins c-bcl-2; Proto-Oncogene Proteins c-fos; Proto-Oncogene Proteins c-jun; Rats, Sprague-Dawley; RNA, Small Interfering; Streptozocin; Transcription Factor AP-1; Transfection; Vascular Endothelial Growth Factor A | 2014 |
Involvement of c-Jun N-terminal kinases activation in diabetic embryopathy.
Topics: Animals; Anthracenes; Diabetes Mellitus, Experimental; Dose-Response Relationship, Drug; Embryo, Mammalian; Enzyme Activation; Female; Genotype; Hyperglycemia; JNK Mitogen-Activated Protein Kinases; Male; Mice; Mice, Inbred C57BL; Mice, Knockout; Mitogen-Activated Protein Kinase 9; Pregnancy; Pregnancy in Diabetics; Rats; Rats, Sprague-Dawley; Time Factors; Tissue Culture Techniques | 2007 |