prolinedithiocarbamate has been researched along with Disease Exacerbation in 4 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (25.00) | 29.6817 |
| 2010's | 3 (75.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Bo, L; Chen, X; Jin, F; Li, C; Liu, Q; Liu, W; Xu, D | 1 |
| Du, L; Lin, R; Liu, Q; Meng, K; Pan, C; Wu, W; Zhao, L | 1 |
| Baker, KM; Chigurupati, S; Gupta, S; Kumar, R; Kumar, S; Sen, S; Seqqat, R; Young, D | 1 |
| Harada, R; Kobayashi, M; Nakata, H; Taguchi, H; Uemura, Y | 1 |
4 other study(ies) available for prolinedithiocarbamate and Disease Exacerbation
| Article | Year |
|---|---|
Activation of TRPV1-dependent calcium oscillation exacerbates seawater inhalation-induced acute lung injury.
Topics: Actin Cytoskeleton; Acute Lung Injury; Administration, Inhalation; Animals; Calcium; Calcium Chelating Agents; Calcium Signaling; Capsaicin; Cell Line, Tumor; Cytosol; Disease Progression; Egtazic Acid; Extracellular Space; Fluorescence; Humans; Inflammation Mediators; Interleukin-1beta; Lung; Male; NF-kappa B; Phosphorylation; Proline; Rats, Sprague-Dawley; Seawater; Thiocarbamates; TRPV Cation Channels; Tumor Necrosis Factor-alpha | 2016 |
Hypoxia activates heparanase expression in an NF-kappaB dependent manner.
Topics: Antioxidants; Disease Progression; Enzyme Activation; Extracellular Matrix; Glucuronidase; Hypoxia; Matrix Metalloproteinase 9; Neoplasm Invasiveness; NF-kappa B; Oligonucleotides, Antisense; Oxygen; Proline; RNA, Messenger; Thiocarbamates; Time Factors | 2010 |
Inhibition of nuclear factor κB regresses cardiac hypertrophy by modulating the expression of extracellular matrix and adhesion molecules.
Topics: Animals; Antioxidants; Cardiomegaly; Cell Adhesion Molecules; Disease Progression; Drug Evaluation, Preclinical; Extracellular Matrix Proteins; Female; Gene Expression Regulation; Intercellular Signaling Peptides and Proteins; Male; Mice; Mice, Transgenic; NF-kappa B; Proline; Thiocarbamates | 2011 |
Cyclooxygenase-2 inhibitor NS-398 suppresses cell growth and constitutive production of granulocyte-colony stimulating factor and granulocyte macrophage-colony stimulating factor in lung cancer cells.
Topics: Cell Division; Cyclooxygenase 2; Cyclooxygenase 2 Inhibitors; Cyclooxygenase Inhibitors; Dinoprostone; Disease Progression; Drug Screening Assays, Antitumor; Enzyme Induction; Enzyme Inhibitors; Flavonoids; Gene Expression Regulation, Neoplastic; Granulocyte Colony-Stimulating Factor; Granulocyte-Macrophage Colony-Stimulating Factor; Humans; Imidazoles; Interleukin-1; Isoenzymes; Lung Neoplasms; MAP Kinase Signaling System; Membrane Proteins; Mitogen-Activated Protein Kinase Kinases; Mitogen-Activated Protein Kinases; Neoplasm Proteins; NF-kappa B; Nitrobenzenes; Proline; Prostaglandin-Endoperoxide Synthases; Pyridines; Recombinant Proteins; Sulfonamides; Thiocarbamates; Tumor Cells, Cultured; Tumor Necrosis Factor-alpha | 2003 |