propranolol has been researched along with Bacteremia in 1 studies
Propranolol: A widely used non-cardioselective beta-adrenergic antagonist. Propranolol has been used for MYOCARDIAL INFARCTION; ARRHYTHMIA; ANGINA PECTORIS; HYPERTENSION; HYPERTHYROIDISM; MIGRAINE; PHEOCHROMOCYTOMA; and ANXIETY but adverse effects instigate replacement by newer drugs.
propranolol : A propanolamine that is propan-2-ol substituted by a propan-2-ylamino group at position 1 and a naphthalen-1-yloxy group at position 3.
Bacteremia: The presence of viable bacteria circulating in the blood. Fever, chills, tachycardia, and tachypnea are common acute manifestations of bacteremia. The majority of cases are seen in already hospitalized patients, most of whom have underlying diseases or procedures which render their bloodstreams susceptible to invasion.
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (100.00) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Prass, K | 1 |
| Meisel, C | 1 |
| Höflich, C | 1 |
| Braun, J | 1 |
| Halle, E | 1 |
| Wolf, T | 1 |
| Ruscher, K | 1 |
| Victorov, IV | 1 |
| Priller, J | 1 |
| Dirnagl, U | 1 |
| Volk, HD | 1 |
| Meisel, A | 1 |
| Trial | Phase | Enrollment | Study Type | Start Date | Status | ||
|---|---|---|---|---|---|---|---|
| Role of Th1-lymphocytes in the Development of Vascular Cognitive Impairment in Young Stroke Patients[NCT03725137] | 77 participants (Anticipated) | Observational | 2020-01-31 | Not yet recruiting | |||
| Predictors of Early Chest Infection in Acute Ischemic Stroke[NCT00906542] | 530 participants (Actual) | Observational | 2009-05-31 | Completed | |||
| [information is prepared from clinicaltrials.gov, extracted Sep-2024] | |||||||
1 other study available for propranolol and Bacteremia
| Article | Year |
|---|---|
Stroke-induced immunodeficiency promotes spontaneous bacterial infections and is mediated by sympathetic activation reversal by poststroke T helper cell type 1-like immunostimulation.
Topics: Animals; Bacteremia; Bacterial Infections; Disease Models, Animal; Female; Humans; Immunization; Imm | 2003 |
Stroke-induced immunodeficiency promotes spontaneous bacterial infections and is mediated by sympathetic activation reversal by poststroke T helper cell type 1-like immunostimulation.
Topics: Animals; Bacteremia; Bacterial Infections; Disease Models, Animal; Female; Humans; Immunization; Imm | 2003 |
Stroke-induced immunodeficiency promotes spontaneous bacterial infections and is mediated by sympathetic activation reversal by poststroke T helper cell type 1-like immunostimulation.
Topics: Animals; Bacteremia; Bacterial Infections; Disease Models, Animal; Female; Humans; Immunization; Imm | 2003 |
Stroke-induced immunodeficiency promotes spontaneous bacterial infections and is mediated by sympathetic activation reversal by poststroke T helper cell type 1-like immunostimulation.
Topics: Animals; Bacteremia; Bacterial Infections; Disease Models, Animal; Female; Humans; Immunization; Imm | 2003 |