prolinedithiocarbamate has been researched along with Cardiomegaly in 5 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 2 (40.00) | 29.6817 |
2010's | 3 (60.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Bolli, R; De Caterina, R; Ferdinandy, P; Geng, YJ; Madonna, R; Patterson, C; Rokosh, G | 1 |
Abdelhamid, G; El-Kadi, AO | 1 |
Baker, KM; Chigurupati, S; Gupta, S; Kumar, R; Kumar, S; Sen, S; Seqqat, R; Young, D | 1 |
Browder, IW; Gao, X; Ha, T; Kao, RL; Kelley, J; Li, C; Li, Y; Williams, DL | 1 |
Barta, P; Dechend, R; Dietz, R; Fiebeler, A; Ganten, D; Haller, H; Luft, FC; Muller, DN; Park, JK; Theuer, J | 1 |
5 other study(ies) available for prolinedithiocarbamate and Cardiomegaly
Article | Year |
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Co-activation of nuclear factor-κB and myocardin/serum response factor conveys the hypertrophy signal of high insulin levels in cardiac myoblasts.
Topics: Animals; Antineoplastic Agents; Cardiomegaly; Cells, Cultured; Dogs; Gene Expression Regulation; Insulin; Mice; Myoblasts, Cardiac; NF-kappa B; Nuclear Proteins; Proline; Serum Response Factor; Thiocarbamates; Trans-Activators; Tumor Necrosis Factor-alpha; Ubiquitin-Protein Ligases | 2014 |
Buthionine sulfoximine, an inhibitor of glutathione biosynthesis, induces expression of soluble epoxide hydrolase and markers of cellular hypertrophy in a rat cardiomyoblast cell line: roles of the NF-κB and MAPK signaling pathways.
Topics: Animals; Antioxidants; Atrial Natriuretic Factor; Butadienes; Buthionine Sulfoximine; Cardiomegaly; Cell Line; Cell Survival; Enzyme Activation; Epoxide Hydrolases; Extracellular Signal-Regulated MAP Kinases; Flavonoids; Gene Expression Regulation; Glutathione; Heart Failure; I-kappa B Proteins; JNK Mitogen-Activated Protein Kinases; MAP Kinase Signaling System; Myoblasts, Cardiac; Natriuretic Peptide, Brain; NF-kappa B p50 Subunit; NF-KappaB Inhibitor alpha; Nitriles; Oxidative Stress; p38 Mitogen-Activated Protein Kinases; Phosphorylation; Proline; Rats; RNA, Messenger; Thiocarbamates; Transcription Factor RelA; Up-Regulation | 2015 |
Inhibition of nuclear factor κB regresses cardiac hypertrophy by modulating the expression of extracellular matrix and adhesion molecules.
Topics: Animals; Antioxidants; Cardiomegaly; Cell Adhesion Molecules; Disease Progression; Drug Evaluation, Preclinical; Extracellular Matrix Proteins; Female; Gene Expression Regulation; Intercellular Signaling Peptides and Proteins; Male; Mice; Mice, Transgenic; NF-kappa B; Proline; Thiocarbamates | 2011 |
NF-kappaB activation is required for the development of cardiac hypertrophy in vivo.
Topics: Adenoviridae; Animals; Antioxidants; Aorta; Atrial Natriuretic Factor; Cardiomegaly; I-kappa B Kinase; I-kappa B Proteins; Ligation; Male; Myocardium; Natriuretic Peptide, Brain; NF-kappa B; NF-KappaB Inhibitor alpha; Proline; Protein Serine-Threonine Kinases; Rats; Rats, Sprague-Dawley; RNA, Messenger; Signal Transduction; Thiocarbamates; Transfection | 2004 |
Angiotensin II induced inflammation in the kidney and in the heart of double transgenic rats.
Topics: Angiotensin II; Angiotensinogen; Animals; Animals, Genetically Modified; Cardiomegaly; Electrophoretic Mobility Shift Assay; Enzyme Inhibitors; Heart; Humans; Intercellular Adhesion Molecule-1; Kidney; Models, Animal; Myocarditis; Necrosis; Nephritis; NF-kappa B; Nitric Oxide Synthase; Nitric Oxide Synthase Type II; Oxidative Stress; Proline; Rats; Rats, Sprague-Dawley; Renin; Thiocarbamates; Vascular Cell Adhesion Molecule-1 | 2002 |