propofol has been researched along with Lung Injury, Acute in 32 studies
Propofol: An intravenous anesthetic agent which has the advantage of a very rapid onset after infusion or bolus injection plus a very short recovery period of a couple of minutes. (From Smith and Reynard, Textbook of Pharmacology, 1992, 1st ed, p206). Propofol has been used as ANTICONVULSANTS and ANTIEMETICS.
propofol : A phenol resulting from the formal substitution of the hydrogen at the 2 position of 1,3-diisopropylbenzene by a hydroxy group.
| Excerpt | Relevance | Reference |
|---|---|---|
| "The present study aimed to investigate the effects of propofol on neonatal acute lung injury (ALI) in a rat model and to examine the molecular mechanisms underlying propofol function." | 7.91 | Protective effects of propofol on experimental neonatal acute lung injury. ( Li, C; Yu, X, 2019) |
| "To investigate the effects of propofol pretreatment on lung morphology and heme oxygenase-1 expression in oleic acid -induced acute lung injury in rats." | 7.88 | Effects of propofol pretreatment on lung morphology and heme oxygenase-1 expression in oleic acid-induced acute lung injury in rats. ( Li, M; Sun, J; Tan, Z; Wang, H, 2018) |
| " This investigation explored the protective effects of propofol and whether propofol potentiates the protective effects of sevoflurane against lipopolysaccharide (LPS)-induced acute lung injury." | 7.85 | Propofol Potentiates Sevoflurane-Induced Inhibition of Nuclear Factor--κB-Mediated Inflammatory Responses and Regulation of Mitogen-Activated Protein Kinases Pathways via Toll-like Receptor 4 Signaling in Lipopolysaccharide-Induced Acute Lung Injury in Mi ( Fang, H; Liu, W; Zhu, H, 2017) |
| "This study aimed to investigate whether propofol pretreatment can protect against liver transplantation-induced acute lung injury (ALI) and to explore whether Nrf2 pathway is involved in the protections provided by propofol pretreatment." | 7.80 | Propofol activation of the Nrf2 pathway is associated with amelioration of acute lung injury in a rat liver transplantation model. ( Chi, X; Hei, Z; Luo, G; Xia, Z; Yao, W; Zhang, A; Zhu, G, 2014) |
| "We evaluated whether the short-term use of dexmedetomidine and propofol may attenuate inflammatory response and improve lung morphofunction in experimental acute lung injury (ALI)." | 7.80 | Effects of short-term propofol and dexmedetomidine on pulmonary morphofunction and biological markers in experimental mild acute lung injury. ( Araújo, MN; Cavalcanti, V; Fernandes, FC; Heil, LB; Morales, MM; Pelosi, P; Rocco, PR; Samary, CS; Santos, CL; Silva, PL; Villela, N, 2014) |
| "The aim of this study was to investigate the effect of propofol pretreatment on lipopolysaccharide (LPS)-induced acute lung injury (ALI) and the role of the phosphoinositide-3-kinase/protein kinase B (PI3K/Akt) pathway in this procedure." | 7.80 | Propofol pretreatment attenuates lipopolysaccharide-induced acute lung injury in rats by activating the phosphoinositide-3-kinase/Akt pathway. ( Hu, GC; Li, JF; Liu, GJ; Zhao, LL; Zhu, SS, 2014) |
| "We investigated the effect of propofol (Prop) administration (10 mg kg-1 h-1, intravenously) on lipopolysaccharide (LPS)-induced acute lung injury and its effect on cluster of differentiation (CD) 14 and Toll-like receptor (TLR) 4 expression in lung tissue of anesthetized, ventilated rats." | 7.79 | Propofol exerts anti-inflammatory effects in rats with lipopolysaccharide-induced acute lung injury by inhibition of CD14 and TLR4 expression. ( Chen, WM; Fujino, Y; Ma, L; Mashimo, T; Uchiyama, A; Wu, XY; Zhang, LH, 2013) |
