proline has been researched along with Movement Disorders in 4 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 1 (25.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (25.00) | 29.6817 |
| 2010's | 1 (25.00) | 24.3611 |
| 2020's | 1 (25.00) | 2.80 |
| Authors | Studies |
|---|---|
| Barwick, K; Benkerroum, H; Bras, J; Campeau, PM; Carr, L; Carss, K; Chong, WK; De Gressi, S; Farabella, I; Gorman, KM; Grozeva, D; Guerreiro, R; Haack, TB; Harvey, RJ; Hirano, Y; Hirata, H; Kamsteeg, EJ; Kurian, MA; Lunt, P; Maher, ER; Mankad, K; Meyer, E; Pall, H; Papandreou, A; Raymond, FL; Reid, KM; Reith, ME; Salian, S; Scott, RH; Sheipouri, D; Simpson, MA; Smith, M; Spaull, R; Sudhakar, S; Topf, M; Vandenberg, RJ; Zhen, J | 1 |
| Cattepoel, S; Eckert, A; Kuehnle, K; Kulic, L; Nitsch, RM; Pagani, L; Rhein, V; Tracy, J; Wollmer, MA | 1 |
| Frasier, M; Haas, C; Kahle, P; Lee, JM; Magnuson, D; McCarthy, L; Walzer, M; Wolozin, B | 1 |
| Barbeau, A; Kastin, AJ | 1 |
4 other study(ies) available for proline and Movement Disorders
| Article | Year |
|---|---|
MED27, SLC6A7, and MPPE1 Variants in a Complex Neurodevelopmental Disorder with Severe Dystonia.
Topics: Animals; Dystonia; Dystonic Disorders; Movement Disorders; Neurodevelopmental Disorders; Proline; RNA; Zebrafish | 2022 |
Combined expression of tau and the Harlequin mouse mutation leads to increased mitochondrial dysfunction, tau pathology and neurodegeneration.
Topics: Adenosine Triphosphate; Age Factors; Animals; Apoptosis; Apoptosis Inducing Factor; Brain; Caspase 3; Electron Transport Complex I; Gene Expression Regulation; Genetic Predisposition to Disease; Humans; In Situ Nick-End Labeling; Leucine; Lipid Peroxidation; Male; Mice; Mice, Inbred C57BL; Mice, Mutant Strains; Mice, Transgenic; Mitochondria; Mitochondrial Diseases; Movement Disorders; Mutation; Nerve Degeneration; Nerve Tissue Proteins; Oxidative Stress; Proline; Proton Pumps; Silver Staining; Statistics, Nonparametric; tau Proteins; Tauopathies | 2011 |
Tau phosphorylation increases in symptomatic mice overexpressing A30P alpha-synuclein.
Topics: Age Factors; Alanine; alpha-Synuclein; Amino Acid Substitution; Animals; Antibodies, Monoclonal; Blotting, Western; Brain; Disease Models, Animal; Female; Glial Fibrillary Acidic Protein; Humans; Immunohistochemistry; JNK Mitogen-Activated Protein Kinases; Male; MAP Kinase Kinase 4; Mice; Mice, Inbred C57BL; Mice, Transgenic; Mitogen-Activated Protein Kinase Kinases; Movement Disorders; Mutation; Nerve Tissue Proteins; Neurons; Parkinson Disease; Phosphorylation; Proline; Synucleins; tau Proteins | 2005 |
Preliminary clinical studies with L-prolyl-L-leucyl-glycine amide in Parkinson's disease.
Topics: Administration, Oral; Aged; Amides; Antiparkinson Agents; Dihydroxyphenylalanine; Female; Glycine; Humans; Injections, Intravenous; Leucine; Male; Middle Aged; Movement Disorders; Parkinson Disease; Peptides; Proline | 1972 |