pyrazolanthrone has been researched along with Adjuvant Arthritis in 5 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 3 (60.00) | 29.6817 |
2010's | 2 (40.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Goto, K; Hiramoto, K; Ooi, K | 1 |
Dai, SM; Qin, YH; Ren, D; Shen, Q; Tang, GS; Wang, ZW; Zhang, J | 1 |
Han, H; Lee, KS; Rong, W; Zhang, G | 1 |
Abbas, S; Abu-Amer, Y; Dai, S; Hirayama, T; Yamanaka, Y | 1 |
Bennett, B; Boyle, DL; Chang, L; Firestein, GS; Han, Z; Karin, M; Manning, AM; Yang, L | 1 |
5 other study(ies) available for pyrazolanthrone and Adjuvant Arthritis
Article | Year |
---|---|
Th2 and Th17 Induce Dry Skin in a Mouse Model of Arthritis.
Topics: Animals; Anthracenes; Arthritis, Experimental; Arylsulfonates; Biomarkers; Gene Expression Regulation; Interleukin-17; Interleukin-6; Interleukin-7; Mice; Mice, Inbred DBA; Random Allocation; Skin Physiological Phenomena; Sulfonium Compounds; Th17 Cells; Th2 Cells; Tumor Necrosis Factor-alpha; Water Loss, Insensible | 2019 |
HMGB1 enhances the proinflammatory activity of lipopolysaccharide by promoting the phosphorylation of MAPK p38 through receptor for advanced glycation end products.
Topics: Animals; Anthracenes; Arthritis, Experimental; Cytokines; Enzyme Inhibitors; Female; Flavonoids; Glycation End Products, Advanced; HMGB1 Protein; Imidazoles; Lipopolysaccharides; Macrophages, Peritoneal; Mice; Mice, Inbred C57BL; Mice, Inbred DBA; Mice, Knockout; p38 Mitogen-Activated Protein Kinases; Phosphorylation; Pyridines; Receptor for Advanced Glycation End Products; Receptors, Immunologic; Toll-Like Receptor 2; Toll-Like Receptor 4; Up-Regulation | 2009 |
Different roles of peripheral mitogen-activated protein kinases in carrageenan-induced arthritic pain and arthritis in rats.
Topics: Animals; Anthracenes; Arthritis, Experimental; Carrageenan; Extracellular Signal-Regulated MAP Kinases; Flavonoids; Imidazoles; Male; MAP Kinase Signaling System; Mitogen-Activated Protein Kinases; Pyridines; Random Allocation; Rats; Rats, Sprague-Dawley | 2012 |
Inhibition of inflammatory bone erosion by constitutively active STAT-6 through blockade of JNK and NF-kappaB activation.
Topics: Animals; Anthracenes; Arthritis, Experimental; Bone Marrow Cells; Bone Resorption; Carrier Proteins; Cells, Cultured; Disease Models, Animal; Dose-Response Relationship, Drug; Drug Combinations; JNK Mitogen-Activated Protein Kinases; Leg Bones; Macrophages; MAP Kinase Kinase 4; Membrane Glycoproteins; Mice; Mice, Inbred BALB C; Mice, Knockout; Mitogen-Activated Protein Kinase Kinases; NF-kappa B; Osteoclasts; RANK Ligand; Receptor Activator of Nuclear Factor-kappa B; Signal Transduction; STAT6 Transcription Factor; Trans-Activators | 2005 |
c-Jun N-terminal kinase is required for metalloproteinase expression and joint destruction in inflammatory arthritis.
Topics: Activating Transcription Factor 2; Animals; Anthracenes; Arthritis, Experimental; Cells, Cultured; Collagenases; Cyclic AMP Response Element-Binding Protein; Enzyme Induction; Enzyme Inhibitors; Flavonoids; Interleukin-1; Isoenzymes; MAP Kinase Kinase Kinase 1; MAP Kinase Signaling System; Matrix Metalloproteinase 13; Mice; Mice, Knockout; Mitogen-Activated Protein Kinase 10; Mitogen-Activated Protein Kinase 3; Mitogen-Activated Protein Kinase 8; Mitogen-Activated Protein Kinase 9; Mitogen-Activated Protein Kinases; p38 Mitogen-Activated Protein Kinases; Phosphorylation; Protein Processing, Post-Translational; Protein Serine-Threonine Kinases; Protein-Tyrosine Kinases; Proto-Oncogene Proteins c-jun; Rats; Rats, Inbred Lew; RNA, Messenger; Synovial Membrane; Transcription Factor AP-1; Transcription Factors | 2001 |