Page last updated: 2024-08-18

pyrazolanthrone and Adjuvant Arthritis

pyrazolanthrone has been researched along with Adjuvant Arthritis in 5 studies

Research

Studies (5)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's3 (60.00)29.6817
2010's2 (40.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Goto, K; Hiramoto, K; Ooi, K1
Dai, SM; Qin, YH; Ren, D; Shen, Q; Tang, GS; Wang, ZW; Zhang, J1
Han, H; Lee, KS; Rong, W; Zhang, G1
Abbas, S; Abu-Amer, Y; Dai, S; Hirayama, T; Yamanaka, Y1
Bennett, B; Boyle, DL; Chang, L; Firestein, GS; Han, Z; Karin, M; Manning, AM; Yang, L1

Other Studies

5 other study(ies) available for pyrazolanthrone and Adjuvant Arthritis

ArticleYear
Th2 and Th17 Induce Dry Skin in a Mouse Model of Arthritis.
    Biological & pharmaceutical bulletin, 2019, Volume: 42, Issue:3

    Topics: Animals; Anthracenes; Arthritis, Experimental; Arylsulfonates; Biomarkers; Gene Expression Regulation; Interleukin-17; Interleukin-6; Interleukin-7; Mice; Mice, Inbred DBA; Random Allocation; Skin Physiological Phenomena; Sulfonium Compounds; Th17 Cells; Th2 Cells; Tumor Necrosis Factor-alpha; Water Loss, Insensible

2019
HMGB1 enhances the proinflammatory activity of lipopolysaccharide by promoting the phosphorylation of MAPK p38 through receptor for advanced glycation end products.
    Journal of immunology (Baltimore, Md. : 1950), 2009, Nov-15, Volume: 183, Issue:10

    Topics: Animals; Anthracenes; Arthritis, Experimental; Cytokines; Enzyme Inhibitors; Female; Flavonoids; Glycation End Products, Advanced; HMGB1 Protein; Imidazoles; Lipopolysaccharides; Macrophages, Peritoneal; Mice; Mice, Inbred C57BL; Mice, Inbred DBA; Mice, Knockout; p38 Mitogen-Activated Protein Kinases; Phosphorylation; Pyridines; Receptor for Advanced Glycation End Products; Receptors, Immunologic; Toll-Like Receptor 2; Toll-Like Receptor 4; Up-Regulation

2009
Different roles of peripheral mitogen-activated protein kinases in carrageenan-induced arthritic pain and arthritis in rats.
    Anesthesia and analgesia, 2012, Volume: 115, Issue:5

    Topics: Animals; Anthracenes; Arthritis, Experimental; Carrageenan; Extracellular Signal-Regulated MAP Kinases; Flavonoids; Imidazoles; Male; MAP Kinase Signaling System; Mitogen-Activated Protein Kinases; Pyridines; Random Allocation; Rats; Rats, Sprague-Dawley

2012
Inhibition of inflammatory bone erosion by constitutively active STAT-6 through blockade of JNK and NF-kappaB activation.
    Arthritis and rheumatism, 2005, Volume: 52, Issue:9

    Topics: Animals; Anthracenes; Arthritis, Experimental; Bone Marrow Cells; Bone Resorption; Carrier Proteins; Cells, Cultured; Disease Models, Animal; Dose-Response Relationship, Drug; Drug Combinations; JNK Mitogen-Activated Protein Kinases; Leg Bones; Macrophages; MAP Kinase Kinase 4; Membrane Glycoproteins; Mice; Mice, Inbred BALB C; Mice, Knockout; Mitogen-Activated Protein Kinase Kinases; NF-kappa B; Osteoclasts; RANK Ligand; Receptor Activator of Nuclear Factor-kappa B; Signal Transduction; STAT6 Transcription Factor; Trans-Activators

2005
c-Jun N-terminal kinase is required for metalloproteinase expression and joint destruction in inflammatory arthritis.
    The Journal of clinical investigation, 2001, Volume: 108, Issue:1

    Topics: Activating Transcription Factor 2; Animals; Anthracenes; Arthritis, Experimental; Cells, Cultured; Collagenases; Cyclic AMP Response Element-Binding Protein; Enzyme Induction; Enzyme Inhibitors; Flavonoids; Interleukin-1; Isoenzymes; MAP Kinase Kinase Kinase 1; MAP Kinase Signaling System; Matrix Metalloproteinase 13; Mice; Mice, Knockout; Mitogen-Activated Protein Kinase 10; Mitogen-Activated Protein Kinase 3; Mitogen-Activated Protein Kinase 8; Mitogen-Activated Protein Kinase 9; Mitogen-Activated Protein Kinases; p38 Mitogen-Activated Protein Kinases; Phosphorylation; Protein Processing, Post-Translational; Protein Serine-Threonine Kinases; Protein-Tyrosine Kinases; Proto-Oncogene Proteins c-jun; Rats; Rats, Inbred Lew; RNA, Messenger; Synovial Membrane; Transcription Factor AP-1; Transcription Factors

2001