protocatechualdehyde has been researched along with Injury, Ischemia-Reperfusion in 4 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 1 (25.00) | 24.3611 |
| 2020's | 3 (75.00) | 2.80 |
| Authors | Studies |
|---|---|
| Ding, ZS; Guo, Y; He, Y; Jin, B; Wan, HT; Wang, Y; Yang, JH; Zhou, HF | 1 |
| Chen, P; Duan, X; Luo, Y; Yang, L | 1 |
| He, Y; Li, W; Shao, C; Wan, H; Yang, J; Zeng, J; Zeng, M; Zhao, X; Zhou, H | 1 |
| Ding, Y; Duan, J; Guan, Y; Guo, C; Jia, N; Wang, S; Wei, G; Wen, A; Xi, M; Yin, Y; Zhu, Y | 1 |
4 other study(ies) available for protocatechualdehyde and Injury, Ischemia-Reperfusion
| Article | Year |
|---|---|
Protocatechuic aldehyde prevents ischemic injury by attenuating brain microvascular endothelial cell pyroptosis via lncRNA Xist.
Topics: Animals; Benzaldehydes; Brain; Catechols; Endothelial Cells; Neuroprotective Agents; Pyroptosis; Rats; Rats, Sprague-Dawley; Reperfusion Injury; RNA, Long Noncoding | 2022 |
Metabolomic analysis and pharmacological validation of the cerebral protective effect of 3,4‑dihydroxybenzaldehyde on cerebral ischemia‑reperfusion injury.
Topics: Adenosine Triphosphate; Animals; Apoptosis; Brain Ischemia; Infarction, Middle Cerebral Artery; Rats; Reperfusion Injury; Uridine Diphosphate | 2023 |
Protocatechudehyde improves mitochondrial energy metabolism through the HIF1α/PDK1 signaling pathway to mitigate ischemic stroke-elicited internal capsule injury.
Topics: Animals; Apoptosis; Benzaldehydes; Brain Ischemia; Catechols; Cell Survival; Disease Models, Animal; Dose-Response Relationship, Drug; Energy Metabolism; Hypoxia-Inducible Factor 1, alpha Subunit; Internal Capsule; Ischemic Stroke; Male; Membrane Potential, Mitochondrial; Neuroprotective Agents; PC12 Cells; Pyruvate Dehydrogenase Acetyl-Transferring Kinase; Rats; Rats, Sprague-Dawley; Reperfusion Injury; Signal Transduction | 2021 |
Protocatechualdehyde Protects Against Cerebral Ischemia-Reperfusion-Induced Oxidative Injury Via Protein Kinase Cε/Nrf2/HO-1 Pathway.
Topics: Animals; Anticoagulants; Benzaldehydes; Brain Ischemia; Catechols; Cell Line, Tumor; Dose-Response Relationship, Drug; Heme Oxygenase-1; Humans; Male; Membrane Proteins; Neuroprotective Agents; NF-E2-Related Factor 2; Oxidative Stress; Protein Kinase C-epsilon; Random Allocation; Rats; Rats, Sprague-Dawley; Reperfusion Injury; Signal Transduction | 2017 |