| "To investigate the effect of postconditioning with propofol on Toll-like receptor 4 (TLR4) expression in the lung tissue in lipopolysaccharide (LPS)-induced acute lung injury (ALI) rats." | 7.78 | [The effects of postconditioning with propofol on Toll-like receptor 4 expression in the lung tissue of rat with acute lung injury]. ( Li, GF; Luan, T; Tong, X; Zang, B, 2012) |
| "To assess the effects of propofol treatments at different time points on acute lung injury and on the expression of transforming growth factor (TGF)-beta1 and the downstream target of TGF-beta1, Smad 2, in the lung tissues in the endotoxic rats." | 7.76 | Early administration of propofol protects against endotoxin-induced acute lung injury in rats by inhibiting the TGF-beta1-Smad2 dependent pathway. ( Ding, N; Gao, J; Xu, SQ; Xue, FS; Zhao, WX; Zhou, LJ, 2010) |
| "Ketamine prevented the increase in markers of oxidative stress and inflammation mediators, both in plasma and lung tissue." | 5.39 | Effects of ketamine, propofol, and ketofol on proinflammatory cytokines and markers of oxidative stress in a rat model of endotoxemia-induced acute lung injury. ( Aricioglu, A; Cumaoglu, A; Ergin, V; Gokcinar, D; Menevse, A, 2013) |
| "Limb RIPC attenuates acute lung injury via improving intraoperative pulmonary oxygenation in patients without severe pulmonary disease after lung resection under propofol-remifentanil anesthesia." | 5.19 | Limb remote ischemic preconditioning attenuates lung injury after pulmonary resection under propofol-remifentanil anesthesia: a randomized controlled study. ( Huang, WQ; Li, C; Li, YS; Liu, KX; Wu, Y; Xu, M, 2014) |
| "The present study aimed to investigate the effects of propofol on neonatal acute lung injury (ALI) in a rat model and to examine the molecular mechanisms underlying propofol function." | 3.91 | Protective effects of propofol on experimental neonatal acute lung injury. ( Li, C; Yu, X, 2019) |
| "To investigate the effects of propofol pretreatment on lung morphology and heme oxygenase-1 expression in oleic acid -induced acute lung injury in rats." | 3.88 | Effects of propofol pretreatment on lung morphology and heme oxygenase-1 expression in oleic acid-induced acute lung injury in rats. ( Li, M; Sun, J; Tan, Z; Wang, H, 2018) |
| "In animal models, both sevoflurane and propofol protect against acute lung injury (ALI), especially when administered prior to ALI onset." | 3.85 | Sevoflurane Posttreatment Attenuates Lung Injury Induced by Oleic Acid in Dogs. ( Du, G; Li, Z; Liu, J; Wang, S, 2017) |
| " This investigation explored the protective effects of propofol and whether propofol potentiates the protective effects of sevoflurane against lipopolysaccharide (LPS)-induced acute lung injury." | 3.85 | Propofol Potentiates Sevoflurane-Induced Inhibition of Nuclear Factor--κB-Mediated Inflammatory Responses and Regulation of Mitogen-Activated Protein Kinases Pathways via Toll-like Receptor 4 Signaling in Lipopolysaccharide-Induced Acute Lung Injury in Mi ( Fang, H; Liu, W; Zhu, H, 2017) |
| "This study aimed to investigate whether propofol pretreatment can protect against liver transplantation-induced acute lung injury (ALI) and to explore whether Nrf2 pathway is involved in the protections provided by propofol pretreatment." | 3.80 | Propofol activation of the Nrf2 pathway is associated with amelioration of acute lung injury in a rat liver transplantation model. ( Chi, X; Hei, Z; Luo, G; Xia, Z; Yao, W; Zhang, A; Zhu, G, 2014) |
