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prolinedithiocarbamate and Cardiomegaly

prolinedithiocarbamate has been researched along with Cardiomegaly in 5 studies

Research

Studies (5)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's2 (40.00)29.6817
2010's3 (60.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Bolli, R; De Caterina, R; Ferdinandy, P; Geng, YJ; Madonna, R; Patterson, C; Rokosh, G1
Abdelhamid, G; El-Kadi, AO1
Baker, KM; Chigurupati, S; Gupta, S; Kumar, R; Kumar, S; Sen, S; Seqqat, R; Young, D1
Browder, IW; Gao, X; Ha, T; Kao, RL; Kelley, J; Li, C; Li, Y; Williams, DL1
Barta, P; Dechend, R; Dietz, R; Fiebeler, A; Ganten, D; Haller, H; Luft, FC; Muller, DN; Park, JK; Theuer, J1

Other Studies

5 other study(ies) available for prolinedithiocarbamate and Cardiomegaly

ArticleYear
Co-activation of nuclear factor-κB and myocardin/serum response factor conveys the hypertrophy signal of high insulin levels in cardiac myoblasts.
    The Journal of biological chemistry, 2014, Jul-11, Volume: 289, Issue:28

    Topics: Animals; Antineoplastic Agents; Cardiomegaly; Cells, Cultured; Dogs; Gene Expression Regulation; Insulin; Mice; Myoblasts, Cardiac; NF-kappa B; Nuclear Proteins; Proline; Serum Response Factor; Thiocarbamates; Trans-Activators; Tumor Necrosis Factor-alpha; Ubiquitin-Protein Ligases

2014
Buthionine sulfoximine, an inhibitor of glutathione biosynthesis, induces expression of soluble epoxide hydrolase and markers of cellular hypertrophy in a rat cardiomyoblast cell line: roles of the NF-κB and MAPK signaling pathways.
    Free radical biology & medicine, 2015, Volume: 82

    Topics: Animals; Antioxidants; Atrial Natriuretic Factor; Butadienes; Buthionine Sulfoximine; Cardiomegaly; Cell Line; Cell Survival; Enzyme Activation; Epoxide Hydrolases; Extracellular Signal-Regulated MAP Kinases; Flavonoids; Gene Expression Regulation; Glutathione; Heart Failure; I-kappa B Proteins; JNK Mitogen-Activated Protein Kinases; MAP Kinase Signaling System; Myoblasts, Cardiac; Natriuretic Peptide, Brain; NF-kappa B p50 Subunit; NF-KappaB Inhibitor alpha; Nitriles; Oxidative Stress; p38 Mitogen-Activated Protein Kinases; Phosphorylation; Proline; Rats; RNA, Messenger; Thiocarbamates; Transcription Factor RelA; Up-Regulation

2015
Inhibition of nuclear factor κB regresses cardiac hypertrophy by modulating the expression of extracellular matrix and adhesion molecules.
    Free radical biology & medicine, 2011, Jan-01, Volume: 50, Issue:1

    Topics: Animals; Antioxidants; Cardiomegaly; Cell Adhesion Molecules; Disease Progression; Drug Evaluation, Preclinical; Extracellular Matrix Proteins; Female; Gene Expression Regulation; Intercellular Signaling Peptides and Proteins; Male; Mice; Mice, Transgenic; NF-kappa B; Proline; Thiocarbamates

2011
NF-kappaB activation is required for the development of cardiac hypertrophy in vivo.
    American journal of physiology. Heart and circulatory physiology, 2004, Volume: 287, Issue:4

    Topics: Adenoviridae; Animals; Antioxidants; Aorta; Atrial Natriuretic Factor; Cardiomegaly; I-kappa B Kinase; I-kappa B Proteins; Ligation; Male; Myocardium; Natriuretic Peptide, Brain; NF-kappa B; NF-KappaB Inhibitor alpha; Proline; Protein Serine-Threonine Kinases; Rats; Rats, Sprague-Dawley; RNA, Messenger; Signal Transduction; Thiocarbamates; Transfection

2004
Angiotensin II induced inflammation in the kidney and in the heart of double transgenic rats.
    BMC cardiovascular disorders, 2002, Volume: 2

    Topics: Angiotensin II; Angiotensinogen; Animals; Animals, Genetically Modified; Cardiomegaly; Electrophoretic Mobility Shift Assay; Enzyme Inhibitors; Heart; Humans; Intercellular Adhesion Molecule-1; Kidney; Models, Animal; Myocarditis; Necrosis; Nephritis; NF-kappa B; Nitric Oxide Synthase; Nitric Oxide Synthase Type II; Oxidative Stress; Proline; Rats; Rats, Sprague-Dawley; Renin; Thiocarbamates; Vascular Cell Adhesion Molecule-1

2002