Page last updated: 2024-11-03

propranolol and Immunologic Deficiency Syndromes

propranolol has been researched along with Immunologic Deficiency Syndromes in 2 studies

Propranolol: A widely used non-cardioselective beta-adrenergic antagonist. Propranolol has been used for MYOCARDIAL INFARCTION; ARRHYTHMIA; ANGINA PECTORIS; HYPERTENSION; HYPERTHYROIDISM; MIGRAINE; PHEOCHROMOCYTOMA; and ANXIETY but adverse effects instigate replacement by newer drugs.
propranolol : A propanolamine that is propan-2-ol substituted by a propan-2-ylamino group at position 1 and a naphthalen-1-yloxy group at position 3.

Immunologic Deficiency Syndromes: Syndromes in which there is a deficiency or defect in the mechanisms of immunity, either cellular or humoral.

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (50.00)29.6817
2010's1 (50.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Wang, H1
Deng, QW1
Peng, AN1
Xing, FL1
Zuo, L1
Li, S1
Gu, ZT1
Yan, FL1
Prass, K1
Meisel, C1
Höflich, C1
Braun, J1
Halle, E1
Wolf, T1
Ruscher, K1
Victorov, IV1
Priller, J1
Dirnagl, U1
Volk, HD1
Meisel, A1

Clinical Trials (2)

Trial Overview

TrialPhaseEnrollmentStudy TypeStart DateStatus
Role of Th1-lymphocytes in the Development of Vascular Cognitive Impairment in Young Stroke Patients[NCT03725137]77 participants (Anticipated)Observational2020-01-31Not yet recruiting
Predictors of Early Chest Infection in Acute Ischemic Stroke[NCT00906542]530 participants (Actual)Observational2009-05-31Completed
[information is prepared from clinicaltrials.gov, extracted Sep-2024]

Other Studies

2 other studies available for propranolol and Immunologic Deficiency Syndromes

ArticleYear
β-arrestin2 functions as a key regulator in the sympathetic-triggered immunodepression after stroke.
    Journal of neuroinflammation, 2018, Apr-10, Volume: 15, Issue:1

    Topics: Animals; beta-Arrestin 2; Brain Infarction; Cell Line, Transformed; Cytokines; Disease Models, Anima

2018
Stroke-induced immunodeficiency promotes spontaneous bacterial infections and is mediated by sympathetic activation reversal by poststroke T helper cell type 1-like immunostimulation.
    The Journal of experimental medicine, 2003, Sep-01, Volume: 198, Issue:5

    Topics: Animals; Bacteremia; Bacterial Infections; Disease Models, Animal; Female; Humans; Immunization; Imm

2003
Stroke-induced immunodeficiency promotes spontaneous bacterial infections and is mediated by sympathetic activation reversal by poststroke T helper cell type 1-like immunostimulation.
    The Journal of experimental medicine, 2003, Sep-01, Volume: 198, Issue:5

    Topics: Animals; Bacteremia; Bacterial Infections; Disease Models, Animal; Female; Humans; Immunization; Imm

2003
Stroke-induced immunodeficiency promotes spontaneous bacterial infections and is mediated by sympathetic activation reversal by poststroke T helper cell type 1-like immunostimulation.
    The Journal of experimental medicine, 2003, Sep-01, Volume: 198, Issue:5

    Topics: Animals; Bacteremia; Bacterial Infections; Disease Models, Animal; Female; Humans; Immunization; Imm

2003
Stroke-induced immunodeficiency promotes spontaneous bacterial infections and is mediated by sympathetic activation reversal by poststroke T helper cell type 1-like immunostimulation.
    The Journal of experimental medicine, 2003, Sep-01, Volume: 198, Issue:5

    Topics: Animals; Bacteremia; Bacterial Infections; Disease Models, Animal; Female; Humans; Immunization; Imm

2003