| "We evaluated whether the short-term use of dexmedetomidine and propofol may attenuate inflammatory response and improve lung morphofunction in experimental acute lung injury (ALI)." | 3.80 | Effects of short-term propofol and dexmedetomidine on pulmonary morphofunction and biological markers in experimental mild acute lung injury. ( Araújo, MN; Cavalcanti, V; Fernandes, FC; Heil, LB; Morales, MM; Pelosi, P; Rocco, PR; Samary, CS; Santos, CL; Silva, PL; Villela, N, 2014) |
| "The aim of this study was to investigate the effect of propofol pretreatment on lipopolysaccharide (LPS)-induced acute lung injury (ALI) and the role of the phosphoinositide-3-kinase/protein kinase B (PI3K/Akt) pathway in this procedure." | 3.80 | Propofol pretreatment attenuates lipopolysaccharide-induced acute lung injury in rats by activating the phosphoinositide-3-kinase/Akt pathway. ( Hu, GC; Li, JF; Liu, GJ; Zhao, LL; Zhu, SS, 2014) |
| "We investigated the effect of propofol (Prop) administration (10 mg kg-1 h-1, intravenously) on lipopolysaccharide (LPS)-induced acute lung injury and its effect on cluster of differentiation (CD) 14 and Toll-like receptor (TLR) 4 expression in lung tissue of anesthetized, ventilated rats." | 3.79 | Propofol exerts anti-inflammatory effects in rats with lipopolysaccharide-induced acute lung injury by inhibition of CD14 and TLR4 expression. ( Chen, WM; Fujino, Y; Ma, L; Mashimo, T; Uchiyama, A; Wu, XY; Zhang, LH, 2013) |
| "The antioxidant properties of propofol have been shown to improve ischemia/reperfusion injury." | 3.78 | Propofol prevents lung injury following intestinal ischemia-reperfusion. ( Agrogiannis, G; Kalimeris, K; Kostopanagiotou, G; Nakos, G; Nomikos, T; Perrea, D; Vasileiou, I; Xanthopoulou, MN, 2012) |
| "To investigate the effect of postconditioning with propofol on Toll-like receptor 4 (TLR4) expression in the lung tissue in lipopolysaccharide (LPS)-induced acute lung injury (ALI) rats." | 3.78 | [The effects of postconditioning with propofol on Toll-like receptor 4 expression in the lung tissue of rat with acute lung injury]. ( Li, GF; Luan, T; Tong, X; Zang, B, 2012) |
| " We investigated the effect of propofol on HIF-1α expression and acute lung injury in LPS-treated mice." | 3.77 | Propofol inhibits lipopolysaccharide-induced lung epithelial cell injury by reducing hypoxia-inducible factor-1alpha expression. ( Cho, W; Chu, CC; Hsing, CH; Lin, MC; So, EC; Wang, JJ; Yeh, CH, 2011) |
| "To assess the effects of propofol treatments at different time points on acute lung injury and on the expression of transforming growth factor (TGF)-beta1 and the downstream target of TGF-beta1, Smad 2, in the lung tissues in the endotoxic rats." | 3.76 | Early administration of propofol protects against endotoxin-induced acute lung injury in rats by inhibiting the TGF-beta1-Smad2 dependent pathway. ( Ding, N; Gao, J; Xu, SQ; Xue, FS; Zhao, WX; Zhou, LJ, 2010) |
| "In acute lung injury (ALI)/acute respiratory distress syndrome (ARDS), recruitment manoeuvres (RMs) are used frequently." | 2.76 | Prolonged moderate pressure recruitment manoeuvre results in lower optimal positive end-expiratory pressure and plateau pressure. ( Lindgren, S; Lowhagen, K; Lundin, S; Odenstedt, H; Stenqvist, O, 2011) |
