propranolol has been researched along with Immunologic Deficiency Syndromes in 2 studies
Propranolol: A widely used non-cardioselective beta-adrenergic antagonist. Propranolol has been used for MYOCARDIAL INFARCTION; ARRHYTHMIA; ANGINA PECTORIS; HYPERTENSION; HYPERTHYROIDISM; MIGRAINE; PHEOCHROMOCYTOMA; and ANXIETY but adverse effects instigate replacement by newer drugs.
propranolol : A propanolamine that is propan-2-ol substituted by a propan-2-ylamino group at position 1 and a naphthalen-1-yloxy group at position 3.
Immunologic Deficiency Syndromes: Syndromes in which there is a deficiency or defect in the mechanisms of immunity, either cellular or humoral.
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (50.00) | 29.6817 |
| 2010's | 1 (50.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Wang, H | 1 |
| Deng, QW | 1 |
| Peng, AN | 1 |
| Xing, FL | 1 |
| Zuo, L | 1 |
| Li, S | 1 |
| Gu, ZT | 1 |
| Yan, FL | 1 |
| Prass, K | 1 |
| Meisel, C | 1 |
| Höflich, C | 1 |
| Braun, J | 1 |
| Halle, E | 1 |
| Wolf, T | 1 |
| Ruscher, K | 1 |
| Victorov, IV | 1 |
| Priller, J | 1 |
| Dirnagl, U | 1 |
| Volk, HD | 1 |
| Meisel, A | 1 |
| Trial | Phase | Enrollment | Study Type | Start Date | Status | ||
|---|---|---|---|---|---|---|---|
| Role of Th1-lymphocytes in the Development of Vascular Cognitive Impairment in Young Stroke Patients[NCT03725137] | 77 participants (Anticipated) | Observational | 2020-01-31 | Not yet recruiting | |||
| Predictors of Early Chest Infection in Acute Ischemic Stroke[NCT00906542] | 530 participants (Actual) | Observational | 2009-05-31 | Completed | |||
| [information is prepared from clinicaltrials.gov, extracted Sep-2024] | |||||||
2 other studies available for propranolol and Immunologic Deficiency Syndromes
| Article | Year |
|---|---|
β-arrestin2 functions as a key regulator in the sympathetic-triggered immunodepression after stroke.
Topics: Animals; beta-Arrestin 2; Brain Infarction; Cell Line, Transformed; Cytokines; Disease Models, Anima | 2018 |
Stroke-induced immunodeficiency promotes spontaneous bacterial infections and is mediated by sympathetic activation reversal by poststroke T helper cell type 1-like immunostimulation.
Topics: Animals; Bacteremia; Bacterial Infections; Disease Models, Animal; Female; Humans; Immunization; Imm | 2003 |
Stroke-induced immunodeficiency promotes spontaneous bacterial infections and is mediated by sympathetic activation reversal by poststroke T helper cell type 1-like immunostimulation.
Topics: Animals; Bacteremia; Bacterial Infections; Disease Models, Animal; Female; Humans; Immunization; Imm | 2003 |
Stroke-induced immunodeficiency promotes spontaneous bacterial infections and is mediated by sympathetic activation reversal by poststroke T helper cell type 1-like immunostimulation.
Topics: Animals; Bacteremia; Bacterial Infections; Disease Models, Animal; Female; Humans; Immunization; Imm | 2003 |
Stroke-induced immunodeficiency promotes spontaneous bacterial infections and is mediated by sympathetic activation reversal by poststroke T helper cell type 1-like immunostimulation.
Topics: Animals; Bacteremia; Bacterial Infections; Disease Models, Animal; Female; Humans; Immunization; Imm | 2003 |