| "Pretreatment with propofol markedly attenuated lung injury (such as reducing the lung edema and permeability), increased MDA content and MPO activity, and restored SOD activity induced by IIR, accompanied by inhibiting the effect of the HMGB1/TLR4/PKR signaling pathway." | 1.62 | Pretreatment with Propofol Reduces Pulmonary Injury in a Pig Model of Intestinal Ischemia-Reperfusion via Suppressing the High-Mobility Group Box 1 Protein (HMGB1)/Toll-Like Receptor 4 (TLR4)/Protein Kinase R (PKR) Signaling Pathway. ( Bian, WY; Chen, YP; Tang, J; Xu, B, 2021) |
| "We present a case of rapid respiratory failure in an otherwise healthy and young patient who used a vaporiser containing tetrahydrocannabinol (THC) during the month prior to admission." | 1.56 | Sedation challenges in patients with E-cigarette, or vaping, product use-associated lung injury (EVALI). ( Markowski, LM; Maslonka, MA; Miller, PJ; Schertz, AR, 2020) |
| "Ketamine prevented the increase in markers of oxidative stress and inflammation mediators, both in plasma and lung tissue." | 1.39 | Effects of ketamine, propofol, and ketofol on proinflammatory cytokines and markers of oxidative stress in a rat model of endotoxemia-induced acute lung injury. ( Aricioglu, A; Cumaoglu, A; Ergin, V; Gokcinar, D; Menevse, A, 2013) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (3.13) | 29.6817 |
| 2010's | 26 (81.25) | 24.3611 |
| 2020's | 5 (15.63) | 2.80 |
| Authors | Studies |
|---|---|
| Liu, Z | 2 |
| Li, C | 4 |
| Li, Y | 1 |
| Yu, L | 1 |
| Qu, M | 1 |
| Zhang, J | 1 |
| Zhang, F | 1 |
| Chang, Y | 1 |
| Maslonka, MA | 1 |
| Schertz, AR | 1 |
| Markowski, LM | 1 |
| Miller, PJ | 1 |
| Bian, WY | 1 |
| Chen, YP | 1 |
| Xu, B | 1 |
| Tang, J | 1 |
| Wang, L | 1 |
| Tang, X | 1 |
| Li, S | 1 |
| Du, G | 1 |
| Wang, S | 1 |
| Li, Z | 1 |
| Liu, J | 1 |
| Liu, W | 1 |
| Zhu, H | 1 |
| Fang, H | 1 |
| Tan, Z | 1 |
| Wang, H | 1 |
| Sun, J | 1 |
| Li, M | 1 |
| Chen, CY | 1 |
| Tsai, YF | 1 |
| Huang, WJ | 1 |
| Chang, SH | 1 |
| Hwang, TL | 1 |
| Yu, X | 1 |
| Feng, Z | 1 |
| Wang, JW | 1 |
| Wang, Y | 1 |
| Dong, WW | 1 |
| Xu, ZF | 1 |
| Ma, L | 1 |
| Wu, XY | 1 |
| Zhang, LH | 1 |
| Chen, WM | 1 |
| Uchiyama, A | 1 |
| Mashimo, T | 1 |
| Fujino, Y | 1 |
| Lu, HL | 1 |
| Qian, YN | 1 |
| Gokcinar, D | 1 |
| Ergin, V | 1 |
| Cumaoglu, A | 1 |
| Menevse, A | 1 |
| Aricioglu, A | 1 |
| Yao, W | 3 |
| Luo, G | 3 |
| Zhu, G | 2 |
| Chi, X | 3 |
| Zhang, A | 1 |
| Xia, Z | 1 |
| Hei, Z | 4 |
| Xu, M | 1 |
| Wu, Y | 1 |
| Li, YS | 1 |
| Huang, WQ | 1 |
| Liu, KX | 2 |
| Zhao, W | 1 |
| Zhou, S | 1 |
| Gan, X | 1 |
| Su, G | 2 |
| Yuan, D | 3 |
| Cavalcanti, V | 1 |
| Santos, CL | 1 |
| Samary, CS | 1 |
| Araújo, MN | 1 |
| Heil, LB | 1 |
| Morales, MM | 1 |
| Silva, PL | 1 |
| Pelosi, P | 1 |
| Fernandes, FC | 1 |
| Villela, N | 1 |
| Rocco, PR | 1 |
| Zhao, LL | 1 |
| Hu, GC | 1 |
| Zhu, SS | 1 |
| Li, JF | 1 |
| Liu, GJ | 1 |
| Ruchalla, E | 1 |
| Ventzke, MM | 1 |
| Leiblein, T | 1 |
| Kugler, M | 1 |
| Wulf, H | 1 |
| Quabach, R | 1 |
| Escher, M | 1 |
| Kinoshita, H | 1 |
| Wang, X | 1 |
| Liu, C | 1 |
| Wang, G | 1 |
| Liu, Y | 1 |
| Feng, J | 1 |
| Zhu, Q | 1 |
| Cai, J | 1 |
| Kellner, P | 1 |
| Müller, M | 1 |
| Piegeler, T | 1 |
| Eugster, P | 1 |
| Booy, C | 1 |
| Schläpfer, M | 1 |
| Beck-Schimmer, B | 1 |
| Haitsma, JJ | 1 |
| Lachmann, B | 1 |
| Papadakos, PJ | 1 |
| Gao, J | 1 |
| Zhao, WX | 1 |
| Xue, FS | 1 |
| Zhou, LJ | 1 |
| Xu, SQ | 1 |
| Ding, N | 1 |
| Vasileiou, I | 1 |
| Kalimeris, K | 1 |
| Nomikos, T | 1 |
| Xanthopoulou, MN | 1 |
| Perrea, D | 1 |
| Agrogiannis, G | 1 |
| Nakos, G | 1 |
| Kostopanagiotou, G | 1 |
| Lowhagen, K | 1 |
| Lindgren, S | 1 |
| Odenstedt, H | 1 |
| Stenqvist, O | 1 |
| Lundin, S | 1 |
| Yeh, CH | 1 |
| Cho, W | 1 |
| So, EC | 1 |
| Chu, CC | 1 |
| Lin, MC | 1 |
| Wang, JJ | 1 |
| Hsing, CH | 1 |
| Li, GF | 1 |
| Tong, X | 1 |
| Luan, T | 1 |
| Zang, B | 1 |
| Trial | Phase | Enrollment | Study Type | Start Date | Status | ||
|---|---|---|---|---|---|---|---|
| Correlation Between Blood Pressure, Heart Rate and Plasma Corticotropin, Cortisol Under Surgical Skin Incision[NCT03892538] | 134 participants (Actual) | Observational | 2018-10-01 | Completed | |||
| Sevoflurane Sedation: A Potentially Promising Immunomodulation in Patients With Septic Shock[NCT03643367] | Phase 2 | 153 participants (Anticipated) | Interventional | 2025-01-31 | Not yet recruiting | ||
| AnaConDa-therapy in COVID-19 Patients[NCT05586126] | 42 participants (Actual) | Observational | 2020-10-01 | Terminated (stopped due to Concerns about possible association between drug and increased ICU mortality) | |||
| A Randomized Pilot Clinical Trial of the Effects in Oxygenation and Hypoxic Pulmonary Vasoconstriction of Sevoflurane in Patient's Whit ARDS Secondary to Severe Acute Respiratory Syndrome Coronavirus 2 (SARS-CoV2)[NCT04998253] | Early Phase 1 | 24 participants (Actual) | Interventional | 2020-10-01 | Completed | ||
| [information is prepared from clinicaltrials.gov, extracted Sep-2024] | |||||||
2 trials available for propofol and Lung Injury, Acute
| Article | Year |
|---|---|
Limb remote ischemic preconditioning attenuates lung injury after pulmonary resection under propofol-remifentanil anesthesia: a randomized controlled study.
Topics: Acute Lung Injury; Aged; Analysis of Variance; Anesthesia, Intravenous; Anesthetics, Intravenous; Ca | 2014 |
Prolonged moderate pressure recruitment manoeuvre results in lower optimal positive end-expiratory pressure and plateau pressure.
Topics: Acute Lung Injury; Aged; Anesthetics, Intravenous; Cardiac Output; Cross-Over Studies; Electric Impe | 2011 |
30 other studies available for propofol and Lung Injury, Acute
| Article | Year |
|---|---|
Propofol Reduces Renal Ischemia Reperfusion-mediated Necroptosis by Up-regulation of SIRT1 in Rats.
Topics: Acute Lung Injury; Animals; Antioxidants; Apoptosis; Caspase 3; Cytokines; Ischemia; Kidney Diseases | 2022 |
Propofol post-conditioning lessens renal ischemia/reperfusion-induced acute lung injury associated with autophagy and apoptosis through MAPK signals in rats.
Topics: Acute Kidney Injury; Acute Lung Injury; Animals; Apoptosis; Autophagy; Disease Models, Animal; Human | 2020 |
Sedation challenges in patients with E-cigarette, or vaping, product use-associated lung injury (EVALI).
Topics: Acute Lung Injury; Analgesia; Computed Tomography Angiography; Deep Sedation; Dose-Response Relation | 2020 |
Pretreatment with Propofol Reduces Pulmonary Injury in a Pig Model of Intestinal Ischemia-Reperfusion via Suppressing the High-Mobility Group Box 1 Protein (HMGB1)/Toll-Like Receptor 4 (TLR4)/Protein Kinase R (PKR) Signaling Pathway.
Topics: Acute Lung Injury; Animals; HMGB1 Protein; Male; Propofol; Protective Agents; Reperfusion Injury; Sw | 2021 |
Propofol promotes migration, alleviates inflammation, and apoptosis of lipopolysaccharide-induced human pulmonary microvascular endothelial cells by activating PI3K/AKT signaling pathway via upregulating APOM expression.
Topics: Acute Lung Injury; Apolipoproteins M; Apoptosis; Cytokines; Endothelial Cells; Humans; Inflammation; | 2022 |
Sevoflurane Posttreatment Attenuates Lung Injury Induced by Oleic Acid in Dogs.
Topics: Acute Lung Injury; Anesthetics, Inhalation; Animals; Arterial Pressure; Cytoprotection; Disease Mode | 2017 |
Propofol Potentiates Sevoflurane-Induced Inhibition of Nuclear Factor--κB-Mediated Inflammatory Responses and Regulation of Mitogen-Activated Protein Kinases Pathways via Toll-like Receptor 4 Signaling in Lipopolysaccharide-Induced Acute Lung Injury in Mi
Topics: Acute Lung Injury; Anesthetics, Inhalation; Anesthetics, Intravenous; Animals; Drug Synergism; Lipop | 2017 |
Effects of propofol pretreatment on lung morphology and heme oxygenase-1 expression in oleic acid-induced acute lung injury in rats.
Topics: Acute Lung Injury; Animals; Heme Oxygenase-1; Immunohistochemistry; Lung; Male; Oleic Acid; Propofol | 2018 |
Propofol inhibits endogenous formyl peptide-induced neutrophil activation and alleviates lung injury.
Topics: Acute Lung Injury; Animals; Cell Movement; Chemotactic Factors; Gene Expression Regulation; Humans; | 2018 |
Protective effects of propofol on experimental neonatal acute lung injury.
Topics: Acute Lung Injury; Animals; Animals, Newborn; Cytokines; Lipopolysaccharides; Lung; Male; Malondiald | 2019 |
Propofol Protects Lung Endothelial Barrier Function by Suppression of High-Mobility Group Box 1 (HMGB1) Release and Mitochondrial Oxidative Damage Catalyzed by HMGB1.
Topics: Acute Lung Injury; Animals; Catalysis; Disease Models, Animal; Endothelial Cells; Endothelium, Vascu | 2019 |
Propofol exerts anti-inflammatory effects in rats with lipopolysaccharide-induced acute lung injury by inhibition of CD14 and TLR4 expression.
Topics: Acute Lung Injury; Animals; Anti-Inflammatory Agents; Inflammation Mediators; Lipopolysaccharide Rec | 2013 |
[Protective effects of propofol combined with ulinastatin on acute lung injury induced by endotoxin in rats].
Topics: Acute Lung Injury; Animals; Endotoxins; Glycoproteins; Male; Propofol; Rats; Rats, Sprague-Dawley | 2013 |
Effects of ketamine, propofol, and ketofol on proinflammatory cytokines and markers of oxidative stress in a rat model of endotoxemia-induced acute lung injury.
Topics: Acute Lung Injury; Administration, Intravenous; Animals; Anti-Inflammatory Agents; Antioxidants; Bio | 2013 |
Propofol activation of the Nrf2 pathway is associated with amelioration of acute lung injury in a rat liver transplantation model.
Topics: Acute Lung Injury; Animals; Disease Models, Animal; Heme Oxygenase-1; Hydrogen Peroxide; Intracellul | 2014 |
Propofol prevents lung injury after intestinal ischemia-reperfusion by inhibiting the interaction between mast cell activation and oxidative stress.
Topics: Acetophenones; Acute Lung Injury; Animals; Cell Degranulation; Enzyme-Linked Immunosorbent Assay; Hy | 2014 |
Effects of short-term propofol and dexmedetomidine on pulmonary morphofunction and biological markers in experimental mild acute lung injury.
Topics: Acute Lung Injury; Animals; Cytokines; Dexmedetomidine; Disease Models, Animal; Endotoxins; Female; | 2014 |
Propofol alleviates acute lung injury following orthotopic autologous liver transplantation in rats via inhibition of the NADPH oxidase pathway.
Topics: Acute Lung Injury; Animals; Disease Models, Animal; Hypnotics and Sedatives; Liver Transplantation; | 2015 |
Propofol pretreatment attenuates lipopolysaccharide-induced acute lung injury in rats by activating the phosphoinositide-3-kinase/Akt pathway.
Topics: Acute Lung Injury; Animals; Blotting, Western; Bronchoalveolar Lavage Fluid; Enzyme-Linked Immunosor | 2014 |
[In Process Citation].
Topics: Acute Lung Injury; Anesthesia, Intravenous; Anesthetics, Intravenous; Female; Humans; Ischemic Preco | 2015 |
Another role of limb remote ischemic preconditioning in patients with lung cancer.
Topics: Acute Lung Injury; Anesthesia, Intravenous; Anesthetics, Intravenous; Female; Humans; Ischemic Preco | 2015 |
In reply.
Topics: Acute Lung Injury; Anesthesia, Intravenous; Anesthetics, Intravenous; Female; Humans; Ischemic Preco | 2015 |
Propofol Protects Rats and Human Alveolar Epithelial Cells Against Lipopolysaccharide-Induced Acute Lung Injury via Inhibiting HMGB1 Expression.
Topics: Acute Lung Injury; Alveolar Epithelial Cells; Animals; Disease Models, Animal; Gene Expression; HMGB | 2016 |
Propofol attenuated liver transplantation-induced acute lung injury via connexin43 gap junction inhibition.
Topics: Acute Lung Injury; Animals; Cell Line; Connexin 43; Connexins; Gap Junction beta-1 Protein; Gap Junc | 2016 |
Sevoflurane Abolishes Oxygenation Impairment in a Long-Term Rat Model of Acute Lung Injury.
Topics: Acute Lung Injury; Anesthetics, Inhalation; Anesthetics, Intravenous; Animals; Anti-Inflammatory Age | 2017 |
Sevoflurane Abolishes Oxygenation Impairment in a Long-Term Rat Model of Acute Lung Injury.
Topics: Acute Lung Injury; Anesthetics, Inhalation; Anesthetics, Intravenous; Animals; Anti-Inflammatory Age | 2017 |
Sevoflurane Abolishes Oxygenation Impairment in a Long-Term Rat Model of Acute Lung Injury.
Topics: Acute Lung Injury; Anesthetics, Inhalation; Anesthetics, Intravenous; Animals; Anti-Inflammatory Age | 2017 |
Sevoflurane Abolishes Oxygenation Impairment in a Long-Term Rat Model of Acute Lung Injury.
Topics: Acute Lung Injury; Anesthetics, Inhalation; Anesthetics, Intravenous; Animals; Anti-Inflammatory Age | 2017 |
Sevoflurane Abolishes Oxygenation Impairment in a Long-Term Rat Model of Acute Lung Injury.
Topics: Acute Lung Injury; Anesthetics, Inhalation; Anesthetics, Intravenous; Animals; Anti-Inflammatory Age | 2017 |
Sevoflurane Abolishes Oxygenation Impairment in a Long-Term Rat Model of Acute Lung Injury.
Topics: Acute Lung Injury; Anesthetics, Inhalation; Anesthetics, Intravenous; Animals; Anti-Inflammatory Age | 2017 |
Sevoflurane Abolishes Oxygenation Impairment in a Long-Term Rat Model of Acute Lung Injury.
Topics: Acute Lung Injury; Anesthetics, Inhalation; Anesthetics, Intravenous; Animals; Anti-Inflammatory Age | 2017 |
Sevoflurane Abolishes Oxygenation Impairment in a Long-Term Rat Model of Acute Lung Injury.
Topics: Acute Lung Injury; Anesthetics, Inhalation; Anesthetics, Intravenous; Animals; Anti-Inflammatory Age | 2017 |
Sevoflurane Abolishes Oxygenation Impairment in a Long-Term Rat Model of Acute Lung Injury.
Topics: Acute Lung Injury; Anesthetics, Inhalation; Anesthetics, Intravenous; Animals; Anti-Inflammatory Age | 2017 |
Additives in intravenous anesthesia modulate pulmonary inflammation in a model of LPS-induced respiratory distress.
Topics: Acute Lung Injury; Adjuvants, Anesthesia; Anesthesia, Intravenous; Animals; Bronchoalveolar Lavage F | 2009 |
Early administration of propofol protects against endotoxin-induced acute lung injury in rats by inhibiting the TGF-beta1-Smad2 dependent pathway.
Topics: Acute Lung Injury; Anesthetics, Intravenous; Animals; Hemodynamics; Humans; Lipopolysaccharides; Lun | 2010 |
Propofol prevents lung injury following intestinal ischemia-reperfusion.
Topics: Acute Lung Injury; Anesthetics, Intravenous; Animals; Antioxidants; Bronchoalveolar Lavage; Intestin | 2012 |
Propofol inhibits lipopolysaccharide-induced lung epithelial cell injury by reducing hypoxia-inducible factor-1alpha expression.
Topics: Acute Lung Injury; Anesthetics, Intravenous; Animals; Apoptosis; Cells, Cultured; Cytokines; Disease | 2011 |
[The effects of postconditioning with propofol on Toll-like receptor 4 expression in the lung tissue of rat with acute lung injury].
Topics: Acute Lung Injury; Animals; Lung; Male; Propofol; Rats; Rats, Sprague-Dawley; RNA, Messenger; Toll-L | 2012